Head trauma Flashcards
TBI primary
immediate structural damage
- lacerations
- hematomas
- contusions
- tissue avulsions
- coup/contracoup injury (injury on another area of brain due to initial impact)
- shear diffuse- axonal injury
TBI secondary
neurotoxic cascade (not same as secondary insult) - intracellular and extracellular derangement
Cerebral concussion definition
does NOT require a LOC
associated with transient neurological changes such as:
transient amnesia
confusion
disorientation
visual changes without any gross cerebellar abnormalities or deficits on exam and a trauma
Cerebral blood flow
head trauma can alter CBF by creating an expanding mass or cellular death
need minimum blood flow to avoid worsening injury and provide nutrients
CBF amount
750 cc/min
MAP means
CPP means
mean arterial pressure
cerebral perfusion pressure
TBI classification- GCS scale
CNS injury is the number one cause of traumatic death
Mild- 13-15, brief LOC
Mod- 9-12, mild confusion or focal deficit
Severe- <9, survivors have life long disability
Eye 1- no eye 2- eye opening to pain 3- eye opening to speech 4- eye opening spontaneously
Verbal 1- non 2- incomprehensive sounds 3- inappropriate words 4- confused 5- oriented
Motor 1- none 2- extension to pain 3- flexion to pain 4- withdraws from pain 5- localizes to pain 6- obeys commands
less than 8= intubate
Cushing’s reflex
hypertension (increased systolic BP)
bradycardia (decreased pulse)
decreased RR
systemic response to increased ICP
Herniations
Medial- temporal lobe (Uncal)= most common
Central
Cingulate
Posterior fossa
Canadian rule
tries to find those who need a CT, doesn’t say you need to get a CT, it says you do not need a CT
Inclusion criteria: GCS- 13-15 Age >16yrs No coagulopathy or on anti-coagulation No obvious open skull fracture
Exclusion- Head CT not required if ALL are absent
(long list)
Likelihood of missing an injury is small
Tries to find those on blood thinners (anti-coagulants), over 60, open fractures
Epidural bleed- epidural hematoma
lenticular- acute collection of blood above the dura (do not extend beyond sutures), usually from middle meningeal artery
LOC–> Lucidity–> rapid decompensation with signs of increased ICP with pupillary dilation and unconsciousness
Subdural bleed
crescent shaped- collection of venous blood between the dura and arachnoid- in subdural space
a result of venous bleed (bridging veins)
Slower onset than epidural but higher incidence of underlying brain injury
Acute= <14 d old, >14= chronic
Diffuse axonal sheer injury
result of abrupt deceleration forces
axons tear along the white/grey matter interface
CT can appear normal or small petechial hemorrhages
MRI- best assessment
Basilar skull fracture
base of skull typically petros portion of temporal bone
associated with CN 7,8 and cerebral vascular problems
raccoon eyes
hemotympanum (blood behind TM)
battle sign bruising behind ears and/or
drainage from nose or ear canal
need head and neck angio + CT
Tx for uncal, central, cingulate, and posterior fossa herniation?
single dose of mannitol over 15 min (acts as a diuretic)
to decrease volume and ICP
Pediatric head injury- PECARN >2
exclusion criteria
AMS severe mechanism clinical signs of baser skull fracture any loc hx of vomiting hx of severe headache
if none of these then no CT indicated
Who is at higher risk for intracranial injury after trauma? Why?
less than 2yo- large heads, weak torso and compressibility of skull (also for non-accidental trauma (NAT)
elderly particularly SDH- cerebral atrophy resulting in bridging vein tears with deceleration forces
alcoholics- greater incidence of trauma and cerebral atrophy and coagulation disorders (vit k dependent)- thiamine deficiency
pts on anticoagulants (or intrinsic bleeding disorders)
Uncal herniation
medial portion of temporal hemispheres shifts below tentorium
ipsilateral 3rd nerve palsy
ptosis
Babinski reflex
eventually cerebral peduncle gets compressed and motor hemiplegia occurs
Central herniation
both hemispheres through tentorium
pinpoint pupils
hemiparesis
Cingulate herniation
cingulate gyrus is forced underneath the falx compression of ventricles and impairing cerebral blood flow
Posterior fossa herniation
bleeding pushes cerebellar tonsils through foramen magnum of upward through tentorium
coma &
brain stem dysfunction occur rapidly causing death
pinpoint pupils that are fixed
What is the goal of TBI tx
maintain cerebral perfusion pressure
avoid the 5 Hs
- hypotension
- hypoxia
- hypoglycemia
- hypercarbia
- hyperthermia
What is important for conditions of increased ICP?
seizure prophylaxis
What happens in MAP less than 50mmHg and more than 110mmHg?
lose the ability to auto regulate
cerebral perfusion pressure will drop
worsening injury occurs