Heart and Vessels Flashcards
ather/o
fatty substance
arter/o, arteri/o
artery
atri/o
atrium
cardi/o
heart
coron/o
heart
pericardi/o
pericardium
rhytm/o
rhythm
sphygm/o
pulse
steth/o
chest
vas/o
vessel
vascul/o
vessel
ven/o, ven/i
vein
ventricul/o
ventricle
pericardial sac (parietal pericardium)
anchors heart to great vessels (aorta and venae cavae)
epicardium (visceral pericardium)
serous membrane covering heart’s surface
secretes pericardial fluid
myocardium
heart’s middle layer composed of cardiac muscle tissue
endocardium
heart’s inner most layer, lines the 4 chambers of the heart
coronary sulcus
marks separation of atria from ventricles
interventricular sulcus
mark separation of left and right ventricles
interatrial septum
separates two atria from each other
interventricular septum
separate ventricles from one another
pulmonary circuit
heart pumping blood to the lungs and back
CO2 is unloaded and O2 is loaded
systemic circuit
once blood is returned from lungs, left side of heart pumps blood to all parts of body to be returned back to heart
O2 is unloaded and CO2 is loaded
Steps of cardiac conduction system
- heartbeat started by SA node
- from SA node, cardiac muscle cells carry electrical impulse across myocardium of both atria causing depolarization
- while step 2 happens, other cardiac cells cary impulse to AV node
- AV bundle carries impulse down interventricular septum to apex
- Purkinje fibers fan out from AV bundle stimulating cardiac muscle cells to depolarize and contract
Phases of cardiac cycle
- Atrial systole: SA node fires, atria depolarize and contract together. increased pressure pushes blood through AV valves to ventricles
- Atrial diastole: atria repolarize and relax. pressure in superior/inferior venae cavae and pulmonary veins is greater than atrial pressure so blood rushes in
- Ventricular systole: once impulse is carried from AV node to purkinje fibers ventricles depolarize and contract together. chordae tendinae also contract to prevent backflow. contraction of ventricles decreases volume and increases pressure inside ventricles. blood pushed through pulmonary and aortic valves.
- Ventricular diastole: ventricles repolarize and relax. blood moves from AV valves to ventricles from atria. all 4 chambers fill with blood during this phase. atria are not contracting during this phase. blood is passively moving from atria to ventricles due to difference in pressure
sinus rythm
normal pace, with 70-80 bpm
vagal tone
a pace normally kept in check by autonomic nervous system through vagus nerve
ectopic focus
occurs when any part of conduction system other than SA node sets pace
nodal rhythm
occurs if AV node is ectopic focus
P wave
shows depolarization of atria
Q, R, S waves
together represent ventricles depolarizing
T wave
represents ventricles repolarizing
What event does normal ECG not show?
atria repolarizing; this happens the same time ventricles are depolarizing which is a much stronger event
cardiac output
amount of blood ejected by each ventricle of the heart each minute
calculated by multiplying heart rate by stroke volume
stroke volume
amount of blood ejected from each ventricle per beat
cardiac reserve
difference between the cardiac output of a heart at rest and the maximum cardiac output the heart can achieve
3 factors that affect stroke volume
preload: amount of tension in the myocardium of ventricular walls
contractility: responsiveness of cardiac muscle to contract
afterload: concerns pressure in pulmonary trunk and aorta during diastole
frank-starling law of heart
states heart must pump out the amount of blood it receives
chronotropic factor
anything that changes the heart rate
positive increase heart rate, negative decrease heart rate
cardiac accelerator center
uses sympathetic neurons to stimulate the SA and AV nodes to speed up heart rate
cardiac inhibitory center
uses parasympathetic neurons of vagus nerve to keep SA node at 70-80 bpm (vagal tone)
if vagus nerve is severed, SA node typically sets pace at 100/bpm
3 types of sensors that feed information to centers in medulla oblongata
proprioceptors, baroreceptors, chemoreceptors
proprioceptors
located in body’s muscles, joints, and tendons
information they send alerts centers to any change in body’s activity level
baroreceptors
located in aorta and carotid arteries
alert the centers to any changes in bp
if bp falls, cardiac accelerator center stimulates SA and AV nodes to increase heart rate in an effort to restore bp to homeostasis
