Intro, Classification, and Paradigms Flashcards

1
Q

Things that play a role in mental health

A

Together, these effect behaviour (ex: someone with social anxiety will hate public speaking):

  1. Emotions
  2. Thoughts (anxiety, self-criticism, obsessions)
  3. Sensations (stress, fatigue)
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2
Q

CAMH (centre for addiction and mental health) statistics

A

•1/5 Canadians per year will experience mental illness
•By age 40, approx 50% of people will experience mental illness
• The leading cause for disabilities in Canada is mental illness
•70% of mental health problems start in adolescence
*Ages 15-24 are the most likely to have mental illness

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3
Q

National Colleges Health Assessment

A

NCHA: a survey of university students (now discontinued in BC) which asked questions about subjective feelings
•Over 50% of students felt overwhelming anxiety and fatigue (not from exercise)
•36% of UBC students were so depressed it was difficult to function
•Factors impacting academic performance: work, stress, sleep difficulties, anxiety, and depression

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4
Q

Video on multiple personality disorder

A

MPD is a social construction: we now call it Dissociative Identity Disorder (DID):
Questions about Gretchen’s experience
•Why does the personality switch happen, how many personalities can someone have, did she experience trauma/brain injury
How to find answers
•Science will be the most important method in class but things such as media can be just as informative
Hypothesis:
•Coping mechanism, genetics?

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5
Q

Where does information on mental disorders come from

A

1) Scientific research: careful systematic observation of the world, creating, testing, and changing hypothesis
•Goals: finds if study is consistent with other observations, tests for confounds, and emphasizes objectivity: not based on our own experiences (bias and inaccuracy) leading to more accurate predictions (principles vs intuition of science)
•Objections: research is often funded by organizations that influence values and results (ex: pharmaceutical companies)
2) Clinical experience
•Historical component
•Clinical experience helps us understand and treat individuals, while being shaped by science
3) Society and culture
•Social environment has impacts on the types of experiences we have (the way we are raised, our culture’s norms)
•Ex: coming to class is a culturally bounded activity

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6
Q

How scientific research, clinical experience, and society and culture work together to impact research

A

Scientific research: valuing peoples’ lives creates ethics/limitations on research
•Ex: interested in the causes of schizophrenia: hypothesis that it could be related to lack of nourishment for a mother during pregnancy, but we can’t conduct studies on this due to ethical limitations
Clinical: respect in clinical settings because this is something we value
Society and culture: different treatments in different places
•Ex: in South Korea, treatment for depression involves placing a patient in their casket so they can fully experience death

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7
Q

Ways to test abnormal behaviour

A
Observational Design 
Experimental Design 
Clinical Population 
Case Study 
Nonclinical Correlational Study 
Randomized Controlled Trial
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8
Q

Observational design

A

Examines one (usually two or more) variables with no manipulation
•Ex: personality traits, psychological symptoms, genes
•Adv: examines many factors that can’t be manipulated, easy to set up (low time/cost)
•Dis: no statistical controls for confounds (uses data to limit the amount of confounds), hard to determine casualty, easy to establish a correlation

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9
Q

Experimental design

A

Examines one or more DVs through manipulation of an IV
•Adv: more statistical control of confounds, stronger evidence for causality
•Dis: can’t examine as many variables

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10
Q

Clinical population (?)

A

Looking at a certain population that has indication or classification of struggling with a clinical problem (psychological disorder)
•Adv: can have a mixed clinical population (?)
•Dis: more of an ethical concern than other measures (ex: getting informed consent)
Two types:
1) Nonclinical population: looking at people in the community as people in nonclinical samples may still meet criteria for relevant clinical disorders
2) Non-human analogues (animal studies):
•Adv: more flexibility on what we can study on animals
•Dis: physiological and psychological differences can have inaccurate representation
•Now there is also computer simulations (?)

