Head injuries Flashcards

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1
Q

why aren’t more adults susceptible to damage similar to shaken baby syndrome?

A

babies have a large head relative to their body, weak cervical musculature, poor control of their head/neck, the cervical cord is different, babies have a higher brain water content, and babies only have a minor degree of myelination

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2
Q

what characterizes a subgaleal hematoma?

A

this hemorrhage is found beneath the galea aponeurotica (directly below the scalp) and is not obvious externally

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3
Q

what characterizes terminal fall injuries?

A

these are often due to loss of consciousness and consist of injuries to the bony prominences of the face - often in *linear distribution. terminal fall injuries may include abrasions, contusions, and lacerations

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4
Q

what characterizes cerebral contusions

A

these appear as a linear streak-like hemorrhage of the cortex and may be due to: coup (stationary head, moving object = wound at impact), contra coup (moving head, stationary object = wound opposite impact), intermediary coup (rotational movement due to battering of head = brain damage due to its own uneven movement), fracture or herniation (w/brain swelling around a falx)

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5
Q

what does a series of oval to round contusions on a pt raise suspicion of?

A

punch

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6
Q

what characterizes transtentorial/uncal herniation?

A

the medial aspect of the temporal lobe becomes compressed against the tentorium, compressing the 3rd nerve = pupillary dilation on the ipsilateral side of the lesion.

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7
Q

what is a bite mark to the tongue indicative (but not diagnostic) of?

A

seizure

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8
Q

what characterizes subarachnoid hemorrhage (SAH)?

A

this hemorrhage occurs between the arachnoid layer and the cerebral cortex. this may occur due to trauma (vertebral artery trauma = basal SAH) or naturally (berry aneurysm, AVM, cocaine, meth). they are not easily wiped off the cortical surface

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9
Q

what characterizes diffuse axonal injury?

A

this due to rapid brain acceleration/deceleration is seen in white matter tracts (corpus callosum, internal capsule, cerebral peduncles) which have a high density of axons. this is most severe in midline and paramidline structures (prone to shearing force). microscopically DAI consists of axonal swelling, disruption of cytoarchitecture/neurofilaments (axonal bulbs), and can be stained for w/beta amyloid precursor protein in as little as 2-3 hours.

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10
Q

what can cause periorbital contusions (racoon’s eyes/spectacle hematomas)?

A

gunshot wound to the head (may generate enough force to fracture the orbital plates)

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11
Q

what are the 2 kinds of cerebral edema? what morphology is associated w/it?

A

1) vasogenic: integrity of the BBB is disrupted, causing *interstitial edema.
2) cytotoxic edema: increase in *intracellular fluid from hypoxic-ischemic insult. morphology: gyri flattened, sulci narrowed

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12
Q

what are the categories of soft tissue injury to the head?

A

subgaleal hematoma
epidural
subdural
subarachnoid hemorrhage

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13
Q

What is epidural hematoma?

A

above dura - typically associated w/a fracture of the temporal bone = middle meningeal artery damage

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14
Q

What is subdural hematoma?

A

below dura - not usually associated w/a fracture, due to tearing of bridging veins - may not require tx

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15
Q

Causes of subarachnoid hemorrhage?

A
ruptured berry aneurysm
cerebral contusion (coup and countercoup injuries)
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16
Q

what is a lefort I fracture?

A

a transverse fracture of the maxilla, above the apices of the teeth and through the nasal septum, maxillary sinuses, and palantine bone of the sphenoid bone

17
Q

what characterizes subdural hematoma?

A

this is due to tears of bridging veins which link superficial cortical veins to the superior sagittal sinus (though occasionally arteries are involved) and result in compression of gyri and sulci = undulating surface of the cortex (unlike the flattened surface seen in EDH). skull fractures may or may not be present (damage is simply due to movement of the brain inside the skull). SDH’s are good tox specimens. over time chronic SDH may produce a flattened cortex similar to EDH, initially w/an inner arachnoid side membrane + outer dural side membrane - eventually becoming a single fibrous membrane.

18
Q

what is the worst result of cerebral edema?

A

herniation, which is due to increased ICP

19
Q

are skull fractures common in children?

A

no - their skulls tend to be more pliable

20
Q

what is the spectrum of diffuse traumatic brain injury?

A

concussion (functional damage w/o structural damage) and diffuse axonal injury (aka DAI = structural damage)

21
Q

what is shaken baby syndrome?

A

this is due to rapid acceleration/deceleration of the brain and may not involve distinct impact sites. sub dural hematoma (torn veins), brain swelling, cervical cord injury (RAS etc), retinal/optical nerve injury (retinal hemorrhages are red flags for abuse), possible gripping injury are all associated w/shaken baby syndrome. “respirator brain” is commonly seen in shaken baby syndrome as the brain swelling causes diffuse ischemia, which leads to further swelling, etc = brain starts to fall apart

22
Q

what characterizes tonsillar herniation?

A

the posterior medial portion of the cerebellum is forced through the foramen magnum

23
Q

what is a gliding contusion?

A

this is when gliding of the brain under the dura leads to diffuse axonal injury (DAI)

24
Q

what are the different types of skull fracture?

A

linear (usually due to a simple fall), depressed (fragments into brain), diastatic (sawtooth appearance, younger person - brain swells and pushes out sutures), basilar (battle sign - contusion behind the ear), orbital roof (periorbital ecchymosis - either due to direct or penetrating injury), hinge (usually fatal - fracture going through one side of the skull to the other), and ring (around foramen magnum - spine is pushed up through by upward force from below)

25
Q

what characterizes the morphology of hypoxia/ischemia in the brain?

A

immediately after insult, the brain may appear normal - but w/in 24-48 hrs, the brain becomes swollen and necrosis may develop = respirator brain?

26
Q

what is a lefort II fracture?

A

a *pyramidal fracture w/a similar posterior path as a lefort I - however, lefort II fractures also curve anteriorly near the zygomatic-maxillary structure and pass through the inferior orbital rim into the orbital floor and through the medial orbital wall and across the nasal bones and septum.

27
Q

what is a laceration?

A

a wound due to a blunt object tearing soft tissue (not a cut wound)

28
Q

what characterizes an epidural hematoma (EDH)?

A

these often involve the lateral temporal and sphenoid bones which the *meningeal arteries are near and present on the surface of the dura. epidural hematomas cause a *linear flattening of the cortex (shelf-like). artifactual EDH may occur in cases like a fire where increased vapor pressure fractures the skull and heated bone marrow moves to the superior surface of the brain. posterior fossa EDH is rare. EDH can be an important tox specimen (b/c it has been sequestered)

29
Q

what is a lefort III fracture?

A

this is a high transverse fracture involving the maxilla. lefort III fractures pass through the nasofrontal suture, through the medial orbital wall and frontozygomatic suture before passing through the sphenoid bone.

30
Q

what characterizes cingulate gyrus herniation?

A

unilateral asymmetric expansion of the cerebral hemisphere displaces the cingulate gyrus under the falx cerebri

31
Q

what is a dentoalveolar fracture?

A

separation of a fragment of the mandible due to anterior or lateral direct force. the fragment often contains a number of fractures

32
Q

what are the categories of facial fracture?

A

dentoalveolar, lefort I-III and sagittal - all of which require a lot of force (not generally produced in a simple fall).

33
Q

what is a sagittal fracture?

A

a fracture running in the sagittal plane which passes through the maxilla