Insects & Flies & Bugs Flashcards

1
Q
  • What is the species and genus of the common housefly?
  • What are its (2) distinguishing features?
A
  • Musca domestica
  1. Four dark stripes on grey thorax
  2. Wing vien 4 bends to meet vein 3 very closely.
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2
Q
  • What is the species name for Stable flies?
  • What size are they?
  • How are they disinguished (2 features) from Musca domestica?
A
  • Stomoxys
  • 8 mm long, about the size of a large housefly
  • grey colour, 4 dark stropes on thorax and several dark spots on the top of the abdomen
  • Differentiating Stomoxys from Musca:
    1. proboscis
    2. wing venation: 4th vein turns up towards 3rd but does not approach so closely
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3
Q
  • Name stages of development of house fly
  • How long to complete llife cycle?
A
  • stages
    1. egg
    2. larva or maggot
    3. pupa
    4. adult
  • 7-10 days
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4
Q
  • What is the family name for non-metallic blowflies?
  • Other common names?
  • How distinguished?
  • What is distinct about life cycle?
A
  • Sarcophagidae or Wohlfartia (flesh flies)
  • Distinguishing features:
    • non-metallic, usually grey or silver in colour
    • thorax 3 longitudinal stripes
    • abdomen marked with “chess-board” (Sarcophagidae) or well-defined dark spots (Wohlfahrtia).
    • eyes red colour
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5
Q
  • What are scientific and common names for metallic blowflies?
  • Distribution?
  • What is their usual behaviour and clinical significance?
  • What are their distinguishing features?
A
  • Calliphora (bluebottles) and Lucilia (greenbottles)
  • worldwide distribution
  • normally lay eggs on rotting meat etc, sometimes also on purulent wounds and excreta. Presence of larvae does not usually lead to serious consequences as they feed on pus and dead tissues.
  • Distinguishing features: blue or green metallic colour on thorax and abdomen, prominent bristes on dorsal surface of thorax (Chryosomia & Cochliomyia - screw worms - lack these)
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6
Q
  • What are screw worms?
  • What is their life cycle?
  • Identification of flies and larvae?
  • What diseases
A
  • larvae of Cochlyiomyia hominovorax (new world screw worm) and Chrysomya bezziana (old world screw worm)
  • obligate parasites of living tissue that invade natural orifices, nose, mouth, eyes, vagina resulting in serious tissue destruction and pain
  • blue-green metallic colour of adult flies
    • no bristles on thorax)
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7
Q
  • Screw worms: life cycle and disease
  • Morphology?
  • Distinguish from what?
A
  • larvae of Cochlyiomya hominovorax (new world screw worm) and Chrysomya bezziana (old world screw worm)
  • eggs laid directly on skin, give rise to larvae that are
    • obligate parasites of living tissue that invade natural orifices, nose, mouth, eyes, vagina resulting in serious tissue destruction and pain
  • larvae have rows of stout spines like a screw thread
  • distinguish from
    • Dermatobia (human bot-fly)
      • larvae have hooks on each segment
    • Cordylobia (tumbu-fly or mango-fly)
      • with smaller spines or spicules over dorsal surface
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8
Q
  • What are Cordylobia and Dermatobia?
  • What disease do they cause?
  • What are the distinguishing features of the larvae and from what should they be distinguished?
A
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9
Q
  • distinguish between screw worms , tumbu-fly or mango-fly and human bot-fly
A
  • Screw worms:
    • larvae of Cochlyiomyia hominovorax (new world screw worm) and Chrysomya bezziana (old world screw worm)
    • obligate parasites of living tissue that invade natural orifices, nose, mouth, eyes, vagina resulting in serious tissue destruction and pain distinguish from
    • flies causing furuncular myiasis:
      • (Dermatobia (human bot-fly)
        • larvae have hooks on each segment
      • Cordylobia (tumbu-fly or mango-fly)
        • with smaller spines or spicules over dorsal surface
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10
Q
  • What is Musca sorbens?
  • What is it’s life cycle and preferred food?
  • Clinical significance?
  • Appearance?
A
  • Musca sorbens is the
    • bazaar fly or bush fly
    • it lives outdoors and in the open
    • lays eggs on human (preferred) or animal excreta in the open, not in pit latrines
    • most frequent of flies seeking eyes, more attracted where discharge from eyes and/or nose
    • may be common in refugee camps
    • major transmitter of trachoma
  • difficult to distinguish from Musca domestica
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11
Q
  • Musca domestica
    • life cycle?
A
  • feeds on human food, garbage, excreta, decayed plant and animal matter
  • feeding sites 100-500 m from breeding sites, often in same decaying organic matter it feeds on