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11
Q

Case study

A

In depth observation of one person, usually done in an interview to support data or:
•Standardized tests: given for level of mood, dissociation symptoms, etc.
•Collateral information: from loved ones, coworkers, etc.
•Adv: in depth understanding of whole person (how different factor interact),
generates possibilities for many issues
•Dis: not generalizable, room for experimenter bias/subjectivity
•Ex: Gretchen findings (other personalities know about her, she has no control, scared)

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12
Q

Nonclinical correlational study (?)

A

Observational design of a nonclinical sample to compare them clinical populations
•Ex: finding differences in genes/personality
•Things important in clinical population may be just as prevalent in nonclinical (ex: clinical depression: nonclinical still experience extreme sadness and can provide insight in how to cope)
Examines variables of potential clinical interest such as
•Genes (SLC6A4: for serotonin transporter 5-HTT)
•Personality traits (attachment style)
•Experience/behaviour/attitudes (substance use, sex)
•Clinical symptoms/diagnosis (?)
Adv: larger subject pool, ability to study many factors
Dis: difficult to examine causation/applicability
•Are nonclinical populations a good representation of clinical populations?

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13
Q

Randomized controlled trial

A

Experimental design in a clinical sample where the researcher randomly assigns participants to different groups to test how certain treatments (IV) impact symptoms/other outcomes (DV)
•Types of control groups: placebo (active ingredient is not there: sugar pills vs antidepressants)
•Obj: harder for therapy (what is the placebo for talk therapy), ethics (is it safe to give a placebo to people who are suffering from clinical problems), solved by head to head comparison
Adv: examine effect of manipulation/treatment
Dis: cost, generalizability

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14
Q

Classification advantages

A

The classification of behaviour and experiences are crucial for research and clinical work because
1. Communication costs time and energy (having clear classification saves this)
2. For more efficient communication (scientists sharing findings, clinicians sharing assessments with doctors)
3. Provides a structure to information
(group behaviours into disorders, and disorders into classifications of disorders)
4. Ensures clarity for definitions and concepts (formal nomenclature: system for naming things)
5. Define the scope and domain of the topic (what we focus on in psych 300 vs what is filtered out/irrelevant)
6. Behaviours, thoughts, and feelings are generally continuous (traits are on a spectrum varying in intensity)
7. Treatments are usually yes or no (either we need to provide it or not: discreet)
8. Allows for easier organization of action/response (?)
9. Helps make treatment consistent (DSM)

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15
Q

Classification disadvantages

A
  1. Loss of information (contextual/individual details aren’t as considered)
  2. Prioritizing some information over others (therapist puts certain emphasis on aspects)
  3. Arbitrary/cut off (what do you do with clients right under cut point)
  4. Misapplication of classification labels (stigma around certain disorders such as schizophrenia)
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16
Q

Definitions of abnormal

A
  1. Subjective distress:
    •Ex: social anxiety causing impairment
    •Obj: may not always be applicable to the client (narcissism) or some contexts (car accident) are reasonable distresses
  2. Maladaptive or functional impairment inhibiting a persons’ daily functioning
    •Ex: OCD, ADHD
    •Obj: context dependent (not all ADHD patients feel impaired in the same setting)
  3. Irrationality or unpredictability
    •Irrationality: thinking something bad is happening with no evidence of it
    •Unpredictability: unexpected emotional responses
    •Obj: if someone believes in aliens (even though their is no evidence) it doesn’t mean you have a psychological disorder
17
Q

Definitions of abnormal cont.

