Hypothermia Flashcards

1
Q

Define hypothermia

A

Body temperature <35oC

mild: 32-35°C
moderate: 28-32°C
severe: < 28°C

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2
Q

Aetiology of hypothermia

A

Either increased (1) heat loss (2) decreased thermogenesis (3) impaired thermogenesis

age (elderly and infants at risk)
environmental – exposure, drowning, alpine environment, poverty (lack of heating or shelter)
drugs/ tox – alcohol, sedatives, vasodilators
Sepsis
CNS disorders e.g. hypothalamic lesions, hypopituitarism
Endocrine/ metabolic – hypothyroidism, adrenal insufficiency, hypothermia, malnutrition
Trauma — burns, spinal cord injury
Shock
Skin disorders — psoriasis, exfoliating conditions
Iatrogenic — cold fluid administration, intra-operative, therapeutic hypothermia
Psychiatric (may lead to exposure)

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3
Q

Signs and symptoms of hypothermia

A

CVS: bradycardia is normal; decreased Q and MAP, vasoconstriction
RESP: decreased CO2 production, decreased PAO2 and PaCO2 due to increased gas solubility, increased dead space, diaphragmatic fatigue, metabolic acidosis -> pulmonary hypertension
GI: decreased hepatic metabolism and blood flow, decreased splanchnic circulation
METABOLIC: decreased BMR, shivering, left shifted oxy-Hb dissociation curve, hyperglycaemia, decreased drug metabolism
CNS: neuroprotection, fixed dilated pupils at < 30 C (mimics brain death)
HAEM: increased bleeding time, PT and APTT, VTE risk, decreased platelet and WCC
RENAL: decreased GFR and RBF, cold-induced diuresis

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4
Q

Investigations for hypothermia

A

1st: axillary/oral temperature – if less than 36.5 C →
2nd: low-temperature PR probe / infra-red ear thermometer

ECG: broad complex bradycardia | prolonged PR/QT | J waves | shivering artefacts

FBC
U&Es
TFTs
Glucose
Amylase
ABG

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5
Q

Management for hypothermia

A
  1. Resuscitation
    - pulse check – palpate for up to 1 minute (consider Echo / Doppler as hard to find – do not delay CPR)
    - move patient gently if <32 degrees due to risk of triggering VF
    - no adrenaline or other drugs until >30C
    - between 30-35C double the dose intervals of ACLS drugs
    - shock VF up to 3 times if necessary, then no further shocks until T>30C
    - ‘not dead until warm and dead’ (30-32C)

Warming: passive, peripheral active, central active

Supportive:
- use esophageal probe preferentially (core temperature, minimal lag time)
- use low reading thermometer
- ABG measurements at 37C (temperature uncorrected values) to allow serial monitoring
- Antibiotics to cover for pneumonia
- Catheter
- Cardiac monitoring

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6
Q

Describe passive warming

A

For Patients who are able to shiver (1.5° C/hr)
Keep dry
Warm environment
Insulation with blankets e.g. aluminium foil
Hat
Allow to mobilise if conscious

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7
Q

Describe peripheral active warming

A

Chemical heat pads
Radiation methods
Forced air warming blankets (1-2C/h)

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8
Q

Describe central active warming

A

warmed (40-46C) humidified inspired gases (1 C/h; 1.5°C/h ET tube)
warm IV fluids (42C) (only give if need fluids, prevents cooling rather than promotes warming) – use Level 1 fluid warmer
body cavity lavage with 40C fluid e.g. peritoneal (3C/h), gastric, bladder, right-sided thoracic lavage (3-6C/h – use 2 ICCs for continuous flow)
RRT
ECMO/ bypass (9-18C/h)

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9
Q

Complications of hypothermia

A

Cardiac arrhythmias
Hypoglycaemia
Hyperkalaemia
Rhabdomyolysis
GI disorders
Bleeding diathesis
Bladder atony
Local injuries

Afterdrop, a drop in core body temperature during rewarming may occur a consequence of peripheral vasodilation and release of cold peripheral blood to the body core. It is not usually significant.

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10
Q

Prognosis for hypothermia

A

Generally, patients with mild hypothermia will recover without any residual effects
If the core temperature was between 26°C and 32°C (78.8°F to 89.6°F) and the patient received acute resuscitation, recovery is likely but with lasting morbidity
Most patients with a core temperature <26°C (<78.8°F) do not survive
The triad of coagulopathy, acidosis, and hypothermia is also associated with increased mortality.

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