Joint Lameness Flashcards

1
Q

What is idiopathic synovitis?

A

chronic synovial effusion of a joint

unknown cause

no lameness, tenderness, head, or radiograph changes

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2
Q

What are examples of idiopathic synovitis?

A

bog spavin of tarsocrural joint

windpuffs of fetlock

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3
Q

What are the causes of bog spavin?

A

poor tarsal conformation

strain and minor trauma

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4
Q

What are the signs of bog spavin?

A

distention of tarsocrural joint

no lameness

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5
Q

How do you diagnose bog spavin?

A

clinical signs

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6
Q

How do you treat bog spavin?

A

do nothing; drainage (will probably fill back up); intraarticular injection of corticosteroids (Depo-Medrol); bandaging

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7
Q

What is bog spavin?

A

a painful swelling in a horse’s hock joint that’s caused by inflammation and fluid buildup

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8
Q

What are windpuffs?

A

fluid-filled bumps that appear on the lower legs of horses

AKA windgalls

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9
Q

What are the causes of windpuffs?

A

straight fetlocks

heavy work

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10
Q

What are the signs of windpuffs?

A

distention of palmar/plantar pouch of fetlock joint

no lameness

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11
Q

How do you diagnose windpuffs?

A

clinical signs

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12
Q

How do you treat windpuffs?

A

do nothing, pressure wraps; osmotic agents

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13
Q

What are the causes of traumatic arthritis?

A

single or repetitive episodes of trauma

can progress to osteoarthritis (OA)

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14
Q

What might traumatic arthritis include?

A

synovitis - inflammation of synovial membrane

capsulitis - inflammation of joint capsule

sprain - injury of specific ligaments associated with joint

meniscal tears - femorotibial joints

intra-articular fracture

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15
Q

What are examples of traumatic synovitis and capsulitis?

A

carpitis

osselets - fetlock joint

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16
Q

What are signs of traumatic synovitis and capsulitis?

A

effusion, heat

pain on flexion

+/- lameness

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17
Q

How do you diagnose traumatic synovitis and capsulitis?

A

rads, CT or MRI to rule-out articular changes

synovial fluid analysis to rule-out infection

arthroscopy

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18
Q

How do you treat traumatic synovitis and capsulitis?

A

conservative: rest, immobilization, hydrotherapy, liminents, DMSO, NSAIDs, shoeing changes

more aggressive: IA medications, synovectomy (surgical excision of synovial membrane)

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19
Q

What are sprains?

A

stretching or tearing of supporting ligament(s) of joint by forced movement beyond normal range

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20
Q

What are signs of sprains?

A

swelling, pain

lameness

+/- avulsion fracture: piece of bone pulled loose

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21
Q

What are luxations?

A

dislocation of joint

may be complete or parietal (subluxation) - complete - pastern, fetlock, hip, hock joints

most involve ligaments (severe pain), capsule and tendons

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22
Q

How do you treat luxations?

A

casting, surgical arthrodesis, femoral head ostectomy (FHO) for hip

will get OA

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23
Q

What are the causes of meniscal tears?

A

primary or secondary to stifle problems

shifting or shear forces between femur and tibia

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24
Q

What are the signs of meniscal tears?

A

lameness worse with exercise

effusion

positive stifle flexion

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25
Q

How do you diagnose meniscal tears?

A

U/S, rads, arthroscopy

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26
Q

How do you treat meniscal tears?

A

stem cells

arthroscopic removal of torn portion

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27
Q

What is the prognosis of meniscal tears?

A

50% with surgery

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28
Q

What are the causes of intra-articular fractures?

A

traumatic injury (fetlock or carpus)

osteochondritis dissecans (OCD)

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29
Q

What is osteochondritis dissecans (OCD)?

A

failure of endochondral ossification

cartilage/bone fragments or flaps that develop along articular surfaces of joints

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30
Q

What is the most common form of developmental orthopedic disease in horses?

A

osteochondritis dissecans

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31
Q

Where is osteochondritis dissecans found?

A

often along non-weight bearing surfaces

most common - stifle, tarsus, fetlock, and shoulder

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32
Q

What are signs of osteochondritis dissecans?

A

joint effusion

mild to moderate lameness

often bilateral

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33
Q

How do you diagnose osteochondritis dissecans?

A

radiographs

exploratory surgery
arthroscopy or arthrotomy

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34
Q

How do you treat osteochondritis dissecans?

A

conservative - rest, joint supplements

surgery - arthroscopy or arthrotomy

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35
Q

What are subchondral bone cysts?

A

sac-like cavities in bone beneath cartilage surface

filled with fluid or soft material

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36
Q

Where are subchondral bone cysts found?

A

often bilateral

articular or non-articular

common locations - stifle (medial femoral condyle), fetlock, elbow

37
Q

What are the causes of subchondral bone cysts?

A

defect of ossification (developmental)

IA subchondral bone trauma

38
Q

How do you diagnose subchondral bone cysts?

A

rads

nuclear scintigraphy

39
Q

How do you treat subchondral bone cysts?

