health and healthy in reduced periodontium Flashcards

1
Q

biologic width

A

epithelial attachment plus supracrestal connective tissue attachment

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2
Q

pocket depth

A

gingival margin to base of sulcus

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3
Q

CAL formula

A

PD + recession

PD-overgrowth

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4
Q

what determines treatment needs?

A

probing pocket depth

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5
Q

what estimates tissue destruction?

A

clinical attachment level

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6
Q

what measures inflammation and disease activity?

A

bleeding on probing

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7
Q

periodontal indices

A

CAL
PD
BOP

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8
Q

inflammation shown by?

A

plaque

BOP

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9
Q

systemic condition

A

smoking
diabetes
disease affecting periodontium

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10
Q

periodontal health, gingival diseases and conditions

A
  • periodontal health and gingival health
  • gingivitis: dental biofilm-induced
  • gingival disease: non-dental biofilm induced
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11
Q

what is the biology of clinical gingival health?

A

generally associated with an inflammatory infiltrate and a host response consistent with homeostasis

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12
Q

on a site level, how do we classify clinical gingival health?

A
  1. clinical gingival health on an intact periodontium
  2. clinical gingival health on a reduced periodontium
    - -stable periodontitis patient
    - -non-periodontitis patient (e.g. recession, crown lengthening)
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13
Q

what does the periodontium consist of?

A
  • pdl
  • gingiva
  • bone
  • cementum
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14
Q

clinical features of health on an intact periodontium

A
  • no attachment loss
  • probing depths less than 3 mm
  • less than 10% BOP
  • no bone loss–1.0-3.0 mm apical to CEJ
  • no symptoms
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15
Q

clinical features of health on reduced periodontium

A
  • no BOP, erythema, edema, or symptoms
  • reduced clinical attachment and bone levels
  • probing attachment loss
  • pockets less than 3 mm
  • less than 10% BOP
  • possible bone loss
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16
Q

intact periodontium

A

NO loss of periodontal tissue

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17
Q

reduced periodontium

A

a periodontium with pre-existing loss or periodontal tissue and bone, but not progressive, ongoing loss of attachment

18
Q

examples of attachment loss/bone loss due to non-periodontitis reasons

A
  • ortho
  • soft tissue recession after surgical crown lengthening
  • gingival abrasion with tooth brushing
  • cervical dental caries
  • physiologic bone resorption in edentulous area
19
Q

dental plaque biofilm-induced gingivitis

A

an inflammatory lesion from plaque biofilm and hosts immune response
-remains within gingiva and does not extend to periodontal attachment (cementum, periodontal ligament, alveolar bone)

20
Q

signs of gingivitis

A
  • swelling
  • bleeding on probing
  • redness
  • discomfort on gentle probing
21
Q

symptoms of gingivitis

A
  • bleeding gums
  • pain
  • halitosis
  • difficulty eating
  • appearance
  • reduced oral health – related to quality of life
22
Q

T/F radiographs can be used to diagnose gingivitis

A

false

you can’t see bone loss bc it’s just soft tissue

23
Q

classifications of gingivitis

A
  • gingivitis on intact perio
  • gingivitis on reduced perio in non-perio patient
  • ging inflammation on reduced perio in a successfully treated perio patient
24
Q

what is the main difference between gingivitis and periodontitis

A

bleeding on probing

25
Q

difference between gingivitis on intact and reduced periodontium

A

-reduced perio has probing attachment loss and possible bone loss

26
Q

stable periodontitis patient

A

perio patient who demonstrates clinical signs of perio health on periodontium who was previously diagnosed with perio but has been successfully managed in past and is currently stable (typical periodontal maintenance patient)
* probing depth less than 4 mm

27
Q

non-dental plaque-induced gingival diseases

A
  • genetic/developmental disorders
  • specific infections
  • inflammatory and immune conditions
  • reactive processes
  • neoplasms
  • endocrine, nutritional, and metabolic diseases
  • traumatic lesions
  • gingival pigmentation
28
Q

viral origins of gingival diseases

A
  • coxsackie virus
  • herpes I & II
  • varicella zoster
  • human papilloma virus
29
Q

fungal origin of gingival dieases

A
  • candidosis

- other mycoses, e.g. histoplasmosis

30
Q

inflammatory and immune conditions

A
  • hypersensitivity reactions
  • autoimmune diseases
  • granulomatous inflammatory lesions
  • hypersensitivity reactions: contact allergy, plasma cell gingivitis, erythema multiforme
31
Q

autoimmune diseases that cause gingival disease

A

pemphigus vulgaris
pemphigoid
lichen planus
lupus erythematosus

32
Q

granulomatous inflammatory lesions

A
  • crohn’s disease

- sarcoidosis

33
Q

reactive processes

A
  1. epulides
    - fibrous epulis
    - calcifying fibroblastic granuloma
    - vascular epulis (pyogenic granuloma)
    - peripheral giant cell granuloma
34
Q

neoplasms

A

I. premalignancy–leukoplakia, erythroplakia

II. malignancy–squamous cell carcinoma, leukemic cell infiltration, lymphoma (hodgkin or non-hodgkin)

35
Q

endocrine, nutritional, and metabolic diseases

A

I. vitamin deficiences – vitamin C deficiency

36
Q

traumatic lesions

A
  • physical/mechanical trauma
  • chemical (toxic) burn
  • thermal insults
37
Q

gingival pigmentation

A
  • melanoplakia
  • smoker’s melanosis
  • drug-induced pigmentation
  • amalgam tattoo
38
Q

diagnosis is not only based on site evaluation but?

A

patient evaluation

39
Q

periodontal health is defined by?

A

absence of clinically detectable inflammation

40
Q

2 broad categories of gingival disease

A
  1. non-dental plaque biofilm-induced gingival diseases

2. dental plaque-induced gingivitis

41
Q

dental plaque-induced gingivitis may arise on what or what

A

intact periodontium or on reduced periodontium

42
Q

the treated and stable periodontitis patient with current gingival health still remains at risk for what?

A

recurrent periodontitis