Hemodynamic Monitoring Flashcards

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1
Q

Radial Artery Catheterization

A

The radial artery is the most common site for invasive blood pressure monitoring because it is technically easy to cannulate and complications are uncommon.

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2
Q

Radial Artery Catheterization Technique

A

Needle Held 30- 45 degrees.
Dorsiflexion of the wrist should be mild at most to avoid attenuating the pulse by stretch or extrinsic tissue pressure. The course of the radial artery proximal to the wrist is identified by gentle palpation, the skin is prepared with an antiseptic, and a local anesthetic is injected intradermally and subcutaneously beside the artery. Arterial catheterization can be performed with a standard IV catheter or an integrated guidewire-catheter assembly designed for this purpose.

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3
Q

Radial Artery Catheterization Care

A

Extreme wrist dorsiflexion following establishment of an A line should be avoided to prevent injury to the median nerve.

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4
Q

Complication Risk: Radial A-line

A
Overall low risk
Increased Risk: 
vasospastic arterial disease
previous arterial injury
thrombocytosis, 
protracted shock
high-dose vasopressor administration,
prolonged cannulation
infection.
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5
Q

Allen Test

A

Technique: compress both the radial and ulnar arteries and asks the patient to make a tight fist, exsanguinating the palm. The patient then opens the hand, avoiding hyperextension of the wrist or fingers. As occlusion of the ulnar artery is released, the color of the open palm is observed.
Normally, the color will return to the palm within several seconds; severely reduced ulnar collateral flow is present when the palm remains pale for more than 6 to 10 seconds.
Predictive value of this test is poor
Ischemic injuries seen even in patients with Normal Allen test

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6
Q

Indications for A-line

A
Elective deliberate hypotension
Wide swings in intra-op BP
Risk of rapid changes in BP
Rapid fluid shifts
Titration of vasoactive drugs
End organ disease
Repeated blood sampling
Failure of indirect BP measurement
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7
Q

A-line Transducer System

A

Transducer system- continuous flush device
System dynamics and accuracy improved by minimizing tube length, limit stop cocks, no air bubbles, the mass of fluid is small, using non compliant stiff tubing
calibration at level of heart (mid-axillary line/right atrium or meatus of ear/circle of Willis if concerned about cerebral perfusion as in sitting pt)

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8
Q

Zeroing and Leveling Transducer

A

All pressures displayed on the monitor are referenced to local atmospheric pressure
The phlebostatic axis is on the 4th intercostal space along the mid axilla line.
The phlebostatic axis is relevant for supine and up to 60 degrees of head-up tilt.
If the transducer has not been levelled to the phlebostatic axis, pressure readings will be either falsely high or falsely low.
A 20-cm difference in height produces a 15-mm Hg difference in pressure
High-low
Low- high

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9
Q

Position and A-line

A

This point must be specifically positioned relative to the patient to ensure correct transducer level. When a significant or unexpected change in pressure occurs, the zero reference value can be rechecked quickly by opening the stopcock and noting that the pressure value on the bedside monitor is still zero.

Despite successful transducer zeroing, the measured blood pressure values seem erroneous, and a malfunctioning pressure transducer, cable, or monitor should be suspected

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10
Q

Underdamped

A

Underdamped- Systolic pressure overshoots and additional small, non-physiologic pressure waves distort the waveform and make it hard to discern the dicrotic notch (If visible, dicrotic notch will be exaggerated).
Several oscillations during square wave test.
Saltatory rather than gradual transitions. Systolic will reported higher and diastolic lower.

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11
Q

Overdamped

A

Overdamped- waveform is recognizable by its slurred upstroke, absent dicrotic notch, and loss of fine detail. Such waves display a falsely narrowed pulse pressure, although MAP may remain reasonably accurate.
One oscillation during square wave test. Dicrotic notch will be hard to visualize. Low systolic and overestimated diastolic.

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12
Q

Causes of Underdamped:

A

Catheter whip or artifact
Stiff non-compliant tubing
Hypothermia
Tachycardia or dysrhythmia

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13
Q

Causes of Overdamped:

A
Loose connections
Air bubbles
Kinks
Blood clots
Arterial spasm
Narrow tubing
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14
Q

Actions to assess and fix damped wave forms:

A
Actions: Damped Waveforms
Pressure bag inflated to 300 mmHg
Reposition extremity or patient 
Verify appropriate scale
Flush or aspirate line
Check or replace module or cable
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15
Q

Square Wave Test

A

Square Wave Testing
Square wave testing can have a direct impact on the validity and accuracy of the hemodynamic values which are obtained from the invasive monitoring device.
It assesses for adequately damped, over-damped, and under damped.
Adequately- damped waveform is when there are only two oscillations that follow the fast-flush wave. Oscillations should be no more than 1/3 the height of the previous oscillation. The subsequent transducing should demonstrate a clear arterial waveform with a discernable dicrotic notch.

