Ischemic Heart Disease I Flashcards
Percent of population that has had an MI
3-4%
MCC of an MI
Atherosclerosis
Pathogenesis of IHD
Supply that does not meet demand - Can be a blockage, or hypotension, or even anemia or bleeding out
IHD continuum
Prinzmetal angina
Stable angina
Unstable angina
Myocardial Infarction: NSTEMI or STEMI
Acute coronary syndrome
When there is plaque rupture and thrombus formation
STEMI, NSTEMI, or Unstable Angina all count
How much blood is getting through in ACS
USA - No occlusion
NSTEMI - Partial occlusion
STEMI - Complete occlusion
Presentation of stable angina
Typical patter with predictability
1-15 minutes persistence
Pain with activity
Presentation of Unstable angina
Unexpected - a change from the patients usual pattern
Pain DOES NOT go away with rest or nitroglycerin
MI is imminent
Continuum of myocyte cell death
Ischemia - As soon as there is a loss of blood flow - hurts but fine
Injury - Some cells will return to normal some won’t
Infarct - Tissue is fully dead and akinetic
Acute infarct
Has happened within 3-5 days
NSTEMI cause and presentation
Subendocardial wall of the LV, Septum or Papillary muscle is infarcted
ST depression or T wave inversion or nothing(won’t do that on a test)!!!!
Non Q wave
STEMI cause and presentation
Transmural MI
ST elevation and Q waves
Label based on area we are seeing it in
ECG tombstoning
Type I MI
Primary coronary event due to rupture of a plaque
Type II MI
Secondary to ischemia d/t oxygen demand and decresed supply - Spasm, embolism, anemia, arrhythmias
Atherosclerosis is NOT THE CAUSE
Type III MI
Sudden cardiac death
Type IV MI
Have had an angioplasty or sent
Type V MI
Associated with CABG
Myocardial stunning
Heart stops working to save itself - restarts on reperfusion