injury to body + post mortem changes Flashcards

1
Q

injury and lesion:

A

Injury - any damage caused by heat, cold, electricity, chemicals, or radiation

The lesion - any area of injury, disease or local degeneration in a tissue causing a change in its structure or function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

physical factors of a potential wound:

A
  • Degree of force applied
  • Area of application of force
  • Duration of application
  • Direction of application
  • Tissue properties
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

kinetic energy:

A

kinetic energy (E) = 1/2 m.v^2

m= mass of object
v= velocity (speed) of object

Imparted when:

when either a moving object strikes the body or the moving body strikes a stationary object

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

blunt force:

A

Injuries caused by impact with a blunt object (either static or mobile) – ground, wall, step, fist, foot, weapon

types:

  • Abrasions (graze, scratch)
    → Injury to skin surface
  • Contusions (bruises)
    → Burst blood vessels in the skin
  • Lacerations (cut, tear)
    → Tear/split of skin due to crushing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

abrasion:

A

a superficial/partial thickness skin injury to the epidermis

Features:
Clinically trivial

Bleeding is slight

Heal quickly by forming a scab

Leaves no scar

Often overlooked

→ Crushing by vertical force (imprint)

→ Scraping by tangential force, graze over a broad surface (road rash)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

contusions/ bruises:

A

→ Crushing of the dermal blood vessels by mechanical impact causing leakage of blood from vessels into the skin

  • Contusion-bruise of internal organs
    e.g. spleen, mesentery, muscles
  • Patterned bruising
    ‘Tram-track’ bruising – rod, baton or plank-like object

Clustered discoid bruises - fingertip pressure

Black eyes – direct trauma, skull fractures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

laceration:

A

Cut/tear/split of skin due to crushing of the skin (may be partial or full thickness)

Caused by:

  • Impact against a flat surface
    e.g. floor, wall, door
  • Impact by an edged or pointed object
    e.g. brick, claw hammer, furniture corner, splintered bone
  • Rotation of tissue on limb/torso (flaying injury), caused by revolving wheel/machinery
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

sharp force injuries:

A
  • Injury caused by any weapon with sharp cutting edge
    → superficial or penetrating

[1] Incised wounds
* Superficial sharp force injury caused by slashing motion
* Injury is longer on the skin surface than it is deep

[2] Stab wounds
* Penetrating injury resulting from thrusting motion
* Wound depth greater than length on the surface

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

incision wound:

A

Superficial Injury caused by any weapon with a sharp cutting edge by slashing motion

features:

clean
bleeding
longer than deep
no bruising

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

stab wound:

A

Penetrating injury caused by any weapon with a sharp cutting edge by thrusting motion

features:

deeper than length
clean

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

algor mortis and difficulties:

A

→ the reduction in body temperature following death, a steady decline until matching ambient temperature.

[1] consider the surrounding climate (hot/cold) in relation to the body’s normal temperature 37 C

[2] requires the measurement of core body temperature:

→ Rectal (Avoid in sexual cases - traumatic and contamination of evidence)
→ Liver (Hepatic) by subcostal stab – creates a wound

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

algor mortis process:

A

→ Loss of heat from the body due to conduction, convection and radiation

→ Little heat lost by evaporation

[1] Cooling occurs immediately after death
→ the external surface of the body (skin) will cool quicker than the interior (organs)

[2] Rapid cooling of the body will occur initially until body temperature reaches the same temperature as the atmospheric temperature
(this can be in a heated house or outdoors, depending on where the body is)
then the rate of cooling will slow down

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

rate of cooling:

A

[1] Body size (larger surface area = greater heat loss)
i.e. obese individual will cool quicker than a thin person

[2] Environmental temperature
(fluctuations from day to night, sunny vs overcast, timed heating in a house etc…)

[3] Drafts & humidity
a body found outdoors or indoors near an open window will cool quicker than body found near a heat source or in the sun

[4] Clothing & coverings
a body in multiple layers of clothing and under bedding will cool slower than a naked person

[5] Flooring
a person on a tiled floor will cool more rapidly than a person on a carpet

[6] Immersion
a body cools quicker in water than in air

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

henssges nomogram:

A

→ technique for estimation of post mortem interval (PMI) utilizing body cooling

  • Allows for correction for:
    Body weight
    Clothing
    Drafts
    Immersion in water
  • Assumptions:
    Normal body temperature at death
    No variation in ambient temperature during cooling period
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

exclusions to using henssges nomogram:

A

Nearby strong heat source (fire, heater, in arid outdoors temperatures-desert)

  • Nearby cooling source (open window, outdoors in cold climates-snow and ice)
  • Surface beneath body a strong conductor of heat (floor tiles)
  • Abnormal body temperature at death (hyper or hypothermia)
  • Body moved between death and temperature reading
    (i.e. to fridge in mortuary)
  • This tool requires very controlled circumstances surrounding death - more often than not - this is not possible!
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

problems with henssges nomogram:

A
  • Measurement errors

→ Accuracy of instrument (is the same instrument being used every time for serial measurements)

→ Accuracy of measurement (who is taking the measurement, are they trained to do so?)

