Hip LCP/AVN Flashcards
Avascular necrosis of the femoral epiphysis before closure of the growth plate due to disruption of blood supply
Legg Calvé Perthes Disease (ages 4-9)
Blood supply to the femoral head comes from three sources
Ligamentum teres artery, Epiphyseal arteries and Metaphyseal arteries
Lateral Epiphyseal Arteries are located on the
femoral neck
pain, inflammation, bone turns to dead tissue; several months to 1 year
AVSCULAR Phase of Legg Calvé Perthes Disease
dead bone cells are cleaned up, new cells are formed; still irritated and painful; 1 to 3 years
Revascularization Phase of Legg Calvé Perthes Disease
deposition of new bone cells predominates; 1 to 2 years
Repair phase of Legg Calvé Perthes Disease
enlarged head, flattened head, large greater trochanter
Deformity phase of Legg Calvé Perthes Disease
most serious functional problems in hip AVN result not from
the body’s attempt to repair the damage done by the ischemic event.
Arthrodesis, AKA
joint fusion
Children with LCPD often have delayed
bone age.
No radiologic changes are present and Scintigraphy may demonstrate decreased or increased uptake due to reparative processes
Stage I Hip AVN
Radiologic changes are present, but the shape of the femoral head is preserved.
Radiologic changes include diffuse osteoporosis, subchondral lytic lesions, sclerotic changes, and mixed osteoporotic or sclerotic changes.
Stage II Hip AVN
Classic radiologic features of osteonecrosis are present.
Findings include collapse of subchondral bone, crescent sign, and flattening of the femoral head.
Patients are symptomatic.
Stage III Hip AVN
Findings include extensive collapse of subchondral bone and severe deformity of the femoral head.
Acetabulum maintains its shape.
Stage IV Hip AVN
Findings include those in stage IV with acetabular involvement
Stage V Hip AVN