Atrial Dysrhythmias Flashcards
Atrial dysrhythmias
originate from ectopic focus in atria, not the SA node
occurs premature (pre normal sinus)
Automaticity
heart self-generates an electrical impulse
Premature Atrial Contractions
(PAC’s)
regular sinus rhythm is interrupted by early abnormally-shaped atrial P wave
* normal or narrow QRS (dependent on AV node health)
PAC cause
- nicotine, caffeine, ETOH
- strong emotions
- MI
- digitalis
- low Mag/K+
PAC ECG
Rhythm –> regular
P wave –> present (may be hidden)
PAC treatment
remove causitive factor
Paroxysmal Atrial Tachycardia
(PAT)
burst of 3 PACS
same causes
may have lightheadedness/palpations
PAT EKG
- starts with PAC
- rate 160-250
- different P wave
- PR 0.12-0.20
SupraVentricular Tachycardia
(SVT)
rapid, sustained artrial tachycardia
* sinus tach
* atrial flutter
* atrial fib
SVT clinical manifestations
increased rate decreases filling time = ↓ CO
SVT treatment
heart rate reduction
1. valsalva maneuver
2. carotid massage
3. adensosince
4. amiodarone/CCB/BB
5. Cardioversion
SVT SKG
rate: >130
P wave: not identifiable
QRS <0.12
Electrical Cardioverson
Synchronized electrical shock with R-wave
What would happen if you delievered a shock during repolarization?
VFIB
Atrial flutter
SVT
Rate: 300 BPM
P waves not present
1:1 300BPM
2:1 150BPM
4:1 7BPM
A flutter clinical manifestations
- ↓CO
- thrombi can form on atria walls (d/t no contraction) = PE, Stroke, MI
A flutter treatments
- Amiodarone
- BB/CCB/Digoxin
- Synchronized Cardioversion
- Long-term anticoagulation
Atrial Fibrillation
ectopic rhythm of 350-500BPM
absent P wave
ventricular (QRS) is irregularly regular (Rapid >100BPM) or controlled (<100BPM)
AFib commonly occurs in
- Heart failure
- CAD
- Pulmonary disease
- Severe Mitral Valve Disease
AFIB EKG
Atrial –> indiscernible
350-500BPM
Ventricular –> irregular
controlled or RVR
QRS <0.12
A fib treatment
- Amiodarone
- BB/CCB/Digozin
- cardioversion,** TEE prior/anticoagulation before if stable**
- Long term anticoagulation
- Ablation therapy (radiofrequency)