Knee Theory (Manual) Flashcards
List the feature of late stage knee OA.
i) osteophytes
ii_ subchondral cysts
iii) meniscal maceration
iv) subchondral sclerosis
List the functions of knee menisci.
i) shock absorption
ii) joint congruity
iii) load distribution
Outline pathological mensical extrusion.
i) extrudes > 3mm any direction
ii) most commonly medially from tibial plateau
iii) can be caused by MCL disruption or OA (causitve or a result of extrusion)
Renders menisci useless
Which type of meniscus tear is most associated with tibiofemoral degeneration/early OA?
medial meniscus posterior horn radial horn tears
Describe how alignment/biomechanics contribute to medial compartment OA.
Increased perpendicular distance between GRF and center of rotation creates a knee adduction moment. This loads the medial compartment and contributes to OA.
Ipsi hip ABD weakness causes COM to shift away from stance leg and increase KAM as well.
Patellar maltracking can contribute to joint stresses as well.
List the criteria to rule in knee OA for given patient:
Knee pain with at least 3 of the following:
- Age > 50
- Stiffness <30min
- Bony Enlargement
- Palpable tenderness with no warmth
- Crepitus
- Bony tenderness
To confirm OA on imaging, what special features are needed?
- WBing views
2. PF views
Outline some medical management recommendations regarding knee OA.
- PRP is only beneficial for short term pain relief post TKA
- Do not use glucosamine chondroitin
- Corticosteroid injections can offer short term relief
- Hyaluronic acid injections can offer relief possibly even past 12 weeks, may have better safety long term than cortisone
- Topical NSAIDs are best to limit other side effects
Describe the etiology of ITBFS.
At 30 deg of knee flexion, IT band moves from anterior to posterior position and crosses over the femoral condyle. This friction can cause inflammation/pain of the tissue itself as well as a fat tissue underneath.
List some risk factors for ITBS.
i) tibial rotation
ii) hip IR
iii) reduced DF
iv) weak hip ER
v) improper footwear
vi) genu varum
vii) recent increase in training
List the special test for ITBS.
i) Renne’s
ii) Noble’s
iii) Can do Ober’s
Outline the main treatment components for ITBS.
i) Education/modification of training regime or footwear
ii) hip Abductor strengthening
iii) address tight TFL and glute max
iv) hip ER strength
v) functional motor control exercises
vi) deep frictions
Outline risk factors for development of patellar tendinopathy.
i) Jumping sport/ability to jump higher
ii) reduced DF
iii) reduced LE posterior chain flexibility
iv) hard playing surface (ex. cement).
v) weak hip ABD and ER
Note: inferior pole impingement of tendon on MRI
Outline the testing/diagnostic process for patellar tendinopathy.
i) palpation
ii) functional testing (jump, lunge, SL squat etc.)
iii) SL squat on a 25 deg slope
iv) VISA-P special outcome measure
Outline the main treatment components for patellar tendinopathy.
i) addressing ankle mobility deficits
ii) addressing hip rotational( ++ evidence for rotational) and ABD deficits
iii) managing tendon load (therapeutic load) w/ exercises and reducing other activities that play excessive loading
iv) BEGIN with isometric 70-80% MVC at 60 deg flexion 45s for 5 reps
v) eccentrics for the quads and stretching for quads, progress to plyometrics etc.
Mild evidence for shockwave.
Note: avoid deep frictions, no effect
List some instrinsic factors that place one at risk for PFPS.
i) large Q angle
ii) increased navicular drop
iii) female
iv) weak quads, secondarily hams\
v) hip IR and reduced knee flex on landing from jump
vi) weak hip ABD and ER
vii) tight quads and gastrocs