Intro Lecture Flashcards
Name 5 clinical applications for Therapeutic Modalities?
1) modulate pain
2) facilitate/inhibit altered skeletal muscle
3) decrease inflammation
4) facilitate tissue healing
5) increase extensibility
In what phase of rehab can US be used?
In early stage after an injury: to decrease inflammation/swelling/compression and to facilitate reparative physiological processes
When you need assistance because you cannot perform parts of your intervention plan.
Name an example of how modalities can increase the effectiveness of other interventions.
hot pack prior to stretching can aid tissue extensibility
Which modalities can help reduce pain What outcome measures would you use?
Cryotherapy
thermotherapy
ultrasound
Electrical stimulation
Pain scales
Functional scales that have a pain component
Which modalities can help increase strength? Which outcome measures would you use?
NMES
Biofeedback
MMT
Dynamometry
Functional Tests
Which modalities can help decrease swelling/inflammation? Which outcome measures would you use?
Cryotherapy
Compression
Girth
Circumferential measurements
volumetrics
What modality can help increase tissue healing when integument is compromised? Which outcome measure would you use?
Electrical Stimulation
Tissue Healing (closure time, wound depth)
Which modalities would you used to increase ROM/flexibility? Which outcome measure would you use?
Thermotherapy
Diathermy
Ultrasound
Goniometric measurements
Name contraindications/precautions for modalities?
Contraindications: comprised/impaired/diminished sensation electronic implants Precautions: compromised/impaired/diminished cognition pregnancy cancer
What is acute pain?
a response to a noxious stimuli that generally lasts less than 6 months and the underlying pathology can be identified.
What is chronic pain?
pain that persists beyond the normal time for healing, some say longer than 3 or 6 months.
early id of those at risk (prolonged, severe, disabling acute pain)
What is referred pain?
pain that is felt distant to the source
is referred from one joint to another from a peripheral nerve to a distal area of innervation OR internal organ to an area of musculoskeletal origin.
How do you tell if the source of the pain is located in the same area as where it is sensed?
musculoskeletal pain in origin varies with position or movement of the painful area
pain caused by other systems generally varies with stress on that system
What else do you want to know if Pt complains of pain?
specific location quality severity (intensity) timing/duration what makes it better/worse how does it affect function/activity/participation
What’s the current theory of pain reception?
Quality of pain depends on the type of tissue from which stimulus orignates and on which of the 2 nerve types transmits the pain. nociceptors –> A-deta fibers or C fibers –> spinal cord.
Intensity of pain is related to the rate of firing.
If pt perceives pain as sharp, pricking, tinging, easy to localize, what is likely the stimulus source?
cutaneous noxious stimulation
If pt perceives pain as dull, heavy, aching, hard to localize, what is likely the stimulus source?
musculoskeletal structures
If pt perceives pain as aching by more superficial, what is likely the stimulus source?
visceral pain
If pt perceives pain as sharp, what is likely the nerve type being stimulated?
A-delta fibers
If pt perceives pain as dull, long lasting and aching, what is likely the nerve type being stimulated?
C fibers
What nerve type normally does not transmit pain?
A-beta
What type of stimuli activate C fibers and how quickly do C fibers transmit signals?
mechanical, thermal, chemical
1m/sec
What type of stimuli activate A-delta fibers and how quickly do A-delta fibers transmit signals?
high intensity mechanical
5-30m/sec bc they are myelinated.
What’s the path that pain signals take to brain?
C/A-delta fibers in spinal nerve–> dorsal root ganglion –> dorsal horn –>spinal thalamic tracts –> brainstem –> thalamus –> cerebral cortex
pain loop
nociceptor stimulated –> axon–>T call in dorsal horn –> anterior horn cell –> stimulates muscle fiber contraction –> nociceptor stimulated
What are two ways our bodies modulate pain?
1) Gait control theory - pain sensation is determined by a balance of excitatory and inhibitory inputs to the T cells in the spinal cord. (nonnociceptive A-beta fibers stimulate inhibitory interneurons)
[how many modalities work]
2) Endogeous Opoid System - opiates like peptides- opiopeptins (endorphins) bind to opiate receptors
What should you document?
- modality used
- area of body treated
- intervention duration
- parameters- position of the patient, use of concurrent compression/elevation
- outcomes- progress toward goals, regression/ complications (change skin appearances, pt response, adverse responses)
- CPT Codes