  • found by day near food sources and breeding sites
  • 7-10 days
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12
Q
  • What is the medical significance of the common house fly? i.e. transmits which diseases?
A
  • omnivorous, feeds on feces and food
  • transmits
    • Enteric bacteria
      • Shigella
      • Salmonella
      • E. coli
    • Cholera
    • Enteroviruses, inc. polio
    • ¿Helminth eggs?
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13
Q
  • How does the common housefly transmit disease?
A
  1. defecation (every 5 minutes)
  2. regurgitation
  3. transport on body surface
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14
Q
  • What is the Lesser House Fly?
  • What is distinctive about it’s appearance?
A
  • Fannia cannicularis
  • worldwide distribution
  • smaller than the common housefly (3.5-6 mm length), does not have same 4th vein pattern
  • otherwise similar in behaviour and clinical significance
  • likes chicken farms
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15
Q
  • what kind of community control measures can be applied to control of houseflies and blowflies?
A
  • although effective, spraying with insecticide is
  1. expensive
  2. leads to resistance
  • therefore good waste disposal practice
    • cover refuse with >25 cm soil
    • keep only working edge of tip exposed
    • make sure tip is tightly packed so increased temp kells larvae
  • Disposal of human and animal excreta
    • ventilated pit latrine
    • manure piles
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16
Q
  • name four flies that are obligate parasites and two that are facultative parasites
A
  • Obligate parasites
    • furuncular boil-like swellings with one fly larva per boil
      • Tumbu-fly or mango-fly (Cordylobia)
      • human bot-fly (Dermatobia)
    • invasive (screw-worms)
      • Cochlyiomyia hominovorax (new world screw worm)
      • Chrysomya bezziana (old world screw worm)
  • Facultative parasites
    • Non-invasive
      • Lucilia (Green bottles)
      • Calliphora (Blue bottles)
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17
Q
  • What is this?
  • What is latin name?
  • What does it look like? where found?
  • Life cycle?
    • What is normal host?
  • lesions
A
  • Tumbu fly larva
  • Cordylobia anthropophaga
  • Adult is inconspicuous brown coloured fly
  • Africa
  • Normal host is rat
  • lays eggs on urine soaked clothes or sandy soil
  • grub-shaped larvae burrow into skin to form boil-like swellings
  • common around buttocks, genitalia and back
  • emerge from skin after 8 days and fall to ground, where they pupate in soil
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18
Q
  • Cordylobia:
    • common name?
    • how treated?
    • how prevented?
A
  • common name tumbu-fly
  • cover surface of furuncle with liquid paraffin
    • this deprives larva of oxygen
    • the paraffin coats the larva, which may then be able to be expelled with pressure applied to the sides of the furuncle
  • conrolled by ironing oclothes
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19
Q
  • Dermatobia
    • common name?
    • life cycle?
A
  • Dermatobia hominis
  • human bot-flye or New World myiasis
  • eggs attached to biting insect
    • mosquito or Stomoxys
    • larvae hatch and drop onto skin
    • burrow into skin forming boil-like swelling
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20
Q
  • What are the identifying characteristics of Dermatobia?
  • (what’s the disease and how transmitted)
A
  • stout rose-thorn spines
  • shape, esp of 2nd instar
  • New World Myiasis
  • dermatobia eggs attach to mosquito and are laied on human hosts
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21
Q
  • What are fleshflies?
  • Common name?
  • where found?
A
  • Sarcophagidae
  • common in refuse, butchers shops etc
  • larvae of some occasionally cause myiasis (facultative parasite)
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22
Q
  • What is non-invasive myiasis?
  • fly habitat?
  • What is it caused by?
  • substrate?
  • therapeutic use?
  • Identify the larvae
A
  • caused by bluebottles (Calliphpra) and green bottles (Lucilia)
  • usually breed in similar habitats to houseflies
  • will infest and consume necrotic tissue e.g. foul smelling wounds, ulcers etc, sometimes found under bandages
  • usually do not cause harm since they feed on dead and decaying tissue
  • Baer’s maggot therapy
  • Larvae without screw thread of spines
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23
Q
  • Dermatobia treatment?
A
  • vaseline strategy may be insufficient
  • may require surgical intervention or injection of anesthetic into base
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24
Q
  • Non-furuncular myiasis treatment?
A
  • remove all visible larvae
  • revisit wound daily to check for emergent larvae
  • risk of secondary infection
  • surgery may be required if extensive tissue damage
  • systemics: oral ivermectin
25
Q

Which flies are obligate parasites?

