Head Trauma Flashcards

1
Q

An epidural hematoma is a blood accumulation between what?

A

skull and dura mater

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2
Q

What is the usual cause of an epidural?

A

trauma to the temporal bone leading to laceration of the middle mengineal artery

less frequently, laceration of the middle meningeal vein or a dural venous sinus

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3
Q

What is the classic presentation of an epidural?

A

a lucid interval immediately after the precipitating event, followed by a decline in the level of consciousness with rapid progression to coma

uncal herniation can develop as the result of hematoma expansion

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4
Q

What will be seen on head CT in an epidural?

A

a lens-shaped hyperdense lesion between the skull and the dura

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5
Q

What is the management for an epidural?

A

surgical evacuation is required

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6
Q

What is the cause of a subdural?

A

tearing of the bridging veins that connect the surface of the brain and the dural sinuses

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7
Q

Subdurals can be either acute after a trauma or chronic. Do they always need evacuation?

A

No - they may require evacuation depending on the severity of the neurological symptoms, but can often resolve on their own

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8
Q

What will a subdural look like on head CT and how does this differ from an epidural?

A

they’ll look like a crescent-shaped hyperdensity overlying the brain surface and underlying the skull. They differ from epidurals in their ability to cross suture lines

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9
Q

What are the recommendations for how long athletes should sit out after concussion?

A

No LOC and symptoms lasting less than 15 min: can return to play right away, but should sit out a week if a second such event occurs that same day

No LOC and symptoms lasting over 15 min: be evaluated frequently by a trainer and sit out for a week

LOC: be evaluated at a hospital and take 1-2 weeks off

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10
Q

What will diffuse axonal injury look like on CT?

A

multiple areas of punctate hemorrhage in the deep white matter and corpus callosum

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11
Q

25% of patients with acute severe head injury will have a posttraumatic seizure within one week. What percentage of these patients will go on to develop epilepsy?

A

25%

(note: AEDs will reduce the incidence of early seizures, but will not change the overall risk for the later development of epilepsy)

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12
Q

Central herniation occurs with downward herniation of the diencephalon through the tentorial notch. How does this present clinically?

A

decreased in the level of alertness
small, reactive pupils due to disruption of sympathetic pathways from the hypothalamus

as it proceeds, the pt may assume a decorticate posture upon stimulation

eventually fixed midposition pupils and decerebrate posturing

final stages - motionless and unresponsive to stimulation

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13
Q

Uncal herniation is most often produced by what?

A

expansion of a mass located laterally within the brain

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14
Q

What is the first deficit seen with an uncal herniation?

A

an ipsilateral IIIrd nerve palsy, bollowed by impairment of consciousness

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15
Q

Continued uncal herniation will cause compression of the contralateral cerebral peduncle against the free edge of the tentorium with a resulting hemiplegia that is ipsilateral or contralateral to the herniating uncus? What is this called?

A

ipsilateral

This is Kernohan’s notch phenomenon

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16
Q

Expansile frontal lobe masses will produce herniation of what?

A

the cingulate gyrus beneath the falx cerebri

17
Q

Often a cingulate gyrus herniation does not change the clinical picture much beyond that of the frontal mass, but what might you see if it’s severe?

A

leg weakness through compression of the ACA

18
Q

What is the normal ICP in an adult?

A

less than 15 mm Hg

19
Q

Cerebral perfusion pressure is defined as what? What is the goal?

A

the difference between the mean arterial pressure and the ICP
goal is a CPP between 60 and 75 mmHg

20
Q

When should you consider monitoring the ICP with an intraventricular pressure monitor?

A

in any head injury patient with a GCS less than 9 and abnormalities on the head CT

21
Q

What are some management options for elevated ICP?

A

elevate the head of the bed to 30 degrees
hyperventilate to a PCO2 between 25 and 30 mmHg
Mannitol or hypertonic saline
IV barbiturates to reduce cerebral metabolism
CSF drainage with a ventricular drain
hemicraniectomy if severe

22
Q

Why does hyperventilation work to reduce ICP?

A

the reduction in PCO2 causes vasoconstriction, reducing cerebral blood volume and thus ICP