HTN, Cerebrovascular, Parkinson's, polymyalgia rheu review Flashcards

1
Q

common form of HTN in the elderly, is defined as sys BP >140 and diastolic BP <90

A

isolated systolic hypertension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

First line pharm therapy for HTN in elderly

A

thiazide diuretic 12.5 mg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

When does SBP more important than DBP as CVD risk factor?

A

50+

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Benefits of lowering BP

A

decreased stroke, MI, heart failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Independent risk factor for CVD

A

LVH

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What are thiazides also good for in addition to BP control?

A

osteopenia/osteoporosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are CCBs also good for in addition to BP control?

A

Raynaud’s syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

CI to thiazides

A

gout or h/o hyponatremia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

CI to BBs

A

2nd/3rd degree heart block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

CI in pregnancy for HTN

A

ACEI/ARB

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

caused primarily by an increase in arterial stiffness due to increased collagen deposition and cross linking, degeneration of elastin fibers, atherosclerotic changes, and age-related endothelial dysfunction

A

isolated systolic hypertension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

BP readings may be falsely elevated in some elderly patients with very stiff, calcified arteries

A

pseudohypertension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Two classifications of stroke

A

ischemic (75%) or hemorrhagic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Brief episodes of focal neurological deficits lasting 2-3 minutes to at most a few hours but no longer than 24 hours leaving no residual deficits with complete functional recovery

A

TIA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Acute, sustained functional neurological deficit lasting from days to permanent. There is neuronal necrosis or infarction

A

complete stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Small, deep infarcts caused by occlusion of the small arteries that penetrate deeper brain structures. Subtle symptoms

A

lacunar

17
Q

stroke present seriously ill. Deteriorate more rapidly and have HA, N/V, and decreased consciousness as prominent signs

A

hemorrhagic stroke

18
Q

Rupture of an artery with bleeding onto the surface of the brain

A

subarachnoid

19
Q

Cause of subarachnoid hemorrhage

A

aneursym (85% Berry)

20
Q

“Worst headache ever” in the patients life radiates to face and neck. Phonophobia or photophobia

A

subarachnoid hemorrhage

21
Q

Cause of intracerebral hemorrhage

A

HTN

22
Q

Rupture of an artery with bleeding into the brain parenchyma

A

intracerebral

23
Q

Treatment of ischemic stroke

A

fibrinolytic therapy (tPA)

24
Q

Timeframe for administration of tPA

A

within 3 hours of onset of signs and symptoms

25
Q

Characteristics of parkinsons

A

resting tremor, muscular rigidity, bradykinesia

26
Q

Initial drug treatment for Parkinsons

A

carbidopa/levodopa or dopamine agonists

27
Q

characterised by severe bilateral pain and morning stiffness of the shoulder, neck and pelvic girdle

A

polymyalgia rheumatica

28
Q

Most useful lab test for PMR

A

ESR

29
Q

Patient presents with new HA, jaw claudication, or visual disturbance. h/o of PMR

A

giant cell arteritis

30
Q

Treatment for PMR

A

prednisone 15mg/day for 18-24 months