Which flies are facultative parasites?

Which flies are accidental parasites?

A
  • Obligate myiasis
    • screworms
      • new world: Cochlyomya hominovorax
      • old world: Chrysomya bezziana
    • human bot fly: Dermatobia
    • tumbu fly or mango fly: Cordylobia
  • Semispecific myiasis (Facultative parasites)
    • usually lay eggs in decomposing animal tissue or vegetable matter, though live hosts can also serve as hosts
    • Myiasis would usually be found in already existing wounds, and blood and foul discharge often serve as an attractant for the insects.
    • Most of the flies responsible for semispecific myiasis come from the family Sarcophagidae. Common examples include
      • Lucillia (green bottle)
      • Calliphora (blue bottle)
      • Wohlfartia
  • Accidental myiasis
    • occurs when flies that do not usually feed on living flesh (e.g. musca domestica, fannia) lay eggs on an open wound
26
Q
  • Control of which vectors would improve with general sanitation (i.e. provision of latrines, waste disposal and water disposal)?
A
  • Musca domestica
  • Culex mosquitoes
    • ​eg Culex quinquefasciatus​
    • ​Vector for Japanese encephalitis, Lymphatic filiariasis, West Nile Fever
  • Culex quinquefasciatus, the urban mosquito, breeds predominantly in polluted water in domesticated situations such as pools, drains, canals and septic tanks. Distribution of C. quinquefasciatus is increasing with urbanisation and human activity due to poor sanitation. Aedes species prefer to breed in unpoluted collections of water. Tsetse flies are not common in domestic urban settings.
27
Q

What are these?

Characteristic features.

What diseases do they transmit?

A
  • males scutum covers whole of dorsal surface, may be patterned
  • females resitricte to anterior part of dorsal surface
    • when fully fed may be hard to see in females
  • Diseases
    • Lyme disease - Borrelia burgdorfi
    • Q fever - Coxiella burnetti
    • Colorado tick fever - Coltivirus
    • Rocky Mountain Spotted Fever - Rickettsia rickettsii
    • African tick typhus (African Spotted fever) - R. africae
    • Mediterranean Spotted Fever - R. conorri
    • Tularemia - Francisella tularensis
    • Tick paralysis - tick toxin in Australia
    • Tick Borne Encephalitis - flavivirus in Europe and Russia
  • (Tick-borne relapsing Fever - Borrellia species) is transmitted by Argasidae - Soft Ticks
28
Q

What is this?

Distinguishing features?

What disease?

A
  • Argasidae - soft ticks
  • No scutum
  • Tick borne relapsing fever - Borrellia species
29
Q

What are these?

Distinguishing characteristics?

A
  • Soft Tick Larvum on left; Hard Tick Larvum on right
  • both have only 6 legs (adults have 8)
  • only the Hard Tick Larvum has a scutum (as do adults)
30
Q

What is this?

Distinguishing characteristics?

What is the disease?

What is the life cycle relevant to the disease?

A
  • Leptotrombidium or Chigger mite, vector of scrub typhus (see eschar with black crust and erythematous halo, picture below)
  • only larval stage feeds on blood
  • larval mite has 6 legs and many fine hairs
  • normally larva fees on small mammals or ground feeding birds, humans are accidental host
  • pathogen similar to Rickettsia but is called Orienta tsugumushi
  • areas of forest clearings, riverbanks, grassy regions best for infected mites
  • more frequent in rainy season
31
Q

What is this?

What might blood test show?

A
  • microscopic scabies mite
  • burrows into upper layer of skin and lays eggs
  • eggs hatch and burrow through skin, may be found at end of burrow
  • Eosinophilia, esp in immunosuppressed with hyper infestation (Norwegian scabies)
32
Q

What is this?

Vector for?