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16
Q

Aline Complications

A
Nerve Damage 
Hemorrhage/ Hematoma 
Infection 
Thrombosis 
Air embolus 
Skin necrosis 
Loss of digits 
Vasospasm 
Arterial aneurysm 
Retained guide wire
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17
Q

ASA closed claims for A-lines

A

ASA closed claims for A-lines: 54% were related to radial artery use (ischemic injury, median or radial nerve injury, or retained wire fragment), less than 8% were associated with use of the brachial artery, and the remainder followed severe thrombotic or hemorrhagic complications after femoral artery monitoring

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18
Q

Complications of Direct Arterial Pressure Monitoring

A

Distal ischemia, pseudoaneurysm, arteriovenous fistula Hemorrhage
Arterial embolization
Infection
Peripheral neuropathy Misinterpretation of data Misuse of equipment

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19
Q

Aortic Stenosis

A
Aortic stenosis
Pulsus parvus (narrow pulse pressure) Pulsus tardus (delayed upstroke)
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20
Q

Aortic Regurgitation

A
Aortic regurgitation
Bisferiens pulse (double peak) Wide pulse pressure
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21
Q

Hypertrophic cardiomyopathy

A

Spike and dome (mid-systolic obstruction)

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22
Q

Systolic left ventricular failure

A
Pulsus alternans (alternating pulse pressure amplitude)
inspiration)
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23
Q

Cardiac tamponade

A

Pulsus paradoxus (exaggerated decrease in systolic blood pressure during spontaneous

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24
Q

Pulse pressure variation

A

Pulse pressure variation (PPV) is calculated as the difference between maximal (PPMax ) and minimal (PPMin ) pulse pressure values during a single mechanical respiratory cycle, divided by the average of these two values.

Yields a %- PPV: grey zone of 9% to 13%, such that those below 9% should receive intravascular volume expansion, whereas those above 13% should not. For those between the two values, the measurement is not able to provide meaningful information and the decision should be made on other criteria

During positive pressure ventilation, increases in lung volume compress lung tissue and displace blood contained within the pulmonary venous reservoir into the left heart chambers, thereby increasing left ventricular preload. Simultaneously, the increase in intrathoracic pressure reduces left ventricular afterload. The increase in left ventricular preload and decrease in afterload produce an increase in left ventricular stroke volume, an increase in cardiac output, and in the absence of changes in peripheral resistance, an increase in systemic arterial pressure. In most patients the preload effects are more prominent, but in patients with severe left ventricular systolic failure, the reduction in afterload plays an important role in increasing ventricular ejection.

To measure PPV accurately: In general, mechanical ventilation with tidal volumes of 8 to 10 mL/kg, positive end-expiratory pressure of 5 mm Hg or greater, regular cardiac rhythm, normal intraabdominal pressure, and a closed chest are necessary to duplicate the experimental conditions.

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25
Q

Pulse Oximeter

A

Method of measuring hemoglobin oxygen saturation (SpO2)
Noninvasive
Measures transmission of light through a solution to the concentration of the solute in the solution
Application of Beer-Lambert Law
Typically used wavelengths of light are 660 and 940 nm
Pulse oximeter probe is composed of a light emitter and a photodetector
ASA/ AANA Standards for Basic Monitoring requires that variable pitch tone be audible when in use

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26
Q

Pulse Oximetry Uses:

A
Detection of hypoxemia
Detection of perfusion
Sites: fingers, toes, nose, ear, tongue, cheek
Inaccuracy
Malposition of probe
Dark nail polish
Different hemoglobin
Dyes
Electrical interference
Shivering
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27
Q

Indications for Central Venous Cannulation

A

Central venous pressure monitoring
Pulmonary artery catheterization and monitoring Transvenous cardiac pacing
Temporary hemodialysis
Drug administration
• Concentrated vasoactive drugs
• Hyperalimentation
• Chemotherapy
• Agents irritating to peripheral veins
• Prolonged antibiotic therapy (e.g., endocarditis)
Rapid infusion of fluids (via large cannulas) • Trauma
• Major surgery
Aspiration of air emboli
Inadequate peripheral intravenous access Sampling site for repeated blood testing