chemoreceptors
monitor pH, carbon dioxide and oxygen in blood
located at aortic arch, on carotid arteries, and in medulla oblongata
much more important for setting respiratory rate
chronotropic effects of chemicals
positive effects: epinephrine, caffeine, norepinephrine, nicotine, and thyroid hormone
negative effects: potassium ions
arteries
carry blood away from heart to capillaries
capillaries
allow for exchange of materials between blood and tissues
veins
deliver blood from capillaries back to heart
tunica externa
outermost layer of vessel wall
tunica media
middle layer of vessel wall
thickest layer
more muscular in arteries than veins of comparable size
may be elastic fibers in this layer depending on vessel
tunica interna
lining of vessel wall
vital that this layer be smooth and secrete chemical to repel platelets
conducting arteries
largest of the arteries (aorta, pulmonary arteries, etc)
carry blood away from heart
need to withstand high pressure, therefore have most muscle/elastic fibers in their walls for expansion
distributing arteries
medium-sized
distribute blood away from conducting arteries to organs
examples are hepatic artery, and renal arteries
resistance arteries
smallest of the arteries
examples are arterioles that deliver blood to capillaries
coronary route
heart’s own circulation route composed of coronary arteries and veins
right and left coronary arteries lead to capillary beds in heart’s tissue
20% of blood from capillaries directly returned to R atrium of heart
systemic route
carry blood from heart to tissues in the body and back again
what are the 2 types of alternative routes?
portal routes and anastomoses
portal routes
contains 2 capillary beds before blood is returned to heart
allows materials to be exchanged twice between blood and tissues before returning to heart
example: hepatic portal route
hepatic portal route
route between intestines and liver
blood travels from heart to arteries to capillary beds in sm intestine/other digestive organs. digested nutrients absorbed into blood through capillaries. blood travels through small veins leading to hepatic portal vein to capillary beds in liver, where nutrients are processed. blood exits liver via hepatic vein on its way back to heart.
anastomoses
involves vessels merging together
3 types
arteriovenous anastomoses
often called a shunt
merges an artery with a vein, skipping capillary bed
used in fingers, palms, toes and ears in condition of extreme cold
protective mechanism to avoid losing heat
arterial anastomoses
merges 2 arteries together to provide collateral routes to same area
can be found in heart, to make sure all parts of heart are adequately fed, and at joints, where movement may block one of routes
venous anastomoses
most common anastomoses
merges veins to drain an organ
venous return
5 mechanisms aid in venous return:
pressure gradient, gravity, thoracic pump, cardiac suction, skeletal muscle pump
pressure gradiant
pressure in veins due to action of heart propels blood toward heart
gravity
blood moves through veins above heart due to gravity and flows downhill
thoracic pump
chest expands every time breath is inhaled increasing the volume and decreasing pressure within chest
as air rushes into lungs to equalize pressure, blood in veins in abdominal cavity is sucked into inferior vena cava
cardiac suction
atria return to shape during atrial diastole creating less pressure in atria than in superior and inferior venae cavae and pulmonary veins, so blood is sucked into atria
skeletal muscle pump
especially effective in limbs
skeletal muscle action massages blood through the veins, while valve in veins prevent backflow
What 3 ways does resistance make a difference in terms of blood pressure?
viscosity (thickness): amount of albumins and RBCs determine blood thickness
vessel length: the greater the vessel length, the more friction that occurs between blood and vessel walls. friction slows blood.
vessel radius: the smaller the radius, the more blood comes in contact with walls of vessel. radius can be controlled in several different ways to regulate bp (vasodilation/vasoconstriction)
pulse pressure
indicates the surge of pressure small arteries must withstand with each ventricular contraction
equation: sytolic pressure minue diastolic pressure (in mmHg)
pulse pressure increases as stroke volume increases
mean arterial pressure (MAP)
average pressure arteries must be able to withstand
determined by equation: diastolic pressure plus 1/3 of pulse pressure