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FINALS RESP > interstitial lung disease > Flashcards

interstitial lung disease Flashcards

1
Q

causes upper zone fibrosis

A

CHARTS // coal workers // hypersensitivity (EEA) // ank spondy // radioation // TB // silicosis + sarcoid

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2
Q

causes lower zone fibrosis

A

Idiopathic // SLE // drug induced // asbestosis

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3
Q

common drugs causing fibrosis

A

amiodarone // bleomycin // methotrexate // nitrofurantoin // dopamine agonists

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4
Q

who gets IPF

A

men 50-70

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5
Q

features IPF

A

exertional SOB // bibasal fine end insp crepitations // dry cough // clubbing

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6
Q

spirometry IPF

A

FEV1 normal // FVC decreased // FEV1/FEV1 increased

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7
Q

gas exchange IPF

A

reduced transfer factor

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8
Q

imaging IPF

A

CXR = ground glass or honeycombing // CT = gold standard

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9
Q

what antibodies may be seen in IPF

A

ANA or RF

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10
Q

mx IPF

A

rehab // O2 + transplant // poor prognosis 3-4 years

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11
Q

how long does coal workers pneumoconiosis take to develop

A

15-20 years

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12
Q

what is simple coal workers pneumoconiosis

A

most common + asymptomatic // may increase risk of COPD // can progress

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13
Q

staging simple coal workers pneumoconiosis

A

cat 1 = some opacity but normal lung markings // cat 2 = large opacities + normal markings // cat 3 + large opacities + abnormal lung markings

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14
Q

what is progressive massive fibrosis coal workers

A

round fibrotic masses in upper lobes // SOB on exertion + cough

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15
Q

invx massive fibrosis coal workers

A

CXR = upper zine fibrosis // spirometry = mixed obstructive + restrictive eg normal or reduced FEV1 and reduced FVC

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16
Q

what causes silicosis

A

fibrosis from inhaled silica eg mining, slate, foundy, pottery

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17
Q

features silicosis

A

upper Zone fibrosis // egg shell calcification

18
Q

what infection are silicosis pts at risk of

A

TB

19
Q

range of lung disease from asbestos

A

plural plaques // pleural thickening // asbestosis // mesothelioma

20
Q

what are pleural plaques

A

common form of asbestos lung disease // benign with no metastatic potential

21
Q

what affects severity of asbestosis

A

length of exposure

22
Q

features asbestosis

A

SOB on exertion // clubbing // end insp crackles // restictive + reduced TCLO

23
Q

most common cancer assoc with asbestos

A

lung cancer

24
Q

features mesothelioma

A

progressive SOB, chest pain, pleural effusion, clubbing

25
Q

what type of malignancy is mesothelioma

A

pleural // right lung more common

26
Q

invx mesothelioma

A

CT // thoracoscopy

27
Q

mx mesothelioma

A

chemo +/- radio or surgery // 8-14 months

28
Q

what type of conditions is Extrinsic allergic alveolitis (EAA)

A

hypersensitivity type III

29
Q

types of EAA

A

bird fanciers // farmers // malt workers // mushroom workers

30
Q

presentation EAA

A

acute = SOB, dry cough, fever // chronic = lethargy, SOB, cough, anorexia + weight loss

31
Q

invx EAA

A

CXR = upper fibrosis // bronchoalveolar lavage = raised lymphocytes // IgG

32
Q

mx EAA

A

avoid triggers // oral steroids

33
Q

type of disease sarcoidosis

A

type IV sensitivity –> non-caseating granuloma

34
Q

who gets sarcoidosis

A

young black women

35
Q

assoc conditions sarcoidosis

A

Lofgens // mikulocz (parotid) // Heerfordts (parotid + fever)

36
Q

features sarcoidosis

A

erytheme nodosum // bilat hilar lymph // swinging fever // polyarthalgia // SOB // cough // malase // lupus // hyperCa!

37
Q

poor prognosis sarcoidosis

A

insidious onset >6months // NO erythema nodosum // extrapulm features // black // CXR stage III-IV

38
Q

bloods sarcoidosis

A

raised ACE + raised Ca

39
Q

CXR staging sarcoidosis

A

1 = bilateral hilar lymph (BHL) // 2 = BHL + infiltrates // 3 = diffuse intsertitial infiltrates only // 4 = diffuse fibrosis

40
Q

mx sarcodoisis

A

steroids

41
Q

indication mx sarcoidosis

A

CXR 2-3 AND symptomatic // hyperCa // eye, heart, neuro involvement

FINALS RESP (12 decks)

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