INTRACRANIAL PRESSURE Flashcards

1
Q

pressure exerted by cranial contents against the skull

A

intracranial pressure

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2
Q

cranial contents of the brain

A

brain
blood
csf

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3
Q

normal ICP

A

10 to 20 mmHg

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4
Q

the pressure that pushes oxygenated blood into the brain

A

cerebral perfusion pressure

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5
Q

normal CPP

A

70 to 100 mmHg

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6
Q

how does ICP affects CPP?

A

CPP decreases when ICP increases

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7
Q

priority nsg dx for increased icp

A

ineffective cerebral tissue perfusion

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8
Q

early or late: changes in LOC

A

early

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9
Q

early or late: headache/dizziness

A

early

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10
Q

early or late: seizure

A

can be both

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11
Q

early or late: changes in speech

A

early

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12
Q

early or late: vomiting

A

can be both

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13
Q

swelling of the optic disc

A

papilledema

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14
Q

early or late: papilledema

A

early

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15
Q

early or late: cushing’s triad

A

late

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16
Q

cushing’s triad s/sx

A

inc. systolic pressure
dec. pulse
dec. respiratory rate

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17
Q

early or late: fever

18
Q

early or late: pupillary change

19
Q

early or late: impaired eye movement

20
Q

early or late: decerebrate

21
Q

early or late: decorticate

22
Q

early or late: flaccid

23
Q

early or late: dec. motor function

24
Q

indication of 8 and below GCS scoring

A

possible brain damage

25
a concept where how the body compensates when ICP is increased
monro-kellie hypothesis
26
what is the first compensatory mechanism according to monro-kellie hypothesis?
displacement of CSF to decrease ICP
27
how does displacement of CSF occurs when there is increased ICP?
some of the CSF will go to the spinal cord to decrease the pressure in the brain
28
what is the second compensatory mechanism according to monro-kellie hypothesis?
decrease blood in the brain
29
what is the first blood to be decreased?
venous blood
30
where will the venus blood go so that pressure in the brain will decrease?
heart
31
what is the third compensatory mechanism according to monro-kellie hypothesis?
increasing systolic pressure
32
what is the last compensatory mechanism according to monro-kellie hypothesis?
brain herniation
33
why does the pulse and respiratory rate decrease during brain herniation?
herniation causes compression on brainstem where cardiovascular and respiratory center is located
34
best position for patient with increased ICP
low fowlers head and neck aligned
35
why is low fowler's position ideal for patients with increased ICP
to promote venous return to the heart
36
why do we need to give O2 to patients with increased ICP
to help cerebral oxygenation
37
osmotic diuretic needed in patients with inc. ICP
mannitol
38
why do we need to do transient hyperventilation in patients with inc. ICP?
to decrease PCO2 to decrease ICP
39
ways to reduce brain metabolism
manage fever low dose sedative
40
why do we need to reduce brain metabolism in patients with inc. ICP?
because inc. metabolism = inc. need of oxygen which compromises cerebral perfusion