Intro Lecture Flashcards

1
Q

Name 5 clinical applications for Therapeutic Modalities?

A

1) modulate pain
2) facilitate/inhibit altered skeletal muscle
3) decrease inflammation
4) facilitate tissue healing
5) increase extensibility

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2
Q

In what phase of rehab can US be used?

A

In early stage after an injury: to decrease inflammation/swelling/compression and to facilitate reparative physiological processes

When you need assistance because you cannot perform parts of your intervention plan.

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3
Q

Name an example of how modalities can increase the effectiveness of other interventions.

A

hot pack prior to stretching can aid tissue extensibility

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4
Q

Which modalities can help reduce pain What outcome measures would you use?

A

Cryotherapy
thermotherapy
ultrasound
Electrical stimulation

Pain scales
Functional scales that have a pain component

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5
Q

Which modalities can help increase strength? Which outcome measures would you use?

A

NMES
Biofeedback

MMT
Dynamometry
Functional Tests

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6
Q

Which modalities can help decrease swelling/inflammation? Which outcome measures would you use?

A

Cryotherapy
Compression

Girth
Circumferential measurements
volumetrics

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7
Q

What modality can help increase tissue healing when integument is compromised? Which outcome measure would you use?

A

Electrical Stimulation

Tissue Healing (closure time, wound depth)

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8
Q

Which modalities would you used to increase ROM/flexibility? Which outcome measure would you use?

A

Thermotherapy
Diathermy
Ultrasound

Goniometric measurements

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9
Q

Name contraindications/precautions for modalities?

A
Contraindications:
comprised/impaired/diminished sensation
electronic implants
Precautions:
compromised/impaired/diminished cognition
pregnancy
cancer
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10
Q

What is acute pain?

A

a response to a noxious stimuli that generally lasts less than 6 months and the underlying pathology can be identified.

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11
Q

What is chronic pain?

A

pain that persists beyond the normal time for healing, some say longer than 3 or 6 months.
early id of those at risk (prolonged, severe, disabling acute pain)

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12
Q

What is referred pain?

A

pain that is felt distant to the source
is referred from one joint to another from a peripheral nerve to a distal area of innervation OR internal organ to an area of musculoskeletal origin.

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13
Q

How do you tell if the source of the pain is located in the same area as where it is sensed?

A

musculoskeletal pain in origin varies with position or movement of the painful area

pain caused by other systems generally varies with stress on that system

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14
Q

What else do you want to know if Pt complains of pain?

A
specific location
quality
severity (intensity)
timing/duration
what makes it better/worse
how does it affect function/activity/participation
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15
Q

What’s the current theory of pain reception?

A

Quality of pain depends on the type of tissue from which stimulus orignates and on which of the 2 nerve types transmits the pain. nociceptors –> A-deta fibers or C fibers –> spinal cord.

Intensity of pain is related to the rate of firing.

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16
Q

If pt perceives pain as sharp, pricking, tinging, easy to localize, what is likely the stimulus source?

A

cutaneous noxious stimulation

17
Q

If pt perceives pain as dull, heavy, aching, hard to localize, what is likely the stimulus source?

A

musculoskeletal structures

18
Q

If pt perceives pain as aching by more superficial, what is likely the stimulus source?

A

visceral pain

19
Q

If pt perceives pain as sharp, what is likely the nerve type being stimulated?

A

A-delta fibers

20
Q

If pt perceives pain as dull, long lasting and aching, what is likely the nerve type being stimulated?

A

C fibers

21
Q

What nerve type normally does not transmit pain?

A

A-beta

22
Q

What type of stimuli activate C fibers and how quickly do C fibers transmit signals?

A

mechanical, thermal, chemical

1m/sec

23
Q

What type of stimuli activate A-delta fibers and how quickly do A-delta fibers transmit signals?

A

high intensity mechanical

5-30m/sec bc they are myelinated.

24
Q

What’s the path that pain signals take to brain?

A

C/A-delta fibers in spinal nerve–> dorsal root ganglion –> dorsal horn –>spinal thalamic tracts –> brainstem –> thalamus –> cerebral cortex

25
Q

pain loop

A

nociceptor stimulated –> axon–>T call in dorsal horn –> anterior horn cell –> stimulates muscle fiber contraction –> nociceptor stimulated

26
Q

What are two ways our bodies modulate pain?

A

1) Gait control theory - pain sensation is determined by a balance of excitatory and inhibitory inputs to the T cells in the spinal cord. (nonnociceptive A-beta fibers stimulate inhibitory interneurons)
[how many modalities work]

2) Endogeous Opoid System - opiates like peptides- opiopeptins (endorphins) bind to opiate receptors

27
Q

What should you document?

A
  • modality used
  • area of body treated
  • intervention duration
  • parameters- position of the patient, use of concurrent compression/elevation
  • outcomes- progress toward goals, regression/ complications (change skin appearances, pt response, adverse responses)
  • CPT Codes