Head, Eyes, Ears, Nose, Mouth & Neck Flashcards

1
Q

What is required for a complete exam of the head and neck?

A

visual inspection, obtaining measurements, palpation, ophthalmoscope, auscultation and transillumination

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2
Q

What state is optimal for assessment?

A

to facilitate an optimal assessment with minimal discomfort, a quiet alert state allows for examination of the eyes and a crying state allows for examination of the oropharynx

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3
Q

What is included in general initial observation?

A

infant’s state; race-specific variations; color of the skin and mucous membranes; size and symmetry of the head and face; obvious deformations, malformations or evidence of birth trauma

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4
Q

What percentage of all anomalies seen at birth occur in the region of the head and neck?

A

90%; minor anomalies of the head and neck are common

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5
Q

What is the first step in the assessment of head size?

A

measurement of the occipital-frontal circumference (OFC)

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6
Q

How is an OFC measurement properly obtained?

A

by measuring 3 times and recording the largest value obtained; encircling the head at the widest occiput prominence and anteriorly 1-2 cm above the glabellar space at the largest frontal prominence

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7
Q

What is the average OFC measurement of a 40 week term infant?

A

35 cm, ranging from 33-37cm between the 10th and 90th percentile

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8
Q

Prior to the closure of the fontanels, the OFC is an indirect measurement of what?

A

intracranial contents including the brain, cerebrospinal fluid, cerebral blood volume and bone

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9
Q

How can an OFC measurement after birth be possibly misleading?

A

r/t cranial molding, scalp edema or hemorrhage under the periosteum; subsequent measurements should be taken for several days after birth

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10
Q

What is required to diagnose symmetric v asymmetric growth restriction and micro- v macrocephaly?

A

the percentile of OFC, weight and length respective to gestational age

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11
Q

What qualifies as microcephaly?

A

OFC <10% for GA

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12
Q

What causes microcephaly?

A

poor brain growth

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13
Q

How are cranial sutures affected by microcephaly?

A

cranial sutures become prematurely fused because the expansive force of brain growth that enlarges the cranial vault is lacking

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14
Q

Is microcephaly an isolated finding?

A

Sometimes, or it may be associated with a genetic syndrome or congenital infx

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15
Q

Under what circumstances might an OFC measurement be less than expected?

A

prematurity; discrepancies may be d/t inaccurate dating and/or pathologic restriction of growth; non-caucasian infants have a smaller OFC as a normal race variation

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16
Q

Which occurs more frequently: growth restriction or growth acceleration?

A

growth restriction

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17
Q

What qualifies as macrocephaly?

A

OFC >90% despite appropriate weight and length for GA

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18
Q

Why might macrocephaly present?

A

may be a familial characteristic or caused by hydrocephalus or a/w dwarfism or OI

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19
Q

What is the typical presentation of familial macrocephaly?

A

more often presents as macrencephaly (large brain volume) without hydrocephalic features

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20
Q

How is familial macrocephaly confirmed?

A

confirmation requires obtaining measurements of the parents’ heads and plotting them on a Weaver curve

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21
Q

When is cranial transillumination indicated?

A

When the infant’s head has an unusual shape or size or the neurological exam is abnormal

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22
Q

What is implied by a ring of light >2cm larger than the light source when performing a cranial transillumination?

A

increased fluid volume or decreased brain tissue in the cranium

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23
Q

Under what circumstances might an examiner obtain a “false positive” when performing a cranial transillumination?

A

when the infant has a large caput because the scalp edema will transmit a halo of light

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24
Q

In the infant presenting with a high level of suspicion for hydrocephalus, transillumination is often replaced by what other more definitive studies?

