Health Psychology Flashcards

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1
Q

3 Types of Non-Adherence: Donovan & Blake (1992)

A
  1. RATIONAL: Deliberately non-adherent
  2. UNWITTING: Not your fault, a misunderstanding
  3. ERRATIC: Forgetful (not your fault) or you run out (your fault)
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2
Q

Why do we rationally decide not to adhere? - Sarafino et al, 1994

A
  • UNPLEASANT OR UNDESIRABLE SIDE EFFECTS
  • BELIEVE TREATMENT IS NOT WORKING
  • PRACTICAL BARRIERS (COST)
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3
Q

Why do we rationally decide not to adhere?

A
  • MULTIPLE PHYSICIANS (Vlasnik et al., 2005) - Decrease patients’ confidence in the prescribed treatment
  • FEAR OF DEPENDENCE (Apter et al., 2003) - Fears of addictive behaviour and adverse side effects
  • RELIGIOUS BELIEFS (Lim & Ngah, 1991) - Preference for natural remedies
  • NO WAY, NOT THAT PHARAMCEUTICAL COMPANY - Not wanting pharmaceutical companies, believing they are profit driven, to benefit
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4
Q

Factors Affecting Erratic (non-deliberate) Non Adherence

A
  • MEMORY: Do patients recall what they have been told?
  • Ley et al (1979) - Asked patients to recall a series of statements presented in two ways:
    1. UNSTRUCTURED
    2. STRUCTURED
    = Recall was 25% better for structured information, and was also better for information presented at the beginning of the consultation (primacy effect) – Perhaps therefore, better to emphasise most important information at the start?
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5
Q

Medical Non Adherence: Implications

A
  • SOKOL ET AL (2005): Relationship between adherence & risk for hospitalisation in diabetes patients – More non-adherence related to greater overall cost
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6
Q

Pain, why do we have it?

A
  • It’s good for us…
    1. Helps to regulate daily activities; it provides us low level feedback about bodily systems, and, by doing so, lets us know when to make adjustments (e.g., shift posture, turn over when sleeping)
    2. It also, when it becomes too intense, leads us to seek treatment
  • Experience before serious injury (e.g., picking up something hot)
  • Sets a limit on activities to avoid damage – lets us know when enough is enough
  • Helps us, by way of reinforcement, avoid noxious stimuli in the future (e.g., hot)
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7
Q

Can we define pain? In two ways, either by its duration or nature

A
  • DURATION
    ∙ Acute (0-6 months):
    ✳︎ Examples such as stubbing toe or breaking bone
    ✳︎ Can be recurrent, e.g., migraines
    ∙ Chronic (more than 6 months):
    ✳︎ Can be either benign, as pain experienced to a similar intensity, like back pain, or can be progressive, as pain that worsens with time, like arthritic pain
  • NATURE - Nature can be subdivided into three categories: severity, pattern and type
    ∙ Severity: From mild discomfort - Excruciating
    ∙ Type: Stabbing, shooting, piercing, throbbing, dull, aching
    ∙ Pattern: Brief, continuous, sporadic, intermittent, kinetic
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8
Q

Can we measure pain?

A
  • Pain, as a subjective experience, is difficult to measure, and what is painful to one person might not be painful, or be less painful, to another, and reactions of different people to the same pain inducing agent will vary
  • We can ask about the pain, using questionnaires and diaries, and can, as HCP, take into account pain behaviours, like facial grimaces, changes in posture and audible expressions. Some studies, to better understand how people respond to pain, and how individual differences, liker personality and mood, affect pain responses, have artificially induced pain in the lab, using pain induction methods like cold pressor, and measured responses
  • McGill Pain Questionnaire
  • Lab based pain induction technique - Cold Pressor
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9
Q

Biological theories of pain

A
  • Specificity theory

- Pattern theory

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10
Q

Biological theories: limited?

A
  • Specificity & Pattern theories = Pain is picked up by receptors in the skin that relay this information, via neurons and their chemical messengers, to the brain, which processes this pain related information, and responds.
  • LIMITATIONS:
    ✳︎ PAIN, BUT NO RECEPTORS
    ✳︎ RECEPTORS, BUT NO PAIN
    ✳︎ PSYCHOLOGICAL FACTORS
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11
Q

Pain but no receptors

A
  • Phantom Limb Pain: Experiencing pain in the absence of pain receptors, i.e., pain as if from an absent limb
    (similar for those with spinal cord injuries/paralysis = 70% experience phantom pain)
  • Explain? - Neural Plasticity?
    ✳︎ Alton Towers victim
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12
Q

Receptors but no pain

A
  • Congenital Analgesia (Congenital Universal Insensitivity to Pain - CUIP)
    A rare genetic condition where, despite an intact pain pathway, people are born with inability to feel pain
    ✳︎ Mrs C
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13
Q

