Infections Flashcards
Secondary lesions from Herpes Type II
Isolated or in groups
Small vesicles or pustules on an erythematous base
Crusts eventually form
Heal in about 1 week
Herpes Type II
Below waist usually
Occurs through: direct contact, respiratory droplets, fluid exposure from infected person
Client is asymptomatic
May be triggered by stressor
S&S of Herpes Type II
Prodromal Phase: burning/tingling at site Red and swollen Vesicles/pustules erupt in 1-2 days Painful, itchy lesions Contagious until scabs are formed
Complications of Herpes Type II
If present at vagina during childbirth, newborn may be infected with meningocephalitis or panvisceral
Herpes encephalitis (rare)
Western Blot can determine:
Antibodies for HSV-1 or HSV-2
Herpes Zoster: Shingles
Acute inflammatory infectious disorder
Painful vesicular eruption on bright red edematous plaques along nerves.
Unilateral most often
Cause of Shingles
Varicella Zoster Virus
Incubation period for shingles
7-21 days
S&S of Shingles
Vesicles and plaques Irritation Itching Fever Malaise Painful lesion
Treatment of Shingles
Aimed at controlling outbreak Reduce pain/discomfort Prevent complications Cold compresses or baths Topical agents Anticonvulsants Antidepressants Antiviral: best if within 72 hours of outbreak
Complications of Herpes
Post-hepatic neuralgia Persistent dermatonal pain Hyperparesthesia Ophthalmia herpes can affect eyesight (Cranial Nerve V) Facial and acoustic nerve involvement Scarring can occur
Prevention of Shingles
Zostavax
Varivax
Avoidance during contagious phase
Meningitis
Inflammation of brain and spinal cord
Organism enters CNS from respiratory tract or bloodstream
Cause of Meningitis
Bacterial or viral (aseptic)
Neisseria meningitis Streptococcus pneumonia Haemophilus flu type b (Hib) Fungal organisms Viral agents
Pathological Changes of Meningitis
Organism invades CNS Meningeal inflammation Increased ICP Hyperemia of Meningeal vessels Edema of brain tissue Generalized inflammatory reaction