Headaches Flashcards

1
Q

Red flags associated with headaches

A

Fever, photophobia or neck stiffness
New neurological symptoms
Dizziness
Visual disturbance
Sudden onset occipital headache
Worse on coughing or straining (raised intracranial pressure)
Postural, worse on standing, lying or bending over
Severe enough to wake the patient from sleep
Vomiting
History of trauma

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2
Q

Important assessment in headaches

A

Fundoscopy examination to look for papilloedema is an important part of an assessment of a headache. Papilloedema indicates raised intracranial pressure, which may be due to a brain tumour, benign intracranial hypertension or an intracranial bleed.

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3
Q

Presentation of tension headaches

A

Classically they produce a mild ache across the forehead and in a band-like pattern around the head.

This may be due to muscle ache in the frontalis, temporalis and occipitalis muscles.

Tension headaches comes on and resolve gradually and don’t produce visual changes.

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4
Q

What are tension headaches associated with

A
Stress
Depression
Alcohol
Skipping meals
Dehydration
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5
Q

Treatment of tension headaches

A

Reassurance
Basic analgesia
Relaxation techniques
Hot towels to local area

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6
Q

What can secondary headaches be secondary to

A

Underlying medical conditions such as infection, obstructive sleep apnoea or pre-eclampsia
Alcohol
Head injury
Carbon monoxide poisoning

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7
Q

How does sinusitis usually present

A

Causes a headache associated with inflammation in the ethmoidal, maxillary, frontal or sphenoidal sinuses.

This usually produces facial pain behind the nose, forehead and eyes. There is often tenderness over the affected sinus, which helps to establish the diagnosis.

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8
Q

Sinusitis disease course

A

usually resolves within 2-3 weeks. Most sinusitis is viral.

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9
Q

Management of sinusitis

A

Nasal irrigation with saline can be helpful.

Prolonged symptoms can be treated with steroid nasal spray.

Antibiotics are occasionally required.

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10
Q

What is an analgesic headache

A

Caused by long term analgesia use. It gives similar non-specific features to a tension headache.

They are secondary to continuous or excessive use of analgesia.

Withdrawal of analgesia important in treating the headache, although this can be challenging in patients with long term pain and those that believe the analgesia is necessary to treat the headache.

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11
Q

What are hormonal headaches related to

A

They produce a generic, non-specific, tension-like headache.

They tend to be related to low oestrogen

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12
Q

Presentation of hormonal headache

A

Two days before and first three days of the menstrual period

Around the menopause

Pregnancy. It is worse in the first few weeks and improves in the last 6 months.

Headaches in the second half of pregnancy should prompt investigation for pre-eclampsia.

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13
Q

What can help improve hormonal headaches

A

Oral contraceptive pill

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14
Q

What is cervical spondylosis caused by

A

Degenerative changes in the cervical spine. It causes neck pain, usually made worse by movement. However, if often presents with headache.

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15
Q

Which condition is associated with trigeminal neuralgia

A

MS

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16
Q

How does trigeminal neuralgia present

A

Presents with intense facial pain that comes on spontaneously and last anywhere between a few seconds to hours. It is often described as an electricity-like shooting pain. Attacks often worsen over time.

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17
Q

Possible triggers for pain in patients with trigeminal neuralgia

A

Cold weather
Spicy food
Caffeine
Citrus fruits

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18
Q

First line treatment for trigeminal neuralgia

A

Carbamazepine

Surgery to decompress or intentionally damage the trigeminal nerve is an option

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19
Q

Typical migraine symptoms

A

Headaches last between 4 and 72 hours. Typical features are:

Moderate to severe intensity
Pounding or throbbing in nature
Usually unilateral but can be bilateral
Discomfort with lights (photophobia)
Discomfort with loud noises (phonophobia)
With or without aura
Nausea and vomiting
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20
Q

What does aura refer to

A

Aura is the term used to describe the visual changes associated with migraines. There can be multiple different types of aura:

Sparks in vision
Blurring vision
Lines across vision
Loss of different visual fields

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21
Q

What type of migraines can mimic stroke

A

Hemiplegic migraines can mimic stroke. It is essential to act fast and exclude a stroke in patients presenting with symptoms of hemiplegic migraine.

22
Q

Symptoms of hemiplegic migraine

A

Symptoms of a hemiplegic migraine can vary significantly. They can include:

Typical migraine symptoms
Sudden or gradual onset
Hemiplegia (unilateral weakness of the limbs)
Ataxia
Changes in consciousness
23
Q

Migraine triggers

A
Stress
Bright lights
Strong smells
Certain foods (e.g. chocolate, cheese and caffeine)
Dehydration
Menstruation
Abnormal sleep patterns
Trauma
24
Q

5 stages of migraine

A

Premonitory or prodromal stage (can begin 3 days before the headache)

Aura (lasting up to 60 minutes)

Headache stage (lasts 4-72 hours)

Resolution stage (the headache can fade away or be relieved completely by vomiting or sleeping)

Postdromal or recovery phase

25
Q

Acute management of migraine

A

Paracetamol
Triptans (e.g. sumatriptan 50mg as the migraine starts)
NSAIDs (e.g ibuprofen or naproxen)
Antiemetics if vomiting occurs (e.g. metoclopramide)

26
Q

Triptans in migraines

A

Triptans are used to abort migraines when they start to develop. They are 5HT receptors agonists (serotonin receptor agonists).

