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410 Patho Exam 4 > Increased Intracranial Pressure > Flashcards

Increased Intracranial Pressure Flashcards

1
Q

This powerpoint is going to be from the recording from the increased intracranial pressure

A
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2
Q

First thing is we must understand what is in our head before we start talking about the fluid that will increase the pressure in our head.

The skull has 3 essential components which are ?

A

Brain tissue
Blood
Cerebrospinal fluid (CSF)

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3
Q

what are some factors that influence increased intracranial pressure ?

A

arterial pressure
venous pressure
intraabdominal and intrathoracic pressure
posture
temperature
blood gases (co2 levels)

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4
Q

something we need to understand and know is something called the

monro-kellie doctrine

what is this ?

A

if one of the three component of the skull, so blood, brain tissue or CSF increases, the other two will decrease in order to maintain balance of intracranial pressure

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5
Q

monro-kellie doctrine only works if what?

A

the skull is closed

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6
Q

what is normal intracranial pressure ?

A

5-15

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7
Q

when is our concern for intracranial pressure, like at what number ?

A

more than 20 and its sustained

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8
Q

what are some normal compensatory adaptations your body does in order to help maintain normal intracranial pressure? (3)

think of the 3 components again

A

changes CSF volume

changes in intracranial blood volume

changes in tissue brain volume

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9
Q

its very important to note that the body ability to compensate is what?

A

very limited

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10
Q

once our ability to compensate is gone, or if intracranial pressure increases, we are going to do what?

A

decompensation

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11
Q

we do look at something called cerebral blood flow when the patient is having increase intracranial pressure.

what does cerebral blood flow mean ?

A

the amount of blood in milliliters passing through 100g of brain tissue in 1 minute

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12
Q

typically what is the normal cerebral blood flow ?

A

50ml/min per 100g of brain tissue

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13
Q

the brain uses __% of the body oxygen

the brain uses __% of its glucose

A

20

25

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14
Q

something to add here, its important to note that we keep talking about perfusion and glucose levels, why do think that?

A

the reason why we keep mentioning about perfusion is because of the fact that when the body is struggling to maintain adequate circulation , the body will shunt oxygen from “unimportant areas.” and focus mainly on the important ones, like the brain and the heart, which is why we see that the brain uses 20% of oxygen

we mentioned before for glucose that a diabetic patient is in more critical care due to hypoglycemia than hyper, and the reason is for the fact that we are using over 25% of our glucose in our brain to function, so when we dont have enough, we have those neuro changes

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15
Q

what is auto regulation ?

this is something the body will do on its own to compensate by the way before we begin to decomp

A

adjusts diameters of blood vessels to ensure consistent blood flow

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16
Q

auto regulation is only effective when ?

A

the mean arterial pressure (map) is 70-150mm hg

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17
Q

what does cerebral perfusion pressure mean ?

A

the amount of pressure needed to ensure blood flow to the brain

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18
Q

what is normal cerebral perfusion pressure ?

A

60-100

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19
Q

less than 50 cerebral blood flow is associated with what 2 things ?

A

ischemia
death of neurons

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20
Q

there are 4 stages of increased intrancranial pressure, which are ?

A

stage 1 : total compensation

stage 2: decrease compensation and risk for increase intracranial pressure

stage 3 : failing to compensate ; clinical manifestations of increase intracranial pressure ( Cushing triad )

stage 4 : herniation imminent -> death

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21
Q

stage 1 its the total compensation, what are the 2 things that are happening ?

A

accommodation and auto regulation intact

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22
Q

stage 3 is the failing to compensate and clinical manifestations are becoming present for increase intracranial pressure

stage 3 is also known for what other situation ?

A

Cushing’s triad

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23
Q

what are the 3 factors that affect cerebral blood vessel tone ?

A

carbon dioxide
oxygen
hydrogren ion concentration

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24
Q

how does carbon dioxide affect cerebral blood vessel tone ?

what happens when its increased

what happens when its decreased

A

increase co2 causes smooth muscle relaxation, dilation of vessels, improved CBF
- slow deep breaths, slower respiratory rate
- improved blood flow

decreased co2 causes vasoconstriction and decreased CBF
- hyperventilation
- decreased blood flow

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25
Q

how does oxygen affect our cerebral blood vessel tone ?

A

low levels of oxygen causes dilation, but will cause anaerobic metabolism and lactic acid build up and then cellular death

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26
Q

how does hydrogen ion concentration affect cerebral blood vessel?

A

results from lactic acid build up, acidosis and vasodilation - auto regulation is lost

usually a result from low oxygen and then goes into metabolic acidosis

then with that lactic acid build up, we have acidosis and vasodilation of the vessels and the ability to compensate is gone pretty much

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27
Q

increased intracranial pressure is life threatening
its caused by an increased in any of the 3 components, which are ?

