L-spine 2 Flashcards

1
Q

What is lumbar spinal stenosis

A

disc degeneration in aging population
arthritic changes to lumbar vertebrae and facet joints
thickening of ligamentum flavum
narrowing of the spinal canal and nerve root canals
compression of neural structures

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2
Q

Osteoarthritic spine shows

A

osteophytes

narrowed disc spaces

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3
Q

How do movements affect pain in lumbar stenosis

A

Worse with lumbar extension

Better with leaning forward (makes disc spaces larger)

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4
Q

Clinical manifestations of lumbar stenosis are

A
50+ y/o 
insidious onset 
Low back and leg pain (morning stiffness in low back, radiates to LE) 
Claudication with walking 
Shopping cart sign 
\+/- numbness and tingling
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5
Q

What is NOT common with lumbar stenosis

A

weakness!

although they can have some foot drop with prolonged walking

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6
Q

What does a neuro exam in lumbar stenosis typically look like

A

Normal!
Few will have sensory changes
motor deficits are usually mild, and hard to differentiate between age related changes

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7
Q

Explain Vascular claudication

A
cramping, tightness 
relief w/ standing 
Bruits (low blood supply) 
Absent pulses 
Shiny skin 
Hair loss
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8
Q

Explain neurogenic claudication

A
numbness, aching, sharp 
relief w/ siting flexed 
occasional atrophy and weakness 
*Back pain 
Limited ROM to spine
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9
Q

What imaging diagnoses lumbar stenosis

A

Radiographs (XR)
MRI
-narrowed disc spaces, narrowing of spinal canal

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10
Q

What is Spondylolithesis

A

Forward movement of one vertebrae on top of another

Can occur commonly during growth spurt

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11
Q

What are the degrees of Spondylolithesis

A

1: <25% translation
2: <50% translastion
3: <75% translation
4: 100% translation
(talking about degrees of slippage)

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12
Q

How do you treat Spondylolithesis

A
APAP (not inflammatory process!) 
NSAIDS (caution in long term use 2/2 metabolism in kidneys), Opioids (breakthrough pain) 
Weight loss 
PT
Epidural injections 
Laminectomy, vertebral fusion
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13
Q

What does this scenario describe:
17 y/o varsity presents w/ b/l low back pain, initially left sided but gradually worsening over 3 months. Pain worse w/ activity and constant. No night Sx. Ibuprofen helps take the edge off. Recently started spring lifting program and football drills
PE: No antalgic gait, pain w/ standing or flexion, scoliosis, or kyphosis. Negative straight leg and seated slump test. L1-L5 dermatomes, myotomes and reflexes symmetric b/l. Positive stork test on right

A

Spondylosis!
A defect in the pars interarticularis of lumbar vertebrae (ex: a fracture)
Typically no neuro or radiculopathy Sx
W>M

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14
Q

What is the stork test

A

Have pt balance on one leg, extend, and turn towards lifted leg (side you are testing). You stand behind them for support. Test both sides

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15
Q

Extension tests

A

Facet joints!

If they are inflamed or rubbing, it will cause pain

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16
Q

What can cause fracture of the interarticularis pars

A

Overloading with load bearing, upright, and weight bearing activities
Trunk extension/hyperextensino
Extension and rotation (why we use the stork test!)

17
Q

What sports are high risk for Spondylosis

A
classic ballet 
Gymnastics 
Figure skating 
Football linemen 
Diving
18
Q

What symptoms indicate we should be concerned about spondylosis

A

Pain persists

Can progress to spondylolithesis (forward movement of one vertebrae on another)

19
Q

How can you evaluate Spondylosis

A

Plain XR: Lateral view (most sensitive) Lateral Oblique (most specific), or AP view

20
Q

Lateral oblique view of Spondylosis will show

A

Scotty dog defect!
On this view, you see a dog shape on every vertebrae. But the affected one has a line in it for the fracture, that makes it look like the collar on a scotty dog

21
Q

What can cause a stress reaction

A

Repetitive mechanical stress (trunk extension and rotation)

22
Q

What is the pathology behind a stress reaction

A

Maladaptive response to repetitive stress; Osteoclasts>Osteoblasts so bone remodeling accelerates
-Must r/o bone pathology as cause!*

23
Q

What stimulates osteoblasts

A

weight bearing activity
growth
fluoride
electricity

24
Q

What inhibits osteoblasts

A
No chronic weight bearing 
chronic malnutrition 
alcoholism 
chronic disease 
normal aging 
hypercortisolism
25
Q

What Inhibits withdrawal of osteoclasts (ex promotes growth)

A

weight bearing
estrogentestosterone
calcitonin
adequate vitamin D and calcium

26
Q

What stimulates withdrawal of osteoclasts (ex bone breakdown)

A
no weight bearing 
space travel 
hyperPTH
Hypercortisol
hyperthyroid
estrogen deficiency (menopause) 
testosterone deficiency 
acidosis
myeloma
lymphoma
normal aging
not enough calcium intake
27
Q

Stress reactions lead to

A

Microfractures
Initiation of inflammatory response
bone stress injury causing stress fracture

28
Q

How do you grade stress reactions

A

I: periosteal edema
II-III: varying severity bone marrow edema
IV: cortical fracture line

29
Q

What imaging does a stress reaction warrant

A

MRI!

after an MRI you can get a bone scan which will show black spots where affected

30
Q

How do you treat stress reactions/Spondylosis

A

Activity restrict 1-4 wks
PT during week 5-12 (work on core strength and LE flexibility)
Gradually return to activity during wk 9-12
Full activity after wk 12
Consider brace

31
Q

When would surgery be an option for Spondylosis

A

Grade III-IV spondylolithesis (75%+)