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28
Q

Indications for Central Venous Cannulation

A

Central venous pressure monitoring
Pulmonary artery catheterization and monitoring Transvenous cardiac pacing
Temporary hemodialysis
Drug administration
• Concentrated vasoactive drugs
• Hyperalimentation
• Chemotherapy
• Agents irritating to peripheral veins
• Prolonged antibiotic therapy (e.g., endocarditis)
Rapid infusion of fluids (via large cannulas) • Trauma
• Major surgery
Aspiration of air emboli
Inadequate peripheral intravenous access Sampling site for repeated blood testing

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29
Q

Insertion Sites for CVCs

A
*Right internal jugular vein
Left internal jugular vein
Subclavian veins
External jugular veins
Femoral veins
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30
Q

RIJ Prefered

A

Reasons for this preference include the consistent, predictable anatomic location of the internal jugular vein, readily identifiable and palpable surface landmarks, and a short straight course to the superior vena cava. An internal jugular vein catheter is highly accessible during most surgical procedures and has a high rate of successful placement (90% to 99%)

31
Q

LIJ

A

Cupola of the pleura is higher on the left, theoretically increasing the risk of pneumothorax. The thoracic duct may be injured during the procedure as it enters the venous system at the junction of the left internal jugular and subclavian veins.129 The left internal jugular vein is often smaller than the right and demonstrates a greater degree of overlap of the adjacent carotid artery.130 Most important, any catheter inserted from the left side of the patient must traverse the innominate (left brachiocephalic) vein and enter the superior vena cava perpen- dicularly. As a result, the catheter tip may impinge on the right lateral wall of the superior vena cava, increasing the risk of vascular injury. This anatomic disadvan- tage pertains to all left-sided catheterization sites and highlights the need for radiographic confirmation of proper catheter tip location. Finally, most operators have less experience performing left internal jugular vein cannulation, which leads to more adverse events and morbidity.

32
Q

SC

A

Pneumothorax is the most common complication of subclavian vein cannulation, although unintended arterial puncture may actually be more frequent.

33
Q

CVCs

A

7 french, 20 cm length, Multiport catheters most common
Placement usually not confirmed by XRAY in OR
Aspirate blood from all ports
After surgery, XRAY
Ideally, tip within the SVC, just above junction of venae cavea and the RA, parallel to vessel walls, positioned below the inferior border of clavicle and above the level of 3rd rib, the T4/T5 interspace, the carina, or takeoff right main bronchus

34
Q

CVC Contraindications

A

R atrial tumor
Contralateral pneumothorax
Infection at site

35
Q

Complications of Central Venous Pressure Monitoring

A

Mechanical Vascular injury
Arterial
Venous
Cardiac tamponade
Respiratory compromise
Airway compression from hematoma Pneumothorax
Nerve injury
Arrhythmias Thromboembolic
Venous thrombosis
Pulmonary embolism
Arterial thrombosis and embolism Catheter or guidewire embolism
Infectious
Insertion site infection Catheter infection Bloodstream infection Endocarditis
Misinterpretation of data Misuse of equipment

36
Q

CVP monitoring

A

CVP pressures = RAP = RV preload.
CVP in a spontaneously breathing patient is approximately 2-7 mmHg
This rises about 3-5 mmHg during mechanical ventilation

37
Q

CVP Waveform

A

5 Phasic Events
Three Peaks (a, c, v)
Two Descents (x, y)
Requires an understanding of the cardiac cycle

38
Q

“a” wave

A

Caused by atrial contraction (follows the P-wave on EKG)
End diastole
Corresponds with “atrial kick” which causes filling of the right ventricle.

39
Q

“c” wave

A

Atrial pressure decreases after the “a” wave as a result of atrial relaxation
The “c” wave is due to isovolumetric right ventricular contraction; tricuspid valve closed bulges back into the right atrium
Occurs in early systole (after the QRS on EKG)
The c wave always follows the ECG R wave because it is generated during onset of ventricular systole.