A
  • adult flea
  • vector for
    • bacteria, viruses and tapeworms, as well as
    • Endemic typhus (murine fever) - Rickettsia typhi
    • Plague (Yersinia pestis)
    • Tularaemia (Francisella tularensis) - also hard ticks
33
Q

What does this picture represent?

A
  • The difference between male and female fleas.
  • female left, male right
  • the difference is presence of clasper on the male
34
Q

What are these?

ID the species?

What are the distinguishing characteristics?

A
  • Fleas:
    • Ctenocephalides felis (Cat flea) on left, similar to dog flea (C. canis)
    • Xenopsylla cheopsis (Oriental rat flea) middle
    • Pulex irritans (Human Flea) right.
  • Tell C. felis or canis from the other two by presence of two combs (genal and pronotal)
  • Tell Xenopsylla from Pulex by the presence of a meral rod on the 2nd thoracic segment.
  • Xenopsylla or Oriental rat flea (no compbs, meral rod on 2nd thoracic segmant) was vector for the plague bacillus, Yersinia pestis
    • ​bacillus multiply in flea blocking upper GI tract of flea causing it to regurgitate bacilli as it tries to take a blood feed
  • Pulex irritans (no combs or meral rod) may transmit Rickettsia typhi, which causes murine typhus.
    • ​bacteria grow in epithelial cells lining flea’s gut wall and are excreted in feces, then scratching rubs bacteria into wound.
35
Q
  • What is this?
  • How can you tell?
  • What disease does it carry?
  • What is the natural reservoir for this disease?
  • Where is it found?
A
  • Pulex irritans, human flea
  • no combs (not cat or dog flea or Ctenocephalides)
  • no meral rod on 2nd thoracic segment ( not oriental rat flea or Xenopsylla)
  • vector for murine (rodent) typhus, which is a disease - duhhh- of rats
  • which are found in warm coastal areas around the world because they are looking for ships they can get onto to jump off of when they sink - (anybody still reading this? If so, see map)
36
Q

What is this?

What disease does it case?

How is it treated?

A
  • Tunga penetrans or Jigger flea (smaller male on right, female on left that has been dug out of someone’s foot)
  • Causes tungiasis or jiggers, which can really be pretty nasty - see picture attached (ddx mycetoma, differentiated because mycetoma is painless whereas tungiasis causes intense itching, pain and inflammation and may get secondarily infected)
  • usually affects the foot but can get anywhere on the body
  • treatment is by careful physical extraction of the intact female
37
Q
  • What is this?
  • What disease are they a vector for?
A
  • body lice (Pediculosis corporis), compare with head lice (Pediculosis capitis) below, which are very hard to tell apart except by context (see picture of P. capitis and egg below)
  • Lice have a hemimetabolous life cycle, which means there is no pupal stage: the larvae hatch directly from egg
  • body lice are vectors of
    • Epidemic or louse-born typhus (Rickettsia prowazekii) and
    • Trench fever (Bartonella quintana)
    • Both are spread by inhalation of louse feces or bacteria in feces being rubbed into an abrasion, not by the bite.
  • also vector for Epidemic relapsing fever (Borrelia recurrentis)
    • louse must be crushed first to infect humans: spirochetes enter through mucosa or via cuts and abrasions
38
Q

What are the vectors and organisms for these diseases?

A
39
Q

What is this?

A

Pubic lice

Pthirus pubis

40
Q

What are the two species of Aedes are the most significant as disease vectors?

For what diseases are they vectors?

When do they bite?

What is the main behavioural difference between them?

A
  • Aedes aegypti & Aedes albopictus
  • Dengue, yellow fever, zika, chikungunya, Rift Valley fever
  • Experimentally a wide variety of other diseases
  • Aedes aegyptus feeds only on humans (anthrophilic only); A. albopictus feeds on a wide range of hosts.
  • daytime, mainly for two hours after sunrise and 2 hrs before sunset.
41
Q

What methods have been tried for control of Aedes for dengue control?

A
  • Source reduction:
    • community mobilization, education and incentive programmes to get rid of cans, tires, open water containers
  • Larviciding
    • larvicidal fish for rice paddies
    • mesocyclops for essential water containers
    • larvicides for water containers: Temephos ‘Abate’, organophosphate; new ‘safer’ treatments including Bti or Pyrifoxen
    • filling in draining periurban collections of water
  • Response to outbreaks by
    • outdoor fogging or spraying but no indor residual effect and repeated treatments required
    • most larvae don’t survive to adulthood anyway so rel ineffective
  • indoor residual spraying
42
Q

Dengue vector control Evidence

What works?

