Joint mobilisation Flashcards

1
Q

what are joint mobilizations

A

passive mobilisations that are within patients control

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

importance of joint motion

A

function, movement, nutrition- stimulating synovial fluid to produced- maintains cartilage health and surrounding tissue, cartilage, bone health, soft tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

the effects of immobilization

A

loss of function, decrease ROM, muscle atrophy, soft tissue length change, cartilage/ bony changes, pain states

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

types of joint motion- osteokineamic

A

physiological movement of the bone- accessory motion needed to assist movement
open chain- no weight, closed chain- weight going through bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

types of joint motion- arthokineimic

A

motion between joint surfaces, accessory movements, open and closed chain activities

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

osteokineamic- explained

A

movement of bones rather than the movement of the articular surfaces, occurs about an axis, measurable as angular displacement in an anatomical plane and referred to as anatomical or physiological range, pathological range may be hyper or hypo mobile

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

arthrokinematics- explained

A

the motion occurring between joint surfaces, roll- refers to the rolling of one joint surface on another, one joint partner usually provides a bade for the other to move on, requires adequate capsular laxity, small movements occurring s part of osteokinematic motion- accessory movement, type of movement depends on the shape of the articular surfaces

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

arthrokinematics- roll, slide, spin, rotation

A

slide- this is a pure translatory movement, refereting to the gliding of 1 component over another, spin-(combine roll and slide to keep joint where it is), rotation of a segment about a stationary axis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

concave convex rule

A

when a concave surfaces moves on a stable concave surface, the sliding of the convex articulating surface occurs in the opposite direction to the motion of the bony lever

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

close pack position

A

position in which the joint ligaments and capsule are maximumly taut, in the majority of joints, the position it is also a position of maximum congruence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is the relevance of the close pack position for joint movement/ rehabiltitaion?

A

open pack/ mid-range- have to consider modifying assessment and treatment technique to match the position patients are having symptoms of

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

limitations to joint movement- function

A

intra-articular structures- ACL and PCL, peri-articular structures- ligs- MCL and LCL, soft tissue length, bony congruence, soft tissue approximation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

limitations to joint movement- dysfunction

A

swelling, pain inhibition, psychosocial, spasm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

indications for joint mobilisation

A

restoration of movement to a joint- accessory movement, physiological, finding what has caused damage, specific to joint, pain, spasm, joint soft tissue tightness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

effect of joint mobilisation

A

improve range of active/ passive/ active movement, increasing length of joint soft tissue and structures, relief of pain, decrease muscle spasm, placebo effect

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

if one session restores motion what does it suggest

A

it suggests some alterations to resting muscle tone, or viscoelastic response in surrounding soft tissue

17
Q

pain control mechanisms- central pain control

A

underlying mechanisms- neuromodulation- altering neural input which moderates pain perception at central/ peripheral level- reducing pain, descending inhibition- blocks pain response from above, increases amount of stimulus needed for message to be sent from spinal cord upwards

18
Q

pain control mechanisms- central pain control- PAG

A

periaqueductal grey matter (PAG) in the brainstem to spinal cord
Dorsal PAG- non opioid, noradrenaline, blocks substance P at spinal cord level
ventral PAG- opioid mechanisms, serotonin

19
Q

pain control mechanisms- pain gate theory

A

low threshold, larger diameter, myelinated AB fibres (mechano fibres), decreased response dorsal horn to A and C fibres, mechanoreceptor coming into spinal cord blocks interneuron within spinal cord, stopping nociceptive transmission- doesn’t perfectly work- reduces pain

20
Q

arthrogenic effects

A

osteo-/artho-kineamatic movements, restoration of accessory glide, capsular stretch, soft tissue receptors, increase in joint ROM, influencing deep/superficial joint muscles, dispersal of inflammatory ‘soup’

21
Q

grades of movements- grade 1 and 2

A

1- small amplitude performed at the beginning of range,

2- large amplitude within a resistance free part of range

22
Q

grades of movements- 3 and 4

A

3- large amplitude performed into resistance up to the limit of range
4- small amplitude movement performed well into resistance

23
Q

dosage variations

A

SIN factors, 30 secs up to 2 mins of more- allows period of time to see if things will change, allow constant reassessment, number of sets dependent of SIN factors

24
Q

dosage variations- severity

A

intensity of symptoms, symptoms constant VS intermittent, pain/ night scale, level of required medication, level of function, ability to return to work, low/ moderate/ high

25
Q

dosage variations- irritability

A

the ease with which a patient’s symptoms are increased or produced, based on the patients aggravating and easing factors , irritable VS non irritable, low/ moderate/ high

26
Q

dosage variations- nature

A

tissue mechanics- pathology and stage of healing, pain mechanisms, psychosocial factors, genetics- myogenic, neurogenic, arthrogenic, inflammatory

27
Q

indications for grades- grade 1/2 and 3/4

A

1/2-high severity/ irritability, high VAS, acute condition
3/4- stiffness more than/worse than pain, low to moderate severity/irritability, low levels of pain- VAS (2,3,4)., sub-acute to chronic stage, movement related pain, end range pain

28
Q

contraindications

A

infection, tumor local, acute inflammatory process, fractures/dislocation, RhA acute flare up

29
Q

precautions

A

joint instability, hypermobility, seronegative arthropathies, high severity/ high irritability

30
Q

joint mobilisation summary

A

it is used to restore joint function, they use arthrokinematics motion to restore functional range of movement to a joint, techniques can be varied in direction, range and amplitude to achieve the desired therapeutic effect, they. are postulated to achieve a number of biomechanical and physiological effects