A
  1. Danger to self or others
    •Ex: engaging in risky impulsive behaviour
    •Obj: many people who have psychological disorders are not a danger
  2. Statistical deviancy
    •Behaviours that are rare
    •Obj: some things are rare (high IQ) but aren’t psychological disorders
  3. Violation of social norms
    •Obj: context dependent
  4. Causes social discomfort
    •Obj: not everyone with a psychiatric disorder (ADHD) causes discomfort
18
Q

What definitions of abnormal should we rely on

A

Make use of all the considerations (setting, context dependency), and that abnormality changes overtime (social context)
•Ex: DSM used to consider homosexuality as a disorder
•Who makes this distinction (research, mental health professionals)
•Self reports vs expert observers

19
Q

DSM-5

A

Formal classification and nomenclature system established by American Psychiatric Association (5th revision)
•Provides criteria and information about disorders, developed by researchers and clinicians using research evidence
•Made with scientific and pragmatic considerations (how to frame things in a useful way)
•Most used diagnostic system in Canada and US

20
Q

DSM history

A
DSM-I: 1952
DSM-II: 1968
DSM-III: 1980
DSM-III-R: 1987
DSM-IV: 1994
DSM-IV-TR: 2000
DSM-5: 2013
Changes acores symptoms 
 •Larger 
 •More detailed about symptoms 
 •More diagnostic criteria
 •Less focus on psychodynamic (Freud)
 •Greater focus on reliability 
 •Greater focus on a theoretical perspective (less explanation)
21
Q

DSM-5 Diagnosis

A

Focuses on describing presenting symptoms
•Prototypical model: describing all the features of the disorder (prototype) and a patients presentation is compared to the prototype
•Diagnosis using X/Y criteria: measures degree of similarity (ex: must have 5/9)
*Can still receive treatment without meeting criteria (ex: depression treatment)
•Requires significant clinical judgment (don’t diagnose yourself)
•Shaped by/shapes research (DSM is based of participant findings in research)

22
Q

ICD

A

International Classification of Disease (ICD 10 and ICD 11) developed by the WHO
•Covers all diseases with a section on psychiatric disorders
•Similar structure and diagnoses as DSM
•Widely used in Europe and rest of world

23
Q

CCMD and PDM

A

Chinese Classification of Mental Disorders (CCMD-3)
•Made by Chinese society of psychiatry
•Similar structure to DSM and ICD, with variations due to cultural values/context

Psychodynamic Diagnostic Manual (PDM-2)
•Made by various psychodynamic organizations, providing a psychodynamic theoretical organization and understanding
•Complement to DSM
•Three dimensions (PDM-I)
•Dimension 1: personality patterns and disorders
•Dimension 2: mental functioning
•Dimension 3: manifest symptoms and concerns

24
Q

Paradigms

A

Certain overarching perspective taken for observing and understanding a phenomenon
•Different paradigms view the same behaviours in different ways because of the information thats considered important, and what types of theories/models are used
•Information can be exchanged within a paradigm, but across paradigms is limited
•Ex: easy to exchange information with cognitive paradigms, but challenging from cognitive to psychodynamic

25
Q

Purpose of paradigms

A
  1. Determine what methods are emphasized in research
  2. Determine what types of questions we ask in research
  3. How we interpret data and information
  4. What symptoms and experiences are focused on in assessment
  5. What types of treatments/interventions are used
26
Q

Psychological symptoms

A
  1. Kraeplinian perspective (Emil Kraeplin)
    •Each diagnosis represents a specific underlying pathology that is related to a hypothetical dysfunction in the brain
    •The symptoms ARE the disorder
    •Treatment: symptom reduction
  2. Freudian perspective (Sigmund Freud)
    •Psychological symptoms are manifestations of underlying personality (often related to personality organization)
    •Different underlying dysfunctions may produce different or similar symptoms to other dysfunctions
    *Ex: symptoms of depression vs symptoms of social anxiety may have the same underlying issue: selfesteem
    •Treatment: revealing/resolving underlying difficulties to fix symptoms
27
Q

Biological perspectives

A

Psychological difficulties are the result of pathological processes in the nervous and endocrine systems (brain, nerves, hormones)
•Influenced by genes and environment
•Most obvious in concussions or dementia (headaches, mood dysregulation - damage shown physically and emotionally), but it’s the case for all disorders

