Hip Disease Flashcards

1
Q

what is the best prevention for juvenile hip disease

A

selective breeding
(hip laxity is highly heritable)

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2
Q

what disease does juvenile hip laxity predispose to

A

osteoarthritis

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3
Q

clinical signs of juvenile hip disease

A

varies from mild to severe depending on if laxity is functional or passive

requirement for surgery depends on level of pain/lameness

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4
Q

functional laxity

A

pathologic laxity that occurs during normal weight-bearing

caused by:
- thickening of femoral head ligament
- high joint fluid volume
- low pelvic muscle mass
- obesity/rapid growth
- early spay/neuter

causes clinical signs and progressive OA

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5
Q

passive laxity

A

laxity that only occurs during manual manipulation of the hips (ex. evaluated during pennhip rads)

presence of passive hip laxity is the NUMBER 1 risk factor for development of OA later in life

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6
Q

is hip disease expressed throughout life or only in certain stages

A

continuous expression throughout life

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7
Q

diagnosis for juvenile hip laxity

A

physical exam and radiographs (VD hip extended + pennHIP)

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8
Q

PE for juvenile hip laxity

A

typically done under sedation

barlow: subluxing the joint
ortolani: reducing the jointback into place

laxity can still be present even if not palpated

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9
Q

radiographs for juvenile hip laxity

A

VD hip extended: can ID subluxations and secondary DJD changes

pennHip: evaluates VDHE, compression view, and distraction view
- best for diagnosing PASSIVE hip laxity by calculating distraction index

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10
Q

what is the best diagnostic method for breeders to use

A

pennHip radiographs - measure distraction index and recommend breeding the top 50% of “tightest” dogs

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11
Q

at what age are pennHip radiographs most diagnostic

A

6 months or older

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12
Q

treatment of juvenile hip disease

A

palliative care ONLY - cannot surgically prevent OA from chronic hip disease

some surgical options (JPS and TPO)

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13
Q

juvenile pubic symphysiodesis (JPS)

A

only used in dogs <4 months

goal: early fusion of the pelvic symphysis to cause ventroversion of the pelvis

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14
Q

triple pelvic osteotomy (TPO)

A

osteotomies of the ischium, pubis, and ilium to rotate the acetabulum

controversial; has high complication rates

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15
Q

diagnosis of adult hip disease

A
  1. clinical signs
  2. PE
  3. radiographs
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16
Q

clinical signs of adult hip disease

A

mild to severe lameness
usually chronic and insidious
stiffness
bunny hopping gate (hind limbs)
exercise intolerance

17
Q

PE for adult hip disease

A

muscle atrophy (bilateral)
dorso-lateral protrusion of the greater trochanter
pain on hip extension
decreased hip ROM
crepitus (crackling)
ortolani sign

18
Q

radiographs for adult hip disease

A

VD hip extended view
1. joint conformation: <50% coverage of femoral head by the dorsal acetabulum
2. joint incongruity: non-parallel margins of dorsal femoral head and acetabulum
3. joint instability: subluxation
4. secondary DJD changes: thickening of femoral head/neck, osteo and enthesiophytes, flattened femoral head

19
Q

treatment for adult hip disease

A

conservative and surgical

20
Q

conservative adult hip disease management

A

weight loss/management
pain contol (NSAIDs)
supplements (EFAs)
exercise modificaiton - need to still be moving the joint

21
Q

surgical options for adult hip disease repair

A
  1. THR
  2. FHNE/FHO
22
Q

total hip replacement (THR) candidates

A
  • clinical signs of OA
  • unable to achieve desired level of activity
  • unresponsive to conservative management
  • no signs of infection
23
Q

THR outcomes

A

return to normal function BUT has higher risk of complications than FHNE

24
Q

cemented vs non-cemented vs hybrid THR

A

cemented: implants are held in by bone cement; easier but less precise

non-cemented: implants are held in by bony ingrowths that develop over time
- most common
- lower risk of infection, immune reaction, breakage, etc
- lateral bolts hold implant in place until bony ingrowth take place

hybrid: cemented stem + non-cemented cap

25
Q

femoral head and neck excision (FHNE) outcomes

A

same as FHO (femoral head osteotomy)

removal of the femoral head and neck to provide pain relief

eliminates pain but does NOT restore normal function - hip joint will fill in with scar tissue so gait will still be abnormal

26
Q

FHNE/FHO candidates

A
  • previous failed repair of femoral head/neck fractures
  • aseptic necrosis of femoral head
  • pain from hip OA
  • non-repairable acetabular fractures
  • luxations that do not stay reduced
  • failed THR implants
27
Q

aseptic necrosis of the femoral head

A

vascular trauma to the circumflex femoral vein leading to anoxia in the epiphysis –> osteoclast/blast remodelling –> revascularization

unable to catch inciting cause of vascular trauma

28
Q

signalment of ANFH

A

mild trauma at 4-12 months old

usually small breed dogs

29
Q

clinical signs of ANFH

A

unilateral NWB or partial WB

30
Q

what is the biggest risk factor for hip dysplasia in cats

A

hip laxity

uncommon but can occur