A

conservative treatment: IA and systemic joint meds

surgical treatment: debridement, injection of corticosteroids in lining, grafting of debrided cyst (bone, chondrocytes, stem cells)

40
Q

What is the prognosis of subchondral bone cysts?

A

good with surgery

41
Q

What is osteoarthritis?

A

AKA degenerative joint disease

progressive deterioration of articular cartilage (with changes in bone and soft tissues of joint)

synovitis and effusion

pain and dysfunction

42
Q

What are the primary types of OA?

A

acute: associated with high-motion joints (carpus and fetlock)

insidious: associated with low motion joints (pastern “ringbone” and tarsus “bone spavin)

43
Q

What are the secondary types of OA?

A

secondary to other identified problems

intra-articular fractures

luxations/sprains

wounds -> septic arthritis

osteochondrosis (OCD or SBC)

44
Q

What is the pathophysiology of primary OA?

A

repeated concussive forces -> thickening and fibrosis of synovial membrane and joint capsule -> breakdown of articular (hyaline) cartilage -> periosteal proliferation and bone formation (osteophytes) -> decreased flexion of joint (decreased range of motion)

45
Q

What are the signs of OA?

A

slight to severe lameness

decreased range of motion

heat, pain, swelling

effusion

positive flexion

46
Q

How do you diagnose OA?

A

radiographs (loss of joint space, periosteal proliferation, osteophytes, enthesophytes, sclerosis)

thermography

nuclear scintigraphy

regional and IA nerve block

47
Q

How do you treat OA?

A

corrective shoeing

medications: NSAIDs, IM PSGAGs, IV hyaluronic acid (HA), IA hyaluronic acid, IA corticosteroids, IA IRAP

surgery: arthroscopic removal of osteophytes, arthrodesis

48
Q

What is ringbone?

A

a degenerative joint disease in horses that affects the pastern (high ringbone) or coffin (low ringbone) joints

49
Q

Where does high ringbone happen?

A

distal PI or proximal PII

50
Q

Where does low ringbone happen?

A

distal PII or proximal PIII

51
Q

What is bone spavin?

A

tarsal OA

common cause of hind limb lameness

involves distal intertarsal and tarsometatarsal joints (often affects dorsomedial surface first)

ankylosis can occur over time –> lameness resolves

52
Q

What are some additional treatment for bone spavin?

A

cunean tenectomy (part of cunean tendon is removed)

surgical ankylosis (drilling)

non-surgical ankylosis (chemical injection)

53
Q

What is sacroiliac disease?

A

involves SI joint and/or adjacent soft tissue structures

54
Q

What are the causes of sacroiliac disease?

A

single event trauma (getting cast)

repetitive injury with performance

55
Q

What are the signs of sacroiliac disease?

A

low grade lameness and poor performance

reduced stride length in one or both hindlimbs

reduced propulsion and engagement

refusal to jump or go downhill

bunny hop at canter

swapping leads or cross cantering

stiff/rigid with lack or lateral flexion in lumbosacral region

behavioral changes, especially when asked to canter

asymmetry of tuber sacrales

gluteal muscle atrophy

palpable pain over tuber sacrales

56
Q

How do you diagnose sacroiliac disease?

A

pain on palpation

local anesthetic

nuclear scintigraphy

thermography

rectal examination

ultrasound (assess dorsal sacral ligaments)

57
Q

How do you treat sacroiliac disease?

A

rest

NSAIDs

muscle relaxers

injection of SI joint/area (corticosteroids, antibiotics)

58
Q

What is septic osteoarthritis?

A

EMERGENCY!

must be diagnosed and treated ASAP to minimize and prevent OA

can destroy joint quickly due to inflammatory mediators and toxins produced by bacteria

inflammatory response contribute to articular degeneration

59
Q

What are the causes of septic OA?

A

adult - trauma allows bacteria into joint

foal - septicemia/hematogenous

60
Q

What are the signs of septic OA?

A

lameness, effusion, swelling, cellulitis,

61
Q

How do you diagnose septic OA?

A

synovial fluid analysis, radiography (lysis)

62
Q

How do you treat septic OA?

A

sterilize joint - systemic and IA antibiotics, regional limb perfusions

allow drainage - lavage with saline and antibiotics, +/- arthroscopy and arthrotomy

restore normal joint environment - IA sodium hyaluronate

63
Q
A

bog spavin

64
Q
65
Q

Describe the evolution of osteoarthritis

66
Q
67
Q
A

meniscal tears

68
Q
A

intra-articular fracture

69
Q
A

osteochondritis dissecans

70
Q
A

top OCD

bottom normal

71
Q

What does OCD look like on a radiograph

75
Q

Describe what a subchondral bone cyst looks like on a radiograph

78
Q

What does a joint affected by osteoarthritis look like?

81
Q

Describe what ringbone looks like

82
Q
A

high ringbone

83
Q
A

low ringbone

84
Q
A

bone spavin

85
Q
A

bone spavin

86
Q
A

bone spavin

87
Q
88
Q