introduction Flashcards

introduction to the course

1
Q

FMD pathogen

A

virus that infects epithelial cells

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2
Q

signs of FMD

A

drooling (excess salivation), loss of appetite, ulceration of tongue and oral cavity

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3
Q

‘wider’ significance of FMD

A

notifiable disease

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4
Q

atrophic rhinitis in growing pigs: pathogen(s)

A

initial colonisation of Bordetella bronchiseptica + infection with Pasteurella multocida

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5
Q

signs of atrophic rhinitis

A

degeneration of osteoblasts, replacement of the bony core of the nasal conchae by fibrous tissue, loss of turbinates, shortening of the snout

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6
Q

secondary effect of atrophic rhinitis

A

loss of immune defences for respiratory diseases –> animal more susceptible to infections of the lower respiratory tract

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7
Q

agents of canine bronchitis (kennel cough)

A

Bordetella bronchiseptica, canine adenovirus type 2, canine parainfluenza virus, canine respiratory coronavirus and canine mycoplasmas

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8
Q

caveat with vaccines against canine/bovine viruses and bacterias involved in pneumonias

A

not 100% effective

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9
Q

cause of meningitis in the pig

A

Streptococcus suis type 2

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10
Q

‘story’ of infection

A

zoonotic bacteria, respiratory pathogen, causes systemic disease results in the organism entering CSF –> often fatal meniigitis

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11
Q

when is the disease most seen in pigs?

A

post-weaned pig

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12
Q

management of the disease

A

sick pigs require antimicrobial treatments, wider consideration of the pigs environment and husbandry measures

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13
Q

Johne’s disease proper description

A

chronic granulomatous inflammtion

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14
Q

pathogen of Johne’s disease

A

Mycobacteriuum avium, a subspecies of paratuberculosis

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15
Q

when are animals mostly infected, when does the disease develop clinical signs?

A

calves often infected before 3 months old, clincal disease in animals usually over 2 years of age

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16
Q

what are the clinical signs and what are they due to?

A

diarrhoea that results from thickened ileum, because bacterium ingested by macrophages in the ileum that recruit further macrophages that thicken the ileum

17
Q

infection of other animals of Johne’s disease

A

organisms shed in the environment via the feces

18
Q

epidemiology of Johne’s disease

A

usually regarded ruminant’s disease but wildlife has been observed to act as a reservoir of infection

19
Q

feline infectious peritonitis what do infected cats develop

A

a vasculitis not necessarily in the peritoneum

20
Q

two forms of the disease

A

wet form= protein rich effusions

dry form= (pyo)granulomas in organs

21
Q

cause of feline infectious peritonitis

A

virus mutation of the less pathogenic feline enteric coronavirus

22
Q

aetiology of feline infectious peritonitis

A

type III hypersensitivity-type reaction in the blood vessels

23
Q

virus defence againts immune defences?

A

survives intracellular killing mechanisms of monocytes

24
Q

outcome of FIP

A

fatal whereas feline enteric coronavirus is not fatal