A

brain tissue, blood, CSF
cerebral edema
hypercapnia, cerebral acidosis, impaired auto regulation, hypertension

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28
Q

in the powerpoint she uses a good diagram to describe the steps of having increased intracranial pressure
there are 5 steps
what is the 5 steps.
first one to start us off is brain injury

A
  1. brain injury
  2. tissue edema causing increased intracranial pressure
  3. compression of brain tissue and decreased blood flow
  4. necrotic tissue and edema
  5. increased CO2 causing vasodilation and increase intracranial pressure
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29
Q

what are some clinical manifestations that we see in a patient who has cerebral edema ? (2)
think very broad

A

change in level of consciousness

change in vital signs

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30
Q

how does the patient appear when they have a change in level of consciousness when they have cerebral edema?

A

flattening of affect -> coma
( no emotion, no facial expression, tone of voice is very flat, avoid eye contact, very bored and uninterested )

  • confusion, agitation, combativness
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31
Q

what are some changes in vital signs when we see a patient who has cerebral edema? (2)

A

Cushing’s triad
change in body temperature

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32
Q

what is Cushing’s triad in cerebral edema Clincal manifestation ?

we do need to know this
5 things apart of this

A

systolic hypertension with widened pulse pressure, bradycardia with bounding pulses, irregular respirations

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33
Q

Cushing’s triad is a what?

A

medical emergency

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34
Q

why do we have a change in body temperature when the patient is having cerebral edema?

usually its a what?

A

the hypothalamus regulates our body temperature, so when we have increased pressure, our hypothalamus can regulate it and will cause us to be unable to maintain a good body temp

hyperthermia ( hot, fever )

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35
Q

patients can have a compression of oculomotor nerve, what do we check for ? (4)

A

unilateral pupil dilation
( occurs on ipsilateral (same) side as the lesions )
- dilation of the pupil of the side of the injury

sluggish or no response to light

inability to move eye upward

eyelid ptosis (drooping of the eyelid)

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36
Q

clinical manifestations
other cranial nerves
- diplopia, blurred vision, eom changes

central herniations can manifest as ___

uncle herination causes ____

papilledema presents with __

A

sluggish but equal pupils

dilation of unilateral pupil
( one pupil )

persistent increased in intracranial pressure
( can also be seen with severe hypertension )

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37
Q

patients may also experience decrease in motor function

what is contralateral hemiparesis/hemiplegia ?

A

a patient may find it very hard to move, typically the opposite side of the lesion

so like I hurt the right side of my brain, so I can’t move anything on my left side of the body

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38
Q

posturing a very important indictor on the severity of the brain injury or overall increased intracranial pressure.

we have 4 types, what are they called ?

A

decorticating posturing
decerebrate posturing
mixed-decorticate
opisthotonic

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39
Q

what is decorticating posturing?

A

flexing the upper extremities

extension of the lower extremities

40
Q

what is decerebrate posturing?

A

stiff extensions of arms
hyperpronation ( hyper extend arms and legs )

41
Q

which out of the 2 main posture is worse?
decorticating or decerebrate?
and why?

A

decerebrate
reason is because at least your body is trying to protect its core, when in decorticating

42
Q

what is mixed-decorticate/decebreate ?

A

one side can be decorticate and the other side can be decerebrate

43
Q

what is opisthotonic posture?

A

bowing of the body with head and heels bent back

so like the heels of the feet and your head are on the bed, but the rest of the body is lifted up in the arm

44
Q

just to go back into the powerpoint, its in the other one and not in the recording, what is an indication of a brain herniation?

what is like the neurologic emergencies we can see with the eyes ?

A

fixed, unilateral, dilated pupil

45
Q

patients usually will have a headache, which makes sense cause of the pressure
- mainly worsen in the morning
- last all day
- never get better

yet they will gave vomiting without what and what type?

A

not preceded by nausea

and projectile

46
Q

vomiting usually is not preceded by nausea, but why do patients vomit though with increased intracranial pressure?

A

trying to remove all the fluid,
it is projectile - its forceful vomit !!

47
Q

what is the 2 complications that can occur from increased intracranial pressure?

A

inadequate cerebral perfusion

cerebral herniation

48
Q

what are the 3 types of herniation ?

A

tentorial herniation
uncal herniation
cingulate herniation

49
Q

what is each//describe
tentorial herniation
uncal herniation
cingulate herniation

A

downward herniation through opening for brain stem

lateral and downward herniation

lateral displacement

50
Q

what are some diagnostic studies we are going to do for patients who have increase intracranial pressure?

A

ct scan/ mri / pet
eeg
cerebral angiography

icp and brain tissue oxygenation measurement

doppler and evoked potential studies

51
Q

what is a heavy heavy contraindication, NO! for diagnostic studies for increased intracranial pressure?

