Important Theories Flashcards

(Stucky study guide)

1
Q

Automaticity

A

automatic processing; automatic in the sense that a fxn does not require a good deal of cognitive effort; habit system (neuroanatomical base: subcortical areas including basal ganglia)

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2
Q

Effortful Processing

A

not automatic, requires greater cognitive effort

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3
Q

Brain-Behavior Relationship

A

the role the brain plays with regard to human behavior including cognition, emotion, sensory-motor, and related abilities

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4
Q

Central Executive

A

Alan Baddeley; system responsible for the control and regulation of cognitive processes, largely related to prefrontal brain regions, helps to regulate attn, working memory, and memory

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5
Q

Executive Function

A

regulatory mechanisms responsible for higher-level human fxns involved in organization, reasoning, flexibility, initiation, and related skills; largely subserved by prefrontal regions and networks

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6
Q

Equipotentiality

A

Karl Lashley; an intact brain region can carry out the fxns of damaged brain regions, thought that the brain has the capacity to transfer “fxnal memory” from damaged regions to intact region

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7
Q

Modular/Localization View

A

discrete brain regions are responsible for specific mental and behavioral functions

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8
Q

Two-Streams Hypothesis

A

as visual info leaves the occipital lobe it follows two main streams: dorsal (associated with objects spatial location; “where” stream) and ventral (associated with an object’s identity; “what” stream)

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9
Q

Whole-Brain

A

domain-general theory; states that the whole brain acts in concert to produce fxns; e.g., gestalt psychologists believed that visual perception was a whole-brain phenomenon

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10
Q

Brain Reserve Hypothesis

A

refers to a brain’s ability to absorb insult and potentially recover; passive threshold model: hinges on brain’s physical health prior to insult/disease onset; critical threshold of brain cell loss must be crossed to get a deficit

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11
Q

Cognitive Reserve Hypothesis

A

education and enriched experience can increase cerebral reserve and are relatively protective against the expression of sxs following brain disease or injury; does not prevent impairment but can modify the functional and clinical expression of conditions; efficiency model: the mind’s resistance to brain damage due to presence of more efficient synaptic networks or preexisting cognitive abilities

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