Psychological Factors: Attention

A
  • EPISODIC ANALGESIA:
  • Pain perception fails to occur immediately post injury
  • Prevalent in competitive sports men and women, and in the context of war, e.g.,
  • Possibly due to:
    1. Distraction/Attention (see
    week 3)
    2. Physiological (sympathetic)
    arousal = diminished pain
    sensitivity
  • James et al, (2002) = our response to and perception of pain is variable - 1) focus on pain: tolerate pain for short time, 2) distracted from pain: can tolerate pain longer (aka keep arm in freezing water longer)
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14
Q

Psychological Factors: Meaning

A
  • Beecher et al (1959): the meaning we give to the injury or pain, and the commensurable emotional response, positive or negative, can influence our pain perception and reporting
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15
Q

Psychological Factors: Mood

A
  • Pinerua et al (1999): Depression & Pain (Cold Pressor)

- those with depression were more likely to rate the experience as painful

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16
Q

Bio-Psycho-Social Model of Pain: Gate Control Theory

A
  • Similar to Specificity and Pattern theories, information from pain receptors is relayed to the brain via neurons and their chemical messengers HOWEVER PAIN GATE IN SPINAL COLUMN
  • OPENS GATE: negative emotions, focussing on the pain, boredom, no medication
  • CLOSES GATE: positive emotions, distraction, medication, relaxation
17
Q

Advantages of Gate Control

A
  • Integrates psychological factors into biological models of pain
  • Allows for individual variability in pain perception (i.e., degree gate is open or closed)
  • ALLOWS FOR PSYCHOLOGICAL INTERVENTIONS IN PAIN TREATMENT…Increase positive emotions and close the gate!
18
Q

STRESSOR (i.e., stimulus)

A
  • Physical, e.g., too hot, cold

- Psychological, e.g., financial worries, relationship problems

19
Q

RESPONSE (i.e., reaction to stimulus)

A
  • Psychological (i.e., emotional): such as > anxiety and < in calm
  • Physiological such as increase in heart rate, blood pressure etc
20
Q

Classic Models (1)… Fight or Flight: Cannon

A
  • Stress → SNS → F/F
21
Q

Classic Models (2) … General Adaptation Syndrome (GAS): Selye

A
  • He said our response to stress is the general one, and it not modified by duration
22
Q

Transactional Model

A

Stress reaction is determined by the interaction between how we appraise events in the environment & our ability to cope with them

23
Q

How coping affects stress responses? - -Kirschbaum et al (1995): Cortisol response to a public speaking stressor

A
  • Stress = Contingent on Coping
  • First day of doing something stress = high cortisol levels
  • Following days = low levels of cortisol because of habituation
24
Q

Stress Pathways

A
  • SAM axis

- HPA axis

25
Q

Stress over the years

A
  • Cavemen era - stressed out by ST physical emergencies like having to fight/flee for our life - e.g. running away from a mammoth
  • Modern times - stressed out by psychological things that are impossible to F/F, and keep coming back
    ✳︎ While things that stress us out have changed, SR has not – constantly churning out stress hormones (adrenaline/cortisol) that are constantly gearing up the CV and metabolic systems for vigorous physical activity (F/F) without any physiological need to do so.
    ✳︎ This is when stress becomes a problem – sustained activation of stress system causes damage to target cells/tissues
26
Q

Modern Day Stress?

A
  • A long time ago: STRESS RESPONSE ADAPTIVE

- Modern day: STRESS RESPONSE HARMFUL

27
Q

Can we measure stress?

A
  • Psychological: questionnaires

- Physiological: CV parameters

28
Q

Measuring stress

A
  • Diurnal cortisol profile: CAR, Diurnal Cortisol Slope
29
Q

Measuring the Effects of Stress

A
  • To assess the psycho-physiological mechanisms that explain the negative effects of stress on health, it is necessary to test people while they are stressed…
    1. Naturalistic Stressors
    2. Controlled Lab Stressors
30
Q

Naturalistic Stressors (Chronic)

A
  • Informal caregiving
  • Bereavement
  • Natural disasters
  • Low SES
  • Exams
  • Divorce
    = GOOD EXTERNAL (ECOLOGICAL) VALIDITY, BUT POOR CONTROL
31
Q

Lab Based Stressors (Acute)

A
  • Public speaking
  • Stroop task
  • Mental arithmetic
  • Mirror tracing
  • Noise manipulation
    = GOOD CONTROL, BUT POORER EXTERNAL (ECOLOGICAL) VALIDITY
32
Q

Coping with Stress?

A
  • Individual differences
33
Q

What about Personality - Does it affect stress?

A
  • Kobasa (1979): HARDINESS, a personality type characterised by CCC, tends to be associated with more adaptive psycho-physiological responding to stress…
    1. Commitment
    2. Control
    3. Challenge