A

no lumbar puncture

52
Q

why is lumbar puncture a big no no when it comes to testing a patient with ICP?

A

because its an instant herniation and death for patients

53
Q

what is the objective assessment tool we may use to help assess a patient with ICP?

A

glasgow coma scale

54
Q

Glasgow coma scale score ranges from what ?

A

3-15
( the higher the number the better )

55
Q

what are the 3 things the Glasgow coma scale measures?

A

eyes, verbal, Motor responses

56
Q

eyes
go from ___ to ___

verbal response
go from ___ to ____

motor response
go from ___ to ___

A

spontaneous to non opening to any stimulus

talking to not even making sound with painful stimulus

obey commands to no response

57
Q

typically when you have a score of ___ or lower, we are worried for what ___and usually you are going to be ___

A

8
respiratory compromise
intubated

58
Q

when we are measuring the amount of increased intracranial pressure in the brain, what are we actually using as a measuring unit?

A

the mean pressure

59
Q

if there is any elevation from the previous measurement of intracranial pressure, you want to what?

A

immediately report it

60
Q

remember when we are placing the device to monitoring the mean pressure, or pretty much the increased intracranial pressure in the brain, it is an invasive procedure, its literally going into their skull

the risk of infection is possible, so we should what out for ?

if the monitor is there for more than 5 days, its more than likely infection can occur

A

csf leak or systemic

61
Q

if there is an increased intracranial pressure in the patient, what do you want to look at first, the patient or the machine ?

A

always the patient

62
Q

you can always get inaccurate readings of increased intracranial pressure caused by what?

test question,
look at the patient first - then look at the system

A

CSF leaks
obstruction in catheter/kinks
difference in height of bolt
incorrect heigh of drainage
bubbles/air in tubing

63
Q

how can we as nurses control increased intracranial pressure ?

A

we can remove it by the ventricular catheter with intermittent or continuous drainage

64
Q

when we are draining out the CSF, we have to what?

A

monitor and essential to keep track cause we dont want to remove too much

65
Q

when we have a patient who have ICP, typically there is an underlying cause, so we obviously are going to treat the underlying cause in order to stop the ICP.

ICP IS A SYMPTOM, NOT A CONDITION.

what are some things that we can do to help aid this?

A

oxygenation - intubation and mechanical ventilation

surgery possible

66
Q

what are the 3 drug therapies we are going to be using for these patients with ICP?

A

mannitol (osmitrol)
hypertonic saline
corticosteroids

67
Q

what does mannitol do?

hypertonic saline does the same thing by the way ^

A

brain fluid into your vessels, and dilation of your blood viscosity

  • improvement of perfusion to the brain as we balance out the fluid
68
Q

why do we use corticosteroids ?

A

mainly help with inflammation

69
Q

take corticosteroids with ___
dont take during what point of the day ? and why?

___your blood sugar

can cause ____blood pressure

A

food cause ulcers can occur

night because it can cause increase activity

increase blood sugar

increase blood pressure

70
Q

when taking a corticosteroids, we usually want to take it with what other medications ?

A

h2 receptor blockers
proton pump inhibitors
concurrent antacids

71
Q

if a patient has a fever, seizures can increase, what medications can we use to aid with this ?

A

antipyretics - help with fever
anti seizure medications
sedatives
analgesics - pain
barbiturates - calm them

72
Q

what 2 nutritional states do you end up having when you have increased intracranial pressure ?

A

hyper metabolic and hyper catabolic state with increase need for glucose

break down of muscles and fat

73
Q

how are we going to feed patient ?

A

enteral or parenteral nutrition
early feedings
normal saline
keep fluid volume normal

74
Q

notes
nursing assessment
- assess history of ICP
- level of consciousness
- glasgow coma assessment
- assess perfusion, ventilation and gas exchange
( bun/creatinine, blood pressure, gas exchange, respiratory rate/depth/rythem, pulse ox)

A
75
Q

we are also going to do some cranial nerves assessment
eye movement
alert and orientation
symmetrical
gait
extra ocular movement

we can do corneal reflect?

we can do oculocephalic reflex?

we can do oculovestibular (caloric stimulation)?

A
  • touching their corneal and see if they blink
  • think of it as blowing into their eyes to get a blink reflex
  • doll eyes reflex, they turn their head sideways, there eyes turn to that side too
  • turn head to the right -> eyes move to the right

squirt cold water in their ear, and when the cold water hits there ear drum, there eyes move away from the cold water, so like right ear -> move eyes to the left

76
Q

to recap what is the reflex name for the blinking?

what is the reflex name for doll eyes ?

what is the reflex name for the ear drums water?

A

corneal
oculocephalic reflex
oculovestibular
(caloric stimulation)

77
Q

how can we assess their motor strength ?
what test ?

