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Immune Responses > Innate Immune Cells and Responses > Flashcards

Innate Immune Cells and Responses Flashcards

1
Q

What are the pros and cons of mast cells?

A

Lecture 7, slide 16-18

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2
Q

What are polymorphonuclear cells?

A

Lecture 7, slide 24, 28

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3
Q

What are some major causes of increased neutrophil numbers?

A

Lecture 7, slide 29

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4
Q

What are some major causes of decreased neutrophil numbers?

A

Lecture 7, slide 30

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5
Q

What is Kostmann disease?

A

Lecture 7, slide 32, 34

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6
Q

What are some opsonins?

A

Lecture 7, slide 38

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7
Q

What are the features of phagocytosis?

A

Lecture 7, slide 39

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8
Q

How can phagocytosis go wrong?

A

Lecture 7, slide 40-41

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9
Q

How can a phagocyte kill a bacteria after phagocytosis?

A

Lecture 7, slide 44

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10
Q

What is a respiratory burst?

A

Lecture 7, slide 45

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11
Q

What is the process of oxygen-dependent bacterial killing?

A

Lecture 7, slide 47-49

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12
Q

What is Chronic Granulomatous Disease?

A

Lecture 7, slide 51

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13
Q

What are some non oxygen-dependent bacterial killing mechanisms?

A

Lecture 7, slide 52

-antimicrobial peptides

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14
Q

What are antimicrobial peptides?

A

Lecture 7, slide 53

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15
Q

What is the process of bacterial killing by reactive nitrogen species?

A

Lecture 7, slide 54

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16
Q

What are NETs?

A

Lecture 7, slide 56-57

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17
Q

What are the advantages and disdvantages of NETs?

A

Lecture 7, slide 58, 59

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18
Q

How do bacteria escape NET killing?

A

Lecture 7, slide 58

19
Q

How can neutrophils damage tissues?

A

Lecture 7, slide 60

20
Q

What is complement?

A

Lecture 7, slide 63

21
Q

What are the three complement pathways?

A

Lecture 7, slide 65

22
Q

What are the three main end results of complement?

A

Lecture 7, slide 66

23
Q

What are the inflammatory mediators generated by the complement pathways? What do they do?

A

Lecture 7, slide 67

-chemotaxis

24
Q

What are the opsonins generated by the complement pathways?

A

Lecture 7, slide 68

25
Q

How is MAC assembled?

A

Lecture 7, slide 69

26
Q

What is the process of the classical pathway of complement activation?

A

Lecture 7, slide 73, 75

  • C3a is an inflammatory mediator
  • C3b is a potent opsonin
27
Q

What is the process of the alternative pathway of complement activation?

A

Lecture 7, slide 76-77

28
Q

How do host cells protect themselves against complement?

A

Lecture 7, slide 78, 80-81

29
Q

Why is complement tightly regulated?

A

Lecture 7, slide 82

30
Q

What are the differences between monocytes and macrophages?

A

Lecture 8, slide 6

31
Q

What are the functions of macrophages?

A

Lecture 8, slide 7, 78

32
Q

What are the different type of macrophages?

A

Lecture 8, slide 11

33
Q

What makes a good PAMP?

A

Lecture 8, slide 26

34
Q

What are PRRs? What is their function?

A

Lecture 8, slide 27

35
Q

What are some intracellular pathogen recognition systems?

A
  • NOD
  • RIG
  • some TLRs
36
Q

What are some macrophage receptors/PRRs?

A

Lecture 8, slide 33, 36

37
Q

What are scavenger receptors?

A

Lecture 8, slide 35

-foam celll: a type of cell that contains cholesterol

38
Q

What is Dectin-1?

A

Lecture 8, slide 40

39
Q

Where can TLRs be found?

A

Lecture 8, slide 48, 56

40
Q

What are Toll Like Receptors?

A

Lecture 8, slide 49

41
Q

What is the process of TLR4 signalling?

A

Lecture 8, slide 53-55

42
Q

What is the role of TLR9 and TLR2?

A

Lecture 8, slide 58, 60-61

43
Q

What is the difference between opsonic and non-opsonic receptors?

A

Lecture 8, slide 63

44
Q

What is the role of TNF-alpha, IL-1beta and IL-6?

A

Lecture 8, slide 62

Immune Responses (3 decks)

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