40
Q

“x” descent

A

Systolic decrease in atrial pressure due to atrial relaxation
Mid-systolic event

41
Q

“v” wave

A

ventricular ejection, which drives venous filling of the atrium
occurs in late systole with the tricuspid still closed
occurs just after the T-wave on EKG

42
Q

“y” descent

A

Diastolic decrease in atrial pressure due to flow across the open tricuspid valve

43
Q

Pulmonary Artery Pressure Monitoring is use for

A
Right-sided heart catheter used for direct bedside assessment of :
Intracardiac pressures (CVP, PAP, PCWP/ PAWP)
Estimate LV filling pressures
Assess LV function
CO 
Mixed venous oxygen saturation
PVR and SVR
Pacing options
44
Q

Describe the make up Pulmonary Artery Pressure Catheters

A
7 french (introducer is 8.5 french)
110 cm length marked at 10 cm intervals
4 lumens
distal port PAP
second port 30 cm more proximal CVP
third lumen balloon
forth wires for temp thermister
45
Q

Pulmonary Artery Pressure Monitoring indications

A
Indications:
LV dysfunction
Valvular disease
Pulmonary HTN
CAD
ARDS/ resp failure
Shock/sepsis
ARF
Surgical procedure: cardiac, aortic, OB
46
Q

Pulmonary Artery Catheter Complications

A

Arrhythmias (including V-fib, RBBB, complete heart block)
Catheter knotting
Balloon rupture
Thromboembolism; air embolism
Pneumothorax
PA rupture
Infection (endocarditis)
Damage to cardiac structures (valves, etc.)
Relative Contraindications– WPW syndrome, Complete LBBB

47
Q

Vena cava and RA junction

A

15

48
Q

Right atrium

A

15-25

49
Q

Right ventricle

A

25-35

50
Q

Pulmonary artery

A

35-45

51
Q

Wedged in pulmonary capillary

A

40-50

52
Q

PCWP Waveform

‘a’ wave

A

The represents contraction of the left atrium. Normally it is a small deflection unless there is resistance in moving blood into the left ventricle as in mitral stenosis.

53
Q

PCWP Waveform

‘c’ wave

A

The ‘c’ wave is due to a rapid rise in the left ventricular pressure in early systole, causing the mitral valve to bulge backward into the left atrium, so that the atrial pressure increases momentarily.

54
Q

PCWP Waveform

‘v’ wave

A

The ‘v’ wave is produced when blood enters the left atrium during late systole.
Prominent ‘v’ wave reflect mitral insufficiency causing large amounts of blood to reflux into the left atrium during systole.

55
Q

Cardiac Output Monitoring

A
Thermodilution
Continuous thermodilution
Mixed venous oximetry
Ultrasound
Pulse Contour
56
Q

Transesophageal Echocardiography

A
7 Cardiac Parameters Observed
Ventricular wall characteristics and motion
Valve structure and function
Estimation of end-diastolic and end-systolic pressures and volumes (EF)
CO
Blood flow characteristics
Intracardiac air
Intracardiac masses
57
Q

Transesophageal Echocardiography Uses in OR

A
Uses in the OR:
Unusual causes of acute hypotension
Pericardial tamponade
Pulmonary embolism
Aortic dissection 
Myocardial ischemia
Valvular dysfunction
Valvular function
Wall motion
58
Q

Transesophageal Echocardiography complications

A
Complications:
Esophageal trauma
Dysrhythmias
Hoarseness
Dysphagia
59
Q

AANA Standard 9: Monitoring and Alarms

A

Monitor, evaluate, and document the patient’s physiologic condition as appropriate for the procedure and anesthetic technique.
When a physiological monitoring device is used, variable pitch and threshold alarms are turned on and audible.
Document blood pressure, heart rate, and respiration at least every five minutes for all anesthetics.
a. Oxygenation Continuously monitor oxygenation by clinical observation and pulse oximetry. The surgical or procedure team communicates and collaborates to mitigate the risk of fire.
b. Ventilation: Continuously monitor ventilation by clinical observation and confirmation of continuous expired carbon dioxide during moderate sedation, deep sedation or general anesthesia. Verify intubation of the trachea or placement of other artificial airway device by auscultation, chest excursion, and confirmation of expired carbon dioxide. Use ventilatory monitors as indicated.
c. Cardiovascular: Monitor and evaluate circulation to maintain patient’s hemodynamic status. Continuously monitor heart rate and cardiovascular status. Use invasive monitoring as appropriate.
d. Thermoregulation: When clinically significant changes in body temperature are intended, anticipated, or suspected, monitor body temperature. Use active measures to facilitate normothermia. When malignant hyperthermia (MH) triggering agents are used, monitor temperature and recognize signs and symptoms to immediately initiate appropriate treatment and management of MH
e. Neuromuscular When neuromuscular blocking agents are administered, monitor neuromuscular response to assess depth of blockade and degree of recovery.

