L12 Allergic Rhinitis, Tinnitus, Urticaria Flashcards
Barotrauma
Damage/discomfort to the ear due to pressure differences b/w middle ear and outside world
Symptoms of barotrauma
pressure, pain, hearing loss, or tinnitus, possible hemotympanum, middle ear effusion, TM rupture
Treatment of barotrauma
avoidance, oral/nasal decongestants, swallowing, valsalva, chewing gum, time
if perilymphatic fistula (sensorineural hearing loss and vertigo) refer to ENT
Acoustic neuroma
vestibular schwannoma
Schwann cell tumors, arise from vestibular portion of CN VIII, slow growing, could result in facial nerve palsies due to compression
clinical presentation of Acoustic neuroma
unilateral sensorineural hearing loss and tinnitus, may cause gait disturbance or other CN involvement
Dx of Acoustic neuroma
audiometry as initial screening test
mri
Treatment of Acoustic neuroma
surgery, radiation, observation due to slow growth
Tinnitus
perception of sound in one or both ears, occurs more in males, associated with depression/anxiety
Tinnitus Etiology
Auditory causes: ototoxic medications, presbycusis, otosclerosis, vestibular schwannoma, chiari malformations, barotrauma,
pulsatile: vascular etiology
Ototoxic medications
aminoglycosides
presbycusis
SN hearing loss w/ aging
otosclerosis
hereditary disorder where bones fuse together
Chiari malformations
cerebellar tinnitus are lower than usual
Pulsatile tinnitus is most commonly caused by?
vascular etiology
Dx of tinnitus
Hx, Physical perform a complete head and neck exam, Auscultate for bruits in patients with possible vascular tinnitus, if pulsatile, refer to ENT
Tinnitus Tx
goal is to lessen awareness and impact on quality of life
behavioral therapy, benzos, white noise
Allergic rhinitis
PAROXYSMS OF SNEEZING, RHINORRHEA, AND NASAL OBSTRUCTION, and nasal obstruction, usually accompanied by itchy eyes, nose and palate
“hay fever” aka seasonal, vs perennial which occurs year round
Allergic rhinitis epidemiology
PEAK INCIDENCE IN CHILDHOOD/ADOLESCENCE, one of the most common chronic diseases in the U.S.
Lots of money and time lost due to this
pathophysiology of Allergic Rhinitis
PRODUCTION OF IGE ANTIBODIES triggering an immune response cascade
IgE binds to mast cells carrying histamine, next time exposed to allergen, histamine is released
Clinical presentation of Allergic rhinitis
RHINORRHEA, SNEEZING, NASAL CONGESTION, itchy eyes/nose/palate, postnasal drip, cough, fatigue
Risk factors of allergic rhinitis
FH of atopy, male, atopic triad, serum IgE above 100 before age 6, first born, early use of abs, maternal smoking
clinical evaluation of allergic rhinitis
personal or FH of allergic rhinitis, asthma, or eczema? 2nd hand smoke? potential triggers? impact on pt?
allergic rhinitis physical exam
Eyes: allergic shiners and denie-morgan lines, pale palpebral conjunctiva may be pale, swollen, conjunctival injection
Nose: pale boggy “BLUISH” mucosa, clear discharge, nasal crease
Throat: post-nasal drainage in posterior pharynx, cobblestoning
Ears: serous otitis media
“allergic shiners”
bluish purple rings around both eyes