→ Calibration of instruments (does the instrument even work?)

  • Variation of printed charts
  • Photocopying distortion
  • Unverified “app” formula
16
Q

Why is Livor mortis a poor indicator of post mortem interval (PMI)?

A
  • Varied interval prior to visibility
  • May appear shortly before death
    i.e. in very moribund patients
  • Development delayed and intensity affected by natural disease, blood loss (anaemia, ruptured abdominal aortic aneurysm, gastric ulceration and haemorrhage) and poisoning
  • May not be evident at all in some cases
16
Q

liver mortis:

A

→ Gravitational pooling of blood in blood vessels due to cessation of circulation in death

  • Seen as pinkish / purple skin discolouration
  • Forms a horizontal fluid level
  • Compression of tissues prevents formation (contact pallor) where blood cannot pool
17
Q

rigor mortis:

A

Rigor mortis is stiffening or contraction of muscles.

  • Muscle fibre relaxation requires ATP to break actin-myosin bonds
    → ATP requires oxygen (energy dependent process)

Two processes that facilitate it:

  • Decrease in ATP post mortem means bonds cannot break causing rigor mortis
  • Calcium build-up post mortem promotes actin-myosin cross bridging - causing muscle contraction
18
Q

factors that affect rate of rigor mortis development:

A
  • Body temperature at death
    (hyperthermia at death increases onset of rigor mortis, hypothermia delays onset of rigor mortis)
  • Ambient temperature
    (rigor mortis occurs sooner in warmer temperatures and slower onset in cooler temperatures)
  • Muscle activity immediately prior to death
    (in times of high intensity exercise, quicker onset of rigor mortis)
19
Q

circumstances that cause rigor mortis to disappear:

A
  • Decomposition
    → The quicker the onset of decomposition, the quicker rigor mortis will disappear
  • ‘Breaking’
    → if an arm or leg forcibly stretched out during autopsy examination, rigor does not re-develop in these areas
20
Q

types of decomposition:

A
  • Maceration: sterile autolysis of foetus. Specific to a foetus in a uterus. No exposure to maternal or environmental bacteria
  • Wet putrefaction: enzymatic & bacterial decomposition
  • Skeletonisation: late stage of decomposition where the skeleton is exposed
  • Adipocere: saponification of soft tissues (requires wet conditions)
  • Mummification: desiccation (removal of moisture) of soft tissues (requires cool, dry conditions)
21
Q

sequence of putrefaction:

A

[1] Green discoloration of the lower abdomen, due toovergrowth of colonic bacteria (within two days)

[2] Greenish black discoloration and swelling of the face and neck due to gas production form bacteria (within 3-4 days)

[3] Reddish brown purge fluid may extrude from the nose and mouth; this should not be confused with blood

[4] Gas formation causes diffuse swelling of the body, most noticeable in the abdomen
- Methane, H2, H2S, NH3, Ptomaines (putrescine, cadaverine) ß Sniffer Dogs!

[5] Skin slippage and blistering; hair slippage from the scalp (days to a week)

[6] Marbling occurs due to breakdown of haemoglobin within blood vessels -prominence of blood vessels on skin (days to a week)

21
Q

mummification:

A
  • Desiccation (removal of moisture) of tissues in dry conditions
    → Warm (desert, airing cupboards)
    → Cool (altitude, derelict buildings, sheds)
  • takes Months – years
  • Skin dries, shrinks and leathery
  • Internal organs may decompose or be preserved
  • More common in infants (greater surface area:mass)
  • Can start to occur within days to weeks if very arid conditions
    e.g. in a desert
21
Q

putrefactive bacteria:

A

temperature dependant bacteria that are involved in decomposition (optimal at 21 – 38 ˚C)

Mainly commensal bacteria from:
→ Gastrointestinal system
→ Respiratory tract

Pathogenic bacteria:
→ Organ/tissue specific infection
→ Septicaemia

22
Q

adipocere (saponification) grave wax:

A

→ Transformation of body fat to oleic, palmitic, and stearic acids, by hydrolysis

Location:
Occurs in Moist (humid/wet) conditions (submerged, water logged grave)

Appearance:
yellow, white, or brown and waxy

Time:
Weeks – months

Causation:
By Clostridium welchii

23
Q

Why is Adipocere (saponification) much more prolific and clear in obese individuals?

A

Because the process involves the Transformation of body fat to oleic, palmitic, and stearic acids, by hydrolysis

Obese individuals have much more % of body fat