What doesn’t?

What makes things worse?

A

2016 Review PLOS Neglected Tropical Diseases, McCall et al

  • in general very little evidence regarding efficacy but based on few studies the following appeared to
  • reduce the risk of dengue
    • house screening
    • community based environmental management and water covers
    • indoor residual spraying
  • no impact
    • repellants
    • insecticide-treated bed nets (LLIN’s)
    • mosquito traps
  • possible negative impact ?due to overconfidence, increased biting risk?
    • knockdown/aerosol sprays
    • mosquito coils
  • unevaluated
    • fogging/space spraying
    • larvicidal fish
    • larvicidal copepods (mesocyclops)
43
Q

Promising technologies for control of Dengue vectors

(Reasons for Optimism)

What are they?

benefits?

A
  1. New insecticide pyriproxyfen (Sumilarv), acts on insect juvenile growth hormone ecdysone promoting dev of larvae into pupae (coincidentally also important in triggering growth from rhabitiform to filariform hormone in strongyloides).
    • Pros:
      • safe to vertebrates
      • effective at low doses (ppb)
      • long lasting, slow release
      • autodissemination: mosquitoes transfer to other breeding sites
      • tasteless, colourless, odourless
    • Cons:
      • caught up in conspiracy theory and accusations that it, rather than Zika is cause of microcephaly in Brasil
  2. Reduce vector biting by house screening , supp by sev small studies as efficacious
    • Pros: who knew? low tech
  3. Some evidence for effetiveness of indoor residual spraying for Ae. aegypti control.
  4. Evidence for effectiveness of community mobilisation from studies in Nicaragua (The Camino Verde Intervention) and Mexico demonstrating reduced dengue sero-conversion following community wide mobilization and cooperative education campaigns to “deny the mosquito a home.”
  5. Emerging Technologies
    1. RIDL: Release of Male Insects carrying a Dominant Lethal Gene
    2. Using Wolbachia symbiont infections to shorten mosquito lifespan and limit infection with dengue, Chikungunya and Plasmodium.
44
Q

What is this?

What other member of this order is medically significant?

A
  • Bedbug, one to two medically significant “True Bugs” order Hemiptera
  • The other is Triatomine Bug
45
Q

What is a Gomez-Nunez Box?

A
  • a triatomine trap used for sampling insect density relevant to Chagas Disease. Essentially an open cardboard box left in a house. Presence of bugs, eggs or feces may then be detected.
46
Q

What methods of vector control may be used for Chagas Disease?

A
  • Control of Triatomines
  • Residual spraying. Primarily pyrethroids • DDT/pyrethoid resistance
  • 3 treatments/year=$25
  • Malaria spraying effective
  • Improvement in housing???

Repairing/ re-plastering

Replace thatched roofs (many spp. lay eggs in palm trees)

Separation of animals from residence
• Transgenic control using symbionts?

47
Q

Contrast vectors for Gambian and Rodesian trypanosomiasis.

A
  • The ecology of riverine tsetse (Palpalis group)
    • Species from this group are generally less mobile, moving ~300 m/day.
    • Restricted to riverine vegetation.
    • Feed on a wide range of hosts such as lizards, livestock and humans.
    • Generally active in the middle of the day
  • The ecology of savanna tsetse (Morsitans Group) - East and South Africa
    • Species from this group are highly mobile, moving 500-1000 m/day.
    • Generally active in the late afternoon.
    • Feed on a restricted range of hosts such as warthog and buffalo, and cattle in farming areas; humans are rarely bitten.
    • Range widely across woodland.
48
Q

What are the features of the life cycle of the vector for African Trypanosomiasis that make it susceptible to control.

A
  • The life cycle of tsetse
  • Slow reproductive strategy

..so we only have to kill a few (~4%/day ) to eliminate a population.

  • • A female produces a single egg which develops into a larva within her uterus.
  • Every nine days later, the mother produces a larva which burrows into the ground where it pupates.
  • The adult fly emerges from the pupa in the ground after about 30 days.
  • 50 days elapse between the emergence of one female fly and the subsequent emergence of its progeny.
49
Q

What 4 methods are available for HAT vector control?

