🩸Hemodynamics Flashcards
Cardiac Output
SV x HR
indicator of inotropic ability
4-8L/MIN
Cardiac Index
CO/Body Surface Area
2.2-4.0L/min
3 Factors of Stroke Volume
- Preload
- Afterload
- Contractility
Preload
volume indicator
stretch before systole “filling pressure”
Elevated Preload causes (5)
Volume overload
Left/Right Ventricular Dysfunction
Valvular stenosis or insufficiency
Cardiac Tamponade
Increased right ventricular vs left ventricular
Right = BODY –> hepato-juglar reflux, JVD, edema (increased systemic pressure)
Left = LUNG –> crackles, S3, cough (increased wedge pressure)
Preload reduction –> direct reduction of blood volume
Diuretics
Preload reduction –> promote vasodilation
Nitrates (Nitroglycerin)
Preload reduction –> patient positioning
Semi-fowlers
High - fowlers
Preload reduction is caused by (5):
Hypovolemia
Hemorrhage
Third Spacing
Diuresis
Vasodilation
Preload reduction assessment findings:
Tachycardia/hypotension
dry, cool skin
dry mucous
poor skin turgor
alt LOC
decreased UOP/vital organ perfusion
Tests for preload
CVP R
PCWP L
Treatment for low preload (Non-pharm)
Volume administration (Crystalloid, colloid, PRBC)
Modified trendelenburg
Afterload
resistance to stretch, FORCE
Afterload critical factor
systemic vascular resistence
afterload elevation is caused by (5):
vasoconstriction (increased SVR)
alpha 1 agonist –> epi, norepi
catecholamine release
Hypertension
increased aortic impedance (stenosis)
Afterload reduction –> arterial dilation
nitroprusside (nipride)
Afterload reduction –> block angio 1 - 2
ACE-I/ARBS
(-pril) (-sartan)
Afterload reduction is caused by (4):
vasodilation (decreased SVR)
inadequate aortic valve function
inflammatory response
hyperthermia
Pharm to increase afterload
Vasopressors
Norepi (levophed)
Phenlyephrine
Dopamine
Vasopressin
Contractility
inotropic action THE PUMP
+ increase
- decrease