60
Q

Cornerstones of Monitoring: Physical Assessment

A

Inspection, palpation, and auscultation
Examples:
Observing chest rise and fall & auscultation of breath sounds pre-op, after intubation, and when ventilatory parameters change
direct palpation of a pulse when a monitor value is questionable
direct observation of a beating heart during cardiac surgery or palpation of the aorta by the surgeon
inspection of mucous membranes, skin color, and skin turgor
hydration, oxygenation, and perfusion
inspecting the surgical field for blood loss, observing urine output
evaluating JVD
pupillary response
MANY other examples

61
Q

Precordial or Esophageal Stethoscope

A

Minimally invasive, cost-effective continuous monitor
Continual assessment of breath sounds and heart tones
Precordial placed on chest surface
Esophageal placed 28-30 cm into esophagus
Very sensitive monitor for bronchospasm, airway obstruction, changes in HR/ rhythm

62
Q

Electrocardiogram

Interval

A

If you need a more in-depth review of ECG, please read Miller Chapter 47 “Electrocardiography, Perioperative Ischemia, and Myocardial Infarction”

The lines are 1 mm apart, with every fifth line intensified. The speed of the paper is standardized to 25 mm/sec. On the horizontal axis, 1 mm represents 0.04 second, and 0.5 cm represents 0.20 second. On the vertical axis, 10 mm represents 1 microvolt (mV). On every recording, a 1-cm (1-mV) calibration mark should indicate that the ECG is appropriately calibrated (MIller’s Anesthesia (8th ed) p. 1430).

63
Q

Electrocardiogram purpose

A
Purpose- 
detect arrhythmias
monitor heart rate
detect ischemia
detect electrolyte changes
monitor pacemaker function

Accurate detection of the R wave and measurement of the R wave–R wave interval serve as the basis from which digitally displayed values are derived and periodically updated (e.g., at 5- to 15-second intervals)- can lead to inaccuracies

64
Q

Proper ECG Placement

A

3-Lead ECG
Electrodes RA, LA, LL
Leads I, II, III
3 views of heart (no anterior view)

65
Q

Three-electrode monitoring

A

Is usually adequate for tracking HR, detecting R waves for synchronized direct-current shock in cardioversion, and detecting ventricular fibrillation. However, it is inadequate for diagnosing more complex arrhythmias for which a true V1 lead is necessary, such as to distinguish between right versus left bundle branch block (LBBB) or ventricular tachycardia (VT) and supraventricular tachy- cardia (SVT) with aberrant ventricular conduction. The three-electrode system is also inadequate for ST-segment monitoring because it does not provide multilead monitoring or precordial leads, which are often most sensitive for detecting ischemia.

66
Q

5-Lead ECG

A

V1 is the preferred lead for special arrhythmia monitoring, whereas the other precordial leads, especially V3 to V5, are the preferred leads for ischemia monitoring. The five electrode monitoring system is currently the standard for monitoring patients with suspected perioperative myocardial ischemia

67
Q

Proper ECG Placement

A

A majority of the dysrhythmias and ischemia seen during anesthesia can be detected by a combination of monitoring leads II and V5.

68
Q

Gain Setting and Frequency Bandwidth

A

Gain should be set at standardization
1 mV signal produces 10-mm calibration pulse
Therefore, a 1-mm ST segment change is accurately assessed
Filtering capacity should be set to diagnostic mode
Filtering out the low end of frequency bandwidth can distort ST segment

69
Q

ECG for Ischemia Detection

5 Principle Indicators:

A
ST segment elevation , ≥1mm 
T wave flattening or inversion 
Development of Q waves 
ST segment depression, flat or downslope of  ≥1mm
Peaked T waves 
Arrhythmias
70
Q

Inferior wall ischemia

A

(right coronary artery) Changes in Lead II, III, AVF

71
Q

Lateral wall ischemia

A

(circumflex branch of left coronary artery) Changes in Lead I, AVL, V5-V6

72
Q

Anterior wall ischemia

A

(left coronary artery) Changes in V3-V4

73
Q

Septal ischemia

A

(left descending coronary artery) Changes in Lead V1-V2