27
Q

How do triptans abort migraines

A

They have various mechanisms of action and it is not clear which mechanisms are responsible for their effects on migraines. They act on:

Smooth muscle in arteries to cause vasoconstriction
Peripheral pain receptors to inhibit activation of pain receptors
Reduce neuronal activity in the central nervous system

28
Q

Migraine prophylaxis

A

Keeping a headache diary can be helpful in identifying the triggers. Avoiding triggers can reduce the frequency of the migraine.

Propranolol
Topiramate (teratogenic)
Amitriptyline

Acupuncture

B2 supplements

29
Q

Most common cause of SAH

A

Head injury - traumatic SAH

30
Q

Causes of spontaneous SAH

A

Berry aneurysm
AVM
Pituitary apoplexy
Aortic dissection

31
Q

Classical features of SAH

A
Thunderclap headache 
Nausea and vomiting 
Meningism 
Coma 
Seizures 
Sudden death
32
Q

How is SAH confirmed

A

CT head

LP if CT is negative 12 hrs after onset of symptoms

33
Q

IX post confirmation of SAH

A

CT intracranial angiogram

+/- digital subtraction angiogram(catheter)

34
Q

Mx of SAH

A

Coil by IR
Craniotomy
Bed rest
Vasospasm treated with 21-day course of nimodipine

35
Q

Complications of aneurysmal SAH

A
Re-bleeding 
Vasospasm(7-14 days after onset)
Hyponatraemia due to SIADH
Seizures
Hydrocephalus
36
Q

Risk factors for SAH

A
HTN
Smoking 
Excessive alcohol consumption 
Cocaine use 
FHx
37
Q

Signs of SAH in LP

A

Red cell count will be raised. If the cell count is decreasing in number over the samples, this could be due to a traumatic lumbar puncture.
Xanthochromia (the yellow colour of CSF caused by bilirubin)

38
Q

When should you suspect bacterial aetiology for sinusitis

A

Symptoms for more than 10 days
Discoloured or purulent nasal discharge (with unilateral predominance).
Severe local pain (with unilateral predominance).
A fever greater than 38°C.
A marked deterioration after an initial milder form of the illness (so-called ‘double-sickening’).
Elevated ESR/CRP

39
Q

Examination in sinusitis

A

Inspecting and palpating the maxillofacial area to elicit swelling and tenderness.

Performing anterior rhinoscopy (using the largest speculum of an otoscope, or a head light and nasal speculum) to identify:
Signs which support a diagnosis of acute sinusitis such as nasal inflammation, mucosal oedema, and purulent nasal discharge.
Associated pathology such as nasal polyps, or anatomical abnormalities such as septal deviation.

40
Q

What should be examined in trigeminal neuralgia

A

Examine the person’s face and oral cavity (including the trigeminal nerves) to rule out dental causes of pain, and to detect any abnormalities that require referral to a neurologist

41
Q

Which conditions can lead to compression of the trigeminal nerve or cause symptoms similar to those of trigemninal neuralgia

A

Tumours, such as posterior fossa tumours, extracranial masses along the trigeminal nerve, perineural spread of existing malignancy, cavernous sinus masses

Multiple sclerosis

Epidermoid, dermoid, or arachnoid cysts

Aneurysm, or arteriovenous malformation

42
Q

Common adverse effects of carbamazepine

A

Nausea and vomiting

Sedation

Dizziness

Ataxia

Leuopenia

Switching to MR preparation of carbamazepine may help

43
Q

Symptoms of cluster headaches

A
Red, swollen and watering eye
Pupil constriction (miosis)
Eyelid drooping (ptosis)
Nasal discharge
Facial sweating
44
Q

Acute management of cluster headaches

A

Triptans (e.g. sumatriptan 6mg injected subcutaneously)

High flow 100% oxygen for 15-20 minutes (can be given at home)

45
Q

Prophylaxis for cluster headaches

A

Verapamil
Lithium
Prednisolone (a short course for 2-3 weeks to break the cycle during clusters)

46
Q

What can trigger cluster headaches

A

Alcohol
Strong smells
Exercise

47
Q

Presentation of parietal lesions

A
sensory inattention
apraxias
astereognosis (tactile agnosia)
inferior homonymous quadrantanopia
Gerstmann's syndrome
48
Q

Presentation of occipital lobe lesions

A

homonymous hemianopia (with macula sparing)
cortical blindness
visual agnosia

49
Q

Presentation of temporal lobe lesions

A

Wernicke’s aphasia
superior homonymous quadrantanopia
auditory agnosia
prosopagnosia (difficulty recognising faces)

50
Q

Presentation of frontal lobe lesions

A
Broca's aphasia 
Disinhibition 
Perseveration 
Anosmia 
Inability to generate a list
51
Q

Presentation of cerebellum lesions

A

midline lesions: gait and truncal ataxia

hemisphere lesions: intention tremor, past pointing, dysdiadokinesis, nystagmus