A

pronator (palmar ) drift test
squeeze hands too
raise foot off bed and bend knees

78
Q

what is the pronator (palmar) drift test?

A

lift there arms up with the palm facing up, on their weak side palm and arm will turn downward

79
Q

typically we want to avoid causing any painful stimuli to a patient of any kind, but in neuro, when the patient isn’t responding, we have to cause pain to see if they even can respond.

so we can do something like what ?

A

sternal rub with their knuckles, see if they withdraw or anything

80
Q

if you think a patient is faking their pain response, a good testing measure is how ?

A

lifting their arm above their face and letting it fall on their face, typically patients will move their arm away if they are faking it cause they dont want to get hit in the face

81
Q

there are 5 abnormal respiratory patterns
ill give you the names , you tell me the description of each

cheyne-strokes
central neurogenic hyperventilation
apneustic breathing
cluster breathing
ataxic breathing

A

cycles of hyperventilation and apnea

sustained, regular rapid, deep breathing

prolonged inspiratory phases or pauses alternating with expiratory pauses

clusters of breaths follow each other with irregular pauses between

completely irregular with some breaths deep and some shallow. random, irregular pauses, slow rate

82
Q

notes
nursing diagnosis
- decreased intracranial adaptive capacity
- risk for ineffective cerebral tissue
- risk for disuse syndrome
- risk for injury

nursing planning overall goals
- maintain a patent airway
- icp within normal limits
- normal fluid and electrolyte balance
- prevent complications secondary to immbolitiy and decreased LOC

A
83
Q

nursing implementation
respiratory

Respiratory function

-Maintain patent airway-at risk for airway occlusion.

  • Elevate head of bed 30 degrees.
  • Suctioning PRN
  • Minimize abdominal distention with NG tube placement
  • Monitor ABGs.
  • Maintain effective ventilation
A
84
Q

the problem with elevating the head of the bed with ICP, we can decreased the perfusion, so we can only do what. (number)

no semi or high fowlers

A

30 degrees

85
Q

remember SUCTIONING AS ?
what do we try first before suctioning to aid with secretions

A

NEEDED.
- avoid it
- try fluids first !

86
Q

what are we going to help aid with pain and anxiety manamgent ?

A

opioids usage
benzodiapzepines
neuromuscular blocking agents

87
Q

what is the issue with opioids when having a patient with ICP, or any neuro issue?

A

it makes you sleepy!
it depresses your respiratory rate as well !

we need to assess their status frequently so be mindful when your giving opioids

88
Q

we can give two medications called
propofol (diprivan) what is the side effect?

dexemedetomidine
(precedex) used for ?

A

hypotension -> decreased cerebral perfusion pressure

to aid patients who are on a vent to not fight with the vent so much

89
Q

nursing implantation notes

Fluid and electrolyte balance
Monitor IV fluids.
Daily electrolytes
Monitor for DI or SIADH.
Monitor and minimize increases in ICP.

typically DI and SIADH are very common things we see in these patients

A
90
Q

what is Diabetes insispidus ?
(4 things )

A

cause by decreased ADH, leads to increased urine output and hyponatremia

too little ADH
pee too much
too little salt
dehydration

91
Q

what is syndrome of inappropriate antidiuretic hormone (SIADH)?
4 main things

A

caused by increased ADH, leads to decreased urine output and hypernatremia

too much ADH
no pee
too much salt
fluid overload

92
Q

how to help optimize ICP and CPP note

  • HOB elevated to 30 degrees
  • Head midline
  • Prevent extreme neck flexion. neutral chin,
    no chest chin or chin to air
  • Turn slowly.
  • Avoid coughing, straining, Valsalva. (holding breath and increase abdominal pressure)
  • stool softener & high fiber diet will aid that
  • Avoid hip flexion.
    hip flex can impair circulation and perfusion
A
93
Q

Minimize complications of immobility.

Protection from self-injury
Judicious use of restraints; sedatives

Seizure precautions, side rails up, padding side rails, lowest bed

Quiet, nonstimulating environment

Psychologic considerations

A
94
Q

evaluation

Expected Outcomes

  • Maintain ICP and CPP within normal parameters.
  • No serious increases in ICP during or following care activities
  • No complications of immobility
A
95
Q

she doesn’t mention this in the recording, but on the slideshow she has the babinski reflex

what is this ?
what is normal(negative)?
what is abnormal (positve)?

A

the lateral aspect of the sole is stroked from the heel to the big toe

planter flexion of all toes

dorsiflexion of the big toe with the other toes fanning out

96
Q

an abnormal positive for babinski reflex is only normal for who ?

A

infants through ages of 2

97
Q

if the patient is positive of a babinski reflex, is often associate with what?

A

CNS disorders

410 Patho Exam 4 (5 decks)

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