28
Q

Genetic vulnerabilities

A

People inherit genes from their parents, which make them more/less likely to develop certain symptoms or disorders
•Chromosomes vs DNA vs genes vs alleles
•Polymorphism: genetic variation resulting in different types of individuals among a single species (almost always this)
•Genotype (carried genes) vs phenotype (observable characteristics)
•Gene-Environment interactions
•Ex: diathesis stress model
•Gene-envioenment correlations
•Ex: passive (outside persons control, you are given genes), evocative (genotype make it more likely that an environment will react to you in a certain way), and active (genotype makes you seek out certain environment)

29
Q

Genetic vulnerabilities findings

A

Most psychological features show significant genetic/heritable component
•Almost always polygenic: determined by multiple genes
•Genes do not code for disorder: they code for proteins that make them more or less vulnerable
•Determining phenotype: Interactions of many genes and the environment
•Identifying heritability (there’s similar genes that make someone vulnerable) vs specific genes (looking at a disorder and finding similar genes) vs mechanism (why does that gene have a correlation with that disorder)

30
Q

Neurotransmitters and hormones

A

Imbalance or dysregulation of singling molecules can produce behavioural changes/symptoms
•Neurotransmitters (chemical released to another specific neuron) vs hormones (released by endocrine broadly into bloodstream):
•Both partially implicated in many disorders and the target of all psychiatric medications to fix
1) Dysregulation in molecule synthesis (to much/little) or release (to slow/fast)
2) Dysregulation in molecule reuptake or breakdown
3) Dysregulation in receptor expression or sensitivity

31
Q

Temperament (biological perspective)

A

Temperament is largely genetic and also environmental
•Children show differences in how they respond to environment
•Typical temperaments: fearfulness, irritability, positive affect, activity level, effortful control
•Related to adult personality: neuroticism, extraversion, constraint

32
Q

Psychological perspective

A

Psychological difficulties are the result of interactions in thoughts, feelings, behaviours, and motivations
•Emphasizes interactions Id: primal, ego: middle, superego: perfect
•Disorders arise from these intrapsychic conflicts (ex: desire for social connection vs desire to avoid rejection)
•May not be aware of conflict, but produces distress (anxiety) that you’re likely aware of
•Behavioural symptoms: defences against anxiety (varies in adaptiveness and effectiveness)
•Resolving underlying conflict reduces symptoms

33
Q

Theories involving the psychodynamic perspective

A

NOT discredited and unsupported perspective
Object-relations theory: interaction with internalized representations (objects) of significant people (driven to form relationships with others)
Interpersonal perspective: motivation/desire connect with and individuate from other people
Attachment theory: early life relations with caregivers shape adult relations

34
Q

Behavioural perspective

A

Emphasize behavioural modification from learned experience
•Disorders arise from maladaptive learning processes (ex: did not learn adaptive patterns or learned patterns that are no longer helpful)
•Maladaptive behaviours (symptoms) are the disorder
•Treatment: reshaping learned behaviours
NOT cold dehumanizing approaches that are especially effective for anxiety:
1) Classical conditioning: learned response to conditioned stimulus that consistently follows unconditioned stimulus
2) Operant conditioning: learned response to stimuli due to subsequent reinforcement (conditioned avoidance response)
3) Observational learning: learning from seeing behaviour from others

35
Q

Cognitive perspective

A

Emphasizes thoughts, beliefs, and information processing
•Disorders rise from unrealistic perspectives of self, others, events, and the world (unrealistic thinking leads to unnecessary negative emotions/unhelpful behaviours)
*Ex: driver cuts me off, appraisal that he wants to kill me
•Unrealistic appraisals reflect maladaptive underlying schema about others, ourselves, and the world
•Treatment: developing a new way of thinking to reduce symptoms (NOT about learning new information, but learning new approaches)
1) Attribution processes: how people assign cause/blame for things that happen
•Stable (is it consistent) vs unstable (changeable)
•Internal (my fault) vs external (environment’s fault)
2) Evaluate the evidence: maladaptive thoughts and beliefs can be challenged by objective examination of evidence