L3 Hip Conditions Flashcards
Instability
excessive mobility that causes pain with or without the symptom of hip joint unsteadiness
can be dysplasia, subluxation, dislocation
Causes of instability
traumatic
atraumatic
Traumatic instability
MVA or other high energy event is the cause
Atraumatic Instability
repetitve axial loading and rotation in the presence of anatomic variation
RF for Atraumatic Instability
golfers, gymnastics, football, hockey, figure skating, ballet, adolescent athletes with mobility
acetabular dysplasia
soft tissue hypermobility
ligamentum teres tear
Instability S/S
pain in groin or anterior thigh
popping, locking, snapping
worsening pain with sitting, walking, running
limping, weakness, sensation of dead leg
unable to perform strenuous activities
Instability Imaging
Center edge angle can be altered
can develop labral hypertrophy to stabilize
can progress to labral tears and OA in adulthood
Center Edge angle
femoral head and acetabulum
<25° dysplasia
>39° over-coverage/pincer type FAI
PT management of instability
- Patient Ed
- Postural re-education
- Avoiding long lever hip flexion and high impact
- Strengthening of rotators, abductors, extensors, adductors
- Neuromuscular training/Proprioceptive
Should have PT for 8-12 weeks before considering surgery
Pericetabular Osteotomy
-for pts <40 and have failed with PT
-may delay or avoid need for THA in future
-high rate of complications
-NWB for 6-8 weeks
-return to school in 12 weeks, sport in 6-12 mo
Dysplasia
(the acetabulum is too shallow)
reduces joint stability, but joint can be unstable without having it
left untreated will lead to labral tears and OA
dysplasia + OA = need for THA
Femoracetabular Impingement
abnormal contact between femoral head/neck and acetabular margin from structural variations in proximal femur and/or acetabulum
may result in labral tears, chondral damage, OA
CAM structure
non-spherical shape of the femoral head, protrusion of the head-neck junction
thickening
PINCER structure
deeper acetabular socket, associated with acetabular retroversion
increased center edge angle
MIXED Structure
combination of cam and pincer, most common category of impingement
Epidemiology of FAI
10-15% of population
CAM = young, male athletes
Pincer = active middle aged women
most likely to be symptomatic with high speed, high impact rotational sports
FAI Clinical Exam
Hx = anterior groin pain, C sign, clicking, giving away
Obs = pelvic tilt
ROM = decreased flexion and IR, tight ilioposoas and joint
overall weakness
gait will show a slower cadence and limited hip extension. Squats will have limited motion
FAI and muscle weakness
not a strong correlation between FAI and specific muscle weakness
global hip muscle weakness moderate predictor of whether FAI was symptomatic
Does FAI mean automatic pain?
asymptomatic individuals is 29%
20-40s are most likely to have pain that is ONLY FAI. vs older will have OA w/FAI
self-efficacy and psychologic factors are more present in pts with pain and weakness
PT Management for FAI
activity modifcation
patient ed
manual therapy
avoid end range stretching
progressive pain-free strengthening
neuro re-education
squat variations
Labral Tear Epidemiology
most common in 4th decade, more women vs men
22-55% of patients with hip or groin pain
74% have no clear MOI
Traumatic Labral Tear
acute, rapid twisting, hyperabduction, falling
compression from MVA
Atraumatic Labral tear
combo of anatomic variants with repetitive forces
RF for Labral tears
female
FAI
higher BMI
capsular laxity
hip dysplasia
illiopsoas overactivity
degeneration of surrounding tissues