A
  • We have four effective methods of tsetse control
    1. Ground spraying
    2. Aerial spraying
      • nsecticide (endosulfan, deltamethrin) is sprayed from an atomiser which creates insecticide droplets of 30-40 microns.

The aerosol is applied from just above the tree canopy, at night when inversion conditions occur.

The applications are repeated for 5 cycles by which time all flies will have emerged.

3. Odour-baits
4. Insecticidal cattle
50
Q

What is Chandipura?

A
  • Note rhabdovirus ss RNA virus related to Rabies, transmitted by sandfly
  • causes severe encephalitic illness with high (~50%) mortality rate in India
  • Chandipura virus (CHPV)

Virus classification

Group:Group V ((−)ssRNA)

Order:Mononegavirales

Family:Rhabdoviridae

Genus:Vesiculovirus

Species:Chandipura vesiculovirus

Chandipura virus (CHPV) is a member of the Rhabdoviridae family that is associated with an encephalitic illness in humans. It was first identified in 1965 after isolation from the blood of two patients from Chandipura village in Maharashtra state, India[1] and has been associated with a number of otherwise unexplained outbreaks of encephalitic illness in central India.

  • transmitted by sandfly
51
Q

What is this?

What are the distinctive features?

What is it’s medical importance?

A
  • Phlebotamine Sandfly
    • small, <4mm
    • V-shaped wing configuration when resting
    • hairy dorsal thorax
    • spindly legs
  • Medical importance

− Biting nuisance and allergies

− Leishmaniasis

− Bartonellosis

− Viruses: sandfly fevers and Chandipura

52
Q

What is sandfly fever?

Disease?

A
  • Pappataci fever (also known as Phlebotomus fever and, somewhat confusingly, sandfly fever and three-day fever) is a vector-borne febrile arboviral infection caused by three serotypes of Phlebovirus. It occurs in subtropical regions of the Eastern Hemisphere. The name, pappataci fever, comes from the Italian word for sandfly, it is the union of the word “pappa” (food) and taci (silent) which distinguishes these insects from blood-feeding mosquitoes, which produce a typical noise while flying.
  • FAR, headache, generally self-limiting
53
Q

What are sandlfy control measures (and their problems)?

A
54
Q

Sandfly Summary

  1. Basic Biology
  2. Vector Surveillance and Control
  3. Diseases
A
    1. Basic aspects of sandfly biology

Sandflies breed in soil, require sugarmeals for energy, mate using pheromones and songs, and blood feed from different animals (saliva).

  1. Vector surveillance and control

Trapping methods, toxic sugar baits, pheromone traps, host removal, saliva biomarkers and vaccines

  1. Diseases transmitted by sandflies

Leishmaniasis, Bartonellosis, sandfly fevers, Chandipura

55
Q

What is this?

What diseases does it cause?

How are the diseases transmitted?

A
56
Q

What is the vector for Leishmaniasis?

Species in Old World?

in New World?

Is there a zoonotic reservoir?

A
  • sandfly
    • phlebotomus in Old World
    • lutzomyia in New World
  • Yes, hyrax, rodents, canids.
57
Q

Ixodedes vectors for which diseases?

What about Argasidae (sp. Ornithodoros or soft tick)?

A
  • Crimean Congo Hemorrhagic Fever - phlebovirius
  • Lyme disease - B. burgdorffi
  • rickettsial diseases
    • African tick typhus - R. africae
    • Med spotted fever - R. conorri
    • RMSF or Bras SF - R. rickettsiae
  • Tick borne encephalitis - flavivirus
  • tularemia - Francisella tularensis
  • Ornithodoros or soft tick transmits Borrellia duttoni, which causes endemic relapsing fever.
58
Q

With respect to the Life Cycle of the Mosquito:

  • How long does it last?
  • How long for larvae to hatch from eggs?
    • Pupa to Form from Larvae
    • Adult to emerge?
    • Female to lay eggs?
  • How does this relate to feeding cycle?
  • How many blood meals does a female take in a lifetime?
  • How many opportunities does she have to transmit infection?
A
  • 10-14 days from one laying to another
    • 2-3 for eggs to hatch to larvae
    • 5-7 to pupae
    • 7-8 for adult to emerg then female feeds and lays eggs in batches after each blood meal
  • Female mosquitoes will live < 4 wks
  • If an adult female takes 3 blood meals because of the malaria life cycle she can only transmit malaria every 3rd blood meal.
59
Q
A