Hip Flashcards

Conditions affecting adult hip

1
Q

What is femeroacteabular impingment syndrome

A

altered morphology of the femoral neck and or acetabulum where extra bone grows giving friction and pain

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2
Q

Types of FAI

A

Mixed
pincer
CAM

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3
Q

CAM type of FAI pathophysiology

A

femoral deformity where there is an aymmetric femoral head with decreased head to neck ratio. Can be related to previous SUFE

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4
Q

Who gets CAM?

A

Young athletic males

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5
Q

Pincer type pathology

A

acetabular deformity creating acetabular overhang

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6
Q

who gets pincer deformity?

A

Females

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7
Q

What complications are there of FAI?

A

Labrum tears/damage
damage to cartilage
osteoarthritis in later life

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8
Q

Patient presentation of FAI

A

activity related groin pain especially in flexion/rotation
difficulty sitting
C sign positive
FADIR +ve

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9
Q

Diagnosis of FAI

A

Radiographs
CT
MRI: gold standard as shows damage to labrum and bony oedema

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10
Q

Management of FAI CAM

A

observation in asymptomatic patient
arthroscopy/ open surgery to remove CAM/ debride tears
arthroplasty in pt with secondary OA

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11
Q

Management of FAI Pincer

A

observation in asymptomatic patient
peri-acetabular osteotomy /debride lateral tears
arthroplasty in secondary OA

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12
Q

What is avascular necrosis?

A

Failure of blood supply to the femoral head

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13
Q

Causes of Avascular necrosis?

A

Idiopathic or trauma

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14
Q

Pathophysiology of trauma and AVN

A

Trauma stops the blood supply to the femoral head: cuts off the medial circumflex artery

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15
Q

Pathophysiology of idiopathic AVN

A

1) Coagulation of intraosseous circulation
2) venous thrombosis causes retrograde aterial occlusion
3) intraosseous hypertension
4) Decreased blood supply to the femoral head
5) necrosis of the blood supply
6) Chondral fracture and collapse

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16
Q

Aetiology of AVN

A

Males> females
35-50 typical age
Bilateral disease in 80% so much check other leg!!!!!!

17
Q

Patient presentation of AVN

A

Insidious onset groin pain exacerbated by impact/stairs

18
Q

Examination findings of AVN

A

usually normal unless advanced and developed OA

19
Q

Diagnosis of AVN

A

Radiographs normal in early disease

MRI more specific and sensitive

20
Q

Classification of AVN

A

stages 0-3: pre subchondral collapse and reversible

stages 4-6: post subchondral collapse and irreversible

21
Q

Management of AVN

A

Prechondral collapse: biphosphonates, core decompression, bone graft/vascularised bone graft and rational osteotomy for small bits
Postchondral collapse: Total hip replacement

22
Q

Risk factors for AVN

A
alcohol
steroids
blood clotting disorders
dysbaric disorders
trauma 
radiation
23
Q

what is Idiopathic transient osteonecrosis of the hip?

A

local hyperemia, impaired venous return with marrow oedema and increased intramedullary pressure (not avascular!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!)

24
Q

Patient presentation of ITOH

A

Progressive groin pain over several weeks, difficulty weight bearing and usually unilateral

25
Epidemiology of ITOH
Males> Females 2 groups of people get it: middle aged men and women in late pregnancy rare
26
Diagnosis of ITOH
Elevated ESR Radiograph: osteopenia of head and neck and thinning of the cortices MRI: gold standard shows oedema
27
Management of ITOH:
self limiting usually resolves in 6-9 months analgesia protect weight bearing to prevent stress fracture
28
What is Trochanteric Bursitis?
The iliotibial band rubs against the trochanteric bursitis causing damage, inflammation and pain
29
Aetiology of Trochanteric bursitis
Women bc wider pelvis | Young runners and older patients
30
Presentation of TB
Pain on lateral hip | pain on palpation of the greater trochanter
31
Diagnosis of TB?
mainly clinical radiograph is unremarkable MRI shows inflammation but not usually indicated
32
Management of Trochanteric Bursitis
``` Analgesia NSAIDS Physio Steroid injections No proven benefit of surgery ```
33
WHat is osteonecrosis?
End point of many diseases. | Degenerative disease of the synovial joints with the loss of cartilage and the thickening and fibrosis of the synovium
34
Aetiology of Osteoarthritis?
Female >men inc incidence with inc age genetic component pre-existing hip disease
35
Pathophysiology of osteoarthritis
loss of homeostatic balance of cartilage synthesis and degradation caused by either increased catabolism or decreased synthesis
36
Patient presentation of OA
``` Groin/buttock pain morning stiffness worse on activity night pain start up pain reduced ROM ```
37
Diagnosis of OA
Radiograph:LOSS
38
Management of OA
``` Analgesia NSAIDs weight loss/walking aids physio steroid injections total hip arthroplasty ```