Hypovolemic Shock Flashcards

1
Q

What is haemodynamic shock?

A

Acute- inadequate blood flow throughout the body

Catastrophic fall in aBP

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2
Q

What causes haemodynamic shock?

A

Fall in CO- mechanical (filling)

  • pump
  • volume loss

Fall in Peripheral resistance- vasodilation

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3
Q

What are the types of shock due to reduced CO?

A

Cardiogenic
Mechanical
Hypovolaemic

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4
Q

What are some causes of cardiogenic shock?

A

MI- damage to LV

Serious arrhythmias- bradycardia or tachycardia

Acute worsening of heart failure

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5
Q

What occurs in cardiogenic shock?

A
  • CVP raised/ normal
  • Dramatic drop in arterial BP (pump failure)
  • Tissues poorly perfused
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6
Q

How does cardiac tamponade cause shock?

A

Restricts filling of heart (increased pressure in all chambers)
High central venous pressure (can’t get into heart)
Low arterial BP

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7
Q

How does a pulmonary embolism cause shock?

A

Massive embolism occludes artery

  • pul arterial pressure high
  • RV can’t empty (CVP high as can’t fill RV)
  • LA pressure low due to small return
  • Arterial BP low
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8
Q

How is cardiac tamponade treated?

A

Pericardiocentensis

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9
Q

How does hypovolaemic shock occur?

A

Due to reduced blood volume

<20% fine
20-30% some shock
30-40% large decrease mean aBP= serious shock

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10
Q

How does the body respond to hypovolaemic shock?

A

Fall in BP detected by baroreceptors
Increased sympa activity
(Increased contractility and hr)
Vaso and veno constriction (increase return to heart)

Reversal of fluid movement in capillaries

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11
Q

How does fluid movement in capillaries change?

A

Net movement of fluid into caps
(Internal transfusion)

Increases PR= reduced cap hydrostatic pressure

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12
Q

What signs does a patient with hypovolaemic shock show?

A

Tachycardia
Weak thready pulse
Pale skin
Cold and clammy extremities (sympa activation)

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13
Q

How can prolonged vasoconstriction lead to decompensation?

A
Peripheral tissues experience hypoxia 
Release of chemical vasodilatory mediators 
TPR decreases 
Dramatic bp fall 
Organs not perfused 

The sympa system can’t overcome vasodilation

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14
Q

What are long term responses to increasing mean aBP?

A

RAAS system
Anti-diuretic hormone

Several days to restore 20% blood loss

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15
Q

How would you treat hypovolaemic shock?

A

IV fluids
Transfusion of blood products

Amount determined by CVP values (careful not to overperfuse)

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16
Q

What is distributive shock?

A

Decrease in TPR

Profound peripheral vasodilation

17
Q

What are types of distributive shock?

A

Toxic (septic)

Anaphylactic

18
Q

How does sepsis cause shock?

A

Endotoxins releases by bacteria trigger excessive inflammatory response

Causes vasodilation, fall in TPR, fall in aBP
Impaired perfusion organs

Capillaries leaky= lose blood volume

Increased coagulation= local hypo perfusion

19
Q

What symptoms/ signs does septic shock cause?

A

Tachycardia (sympa trying maintain BP)

Initially warm red extremities 
Later vasoconstriction (syma)= hypoperfusion
20
Q

How does anaphylaxis cause shock?

A

Release of histamine from mast cells
(Powerful vasodilator that syma response can’t overcome) = fall in TPR

Drop in aBP
Impaired perfusion organs
Bronchoconstriction ect

21
Q

What signs/ symptoms will be present in anaphylactic shock?

A

Difficulty breathing
Rapid hr (syma trying to increase CO)
Red and warm extremities (vasodilation in peripheries)

22
Q

How is anaphylactic shock treated?

A

Pharmacological does of Adrenaline

Causes vasoconstriction of alpha 1 adrenoreceptors, increase TPR

23
Q

What are the general features of heaodynamic shock?

A

Fast pulse
Low aBP
Activation of sympa system and effects that come along with that