Heart blocks and heart failure Flashcards

1
Q

What is the electrocardiograph?

A

The machine which produces an ECG recording

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2
Q

What are the standard limb leads used for an ECG?

A

Right arm, Left arm and left leg

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3
Q

Where is lead 1?

A

Between the right and left arm?

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4
Q

Where is lead 2?

A

Between the right arm and left leg?

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5
Q

Where is lead 3?

A

Between the left arm and the left leg

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6
Q

Which lead shows the biggest deflection?

A

Lead 2 as depolarisation occurs in this direction

Lead 1 = smallest

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7
Q

How long is the PR interval?

A

0.12-0.2 seconds

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8
Q

How long is the QRS complex?

A

0.08-0.1 seconds

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9
Q

How long is the QT interval?

A

0.4-0.43 seconds

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10
Q

How long is the ST interval on average?

A

0.32 seconds

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11
Q

What is a high p wave amplitude a result of?

A

Atrial hypertrophy - more muscle mass = bigger depolarisation = bigger deflection

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12
Q

What does a low T amplitude show?

A

Ventricular hypoxia - not enough oxygen present for ventricles to contract

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13
Q

What is sinus arrhythmia?

A

Heart rate increases by 15% during inspiration - seen less in adults

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14
Q

What are first degree heart blocks?

A

Interruption somewhere between the SA and AV nodes slowing down conduction between the two nodes
Longer PR interval

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15
Q

What is mobitz type 1?

A

Progressive prelongation of the PR interval culminating in a non-conducted p wave
The PR interval is longest immediately before the dropped beat and shortest immediately after
P waves continue at the same rate

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16
Q

How can you tell the difference between mobitz type 1 and mobitz type 2?

A

Mobitz 1 = PR intervals change

Mobitz 2 = PR intervals do not change

17
Q

What is mobitz 2?

A

Usually due to the failure of conduction at the level of the His purkinje system
More likely to be due to structural damage to the conducting system

18
Q

How is mobitz 2 all or nothing?

A

His -purkinje cells suddenly and unexpectedly fail to conduct a supra ventricular impulse

19
Q

What is a third degree heart block?

A

Complete absence of AV concution - none of the supraventricular impulses are conducted to the ventricles
The perfusing rhythm is maintained by a junctional or ventricular escape rhythm
Alternatively the patient may suffer ventricular standstill = syncope or sudden cardiac death
Typically the patient will have severe bradychardia with independent atrial and ventricular rates ie AV dissociation

20
Q

What is sick sinus syndrome?

A

Impaired SA node firing eg brady or tachycardia

21
Q

What is happening if the jugular vein is pulsing?

A

Atria are contracting against a closed tricuspid - sends a cannon wave in the jugular vein
The right atria and ventricle contract at the same time so a pressure wave is sent back up the jugular veins

22
Q

What can atrial fibrilation cause?

A
Rapid/irregular heart rates
Exercise intolerance
Angina
Shortness of breath
Oedema of the ankles
23
Q

How can atrial fibrilation be treated?

A
Flecainide
Beta blockers, especially sotalol
Amiodamine
Dronedarone (only for certain people)
Warfarin and other anticoagulants - stop clots forming if the blood is pooling as this could cause a pulmonary embolism
24
Q

What is a circus movement?

A

An electrical signal not completing the normal circuit but rather an alternative circuit looping back on itself rapidly

25
Q

How can refractory muscle become non-refractory?

A

Unidirectional block

Transient biderectional block

26
Q

What are the stages in re-entrant excitation?

A

1) The originating impulse will set up the re-entrant excitement amd the unidirectional conduction block prevents further transmission
2) System behaves as an independent pacemaker with a rate higher than the originating impulse
3) The wave of excitation can travel in the reverse direction
4) The block allows retrograde transmission