A

cranial ultrasound, CT scan or MRI

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25
The relative shape of an infant's head typically relates to what?
molding of the skull during delivery
26
What is the typical shape of an infant's head delivered via CSX?
well rounded
27
What is the typical shape of an infant's head delivered in breech position?
molded posteriorly into an egg shape with a prominent occiput
28
What is the typical shape of an infant's head delivered following prolonged diagonal pressure?
"out of round" or asynclitic when viewed from above
29
What can be expected of distorted skull shape due to positional, external pressures in utero or during labor?
to spontaneous resolve; molding usually resolves within a few weeks following birth
30
What is a suture?
fibrous bands of tissue that separate bones
31
What is a fontanel?
occur at the intersection of sutures
32
Where is the metopic suture located?
the metopic suture extends midline down the forehead between the two frontal bones and intersects with the coronal suture
33
Where is the coronal suture located?
the coronal suture is situated anterolaterally across the cranium separating the frontal and parietal bones
34
What is formed at the intersection of the metopic, coronal and sagittal sutures?
the anterior fontanel (AF)
35
What is the typical size of the AF?
the size of the AF varies from 0.6-3.6cm across; in African American infants, it is commonly larger (1.4-4.7cm)
36
How are fontanels measured?
diagonally from bone to bone rather than from suture to suture
37
What can fontanel measurement provide?
because there is a wide variation of fontanel size at birth, the measurement may serve only as a baseline for serial comparison. Individual measurements have no clinical significance and limited reproducibility
38
How is the AF typically described?
flat and soft is assessed with the infant in a quiet state with the infant held in the sitting position
39
What might a tense or bulging fontanel indicate?
increased ICP or may occur with a crying infant
40
What might a sunken fontanel indicate?
severe dehydration and is rarely seen in the newborn nursery; may indicate excessive or acute decompression by an EVD
41
What might a very large AF indicate?
congenital hypothyroidism
42
When is the AF expected to close?
between 6-24mo of age
43
Where is auscultation performed?
over the fontanels and lateral skull bones
44
When is auscultation of the fontanels specifically indicated?
infants with multiple hemangiomas or heart failure
45
What does the appreciation of a bruit over the fontanels or lateral skull indicate?
may be a normal finding; a bruit in infant with suspected cardiac failure is a/w an intracranial arteriovenous malformation- which may be the cause of the CHF
46
Where is the sagittal suture located?
the sagittal suture extends midline between the two parietal bones to the PF
47
Where is the lambdoidal suture located?
the lambdoidal suture extends posterolaterally across the cranium to separate the occipital and parietal bones
48
What is formed at the intersection of the lambdoidal and sagittal sutures?
the PF
49
What is the approximate size of the PF?
0.5cm in Caucasian infants; 0.7cm in AA
50
When does the PF close?
approximately by 2-3mo of age
51
How does the PF typically present?
soft and flat on palpation
52
If present, where can a third fontanel be found?
may occur along the sagittal suture between the AF and PF
53
Is the third fontanel a true fontanel?
No, it is a defect of the parietal bone
54
What might the presence of a third fontanel indicate?
may be a normal variation or may be a/w Down syndrome or congenital hypothyroidism
55
Where is the squamosal suture located?
extends above the ear to separate the temporal bone from the parietal bone
56
Where is the sphenoid fontanel located?
at the intersection of the coronal and squamosal sutures and posterior to the frontal bone, distal to the parietal bone and anterior to the temporal bone
57
Where is the mastoid fontanel located?
at the intersection the squamosal and lambdoidal sutures and posterior to the temporal bone, distal to the parietal bone and anterior to the occipital bone
58
Under what conditions can the mastoid fontanel, sphenoid fontanel and squamosal suture be palpated?
only with increased ICP as with severe hydrocephalus, but may be palpable in preterm infant r/t rapid brain growth
59
How is mobility of the sutures assessed?
placing the thumbs on the bones on either side of the suture and gently pressing down alternately with one thumb then the other
60
How are normal sutures described?
approximated and mobile, may be split up to 1 cm
61
What are overriding sutures?
with molding, the edge of the bone on one side of the suture will feel as if is it on top of the edge of the opposing bone
62
What suture is commonly overriding?
lambdoidal sutures- the parietal bone on top of the occipital bone due to molding after birth or with minor decrease in hydration
63
What is craniosynostosis?
premature fusion of the suture
64
How is craniosynostosis differentiated from an overriding suture?
presents as a peaking of the approximated bones and is immobile
65
How is head shape affected by craniosynostosis?
premature closure stops perpendicular growth to the suture but allows parallel bone growth and compensatory expansion at the functional sutures leading to abnormal head shape
66
When can craniosynostosis present?
may be present at birth, or later in infancy
67
How does brachycephaly occur?
fused coronal sutures limit forward growth of the skull and lead to a broad shaped skull
68
How does scaphocephaly occur?
premature closure of the sagittal suture limits lateral growth and results in a long, narrow head
69
How does plagiocephaly occur?
closure of a laterally positioned suture results in asymmetric skull shape
70
What is the term for an abnormally shaped preterm infant's head relating to postnatal positioning and without craniosynostosis?
dolichocephaly
71
How can craniosynostosis present?
can be a primary finding or may be a/w a genetic syndrome (Apert or Crouzon) or the result of a metabolic disorder (hypothyroidism)
72
what is the incidence of isolated craniosynostosis?
0.4-1 per 1,000 live births
73
What are craniotabes?
palpation of the skull may reveal areas of soft or thinning bone
74
What is the Macewen sign?
palpating the skull in an area of thinning bone that elicits collapse with recoil of the underlying bone and a snapping sensation
75
Why are craniotabes sometimes present?
an incidental finding on the parietal bones near the sagittal suture; d/t external pressure from prolonged vertex engagement or pressure of the fetal head on the uterine fundus with breech position; or r/t internal pressure and hydrocephalus
76
If craniotabes are present due to external pressures, when can they be expected to resolve?
a few weeks
77
If craniotabes are present due to internal pressures, when can they be expected to resolve?
resolution depends on the degree and persistence of internal pressure causing bone thinning
78
What is the most common form of trauma to the head during birth?
caput succedaneum
79
What is a caput?
edema of the presenting part of the scalp caused by pressure that restricts the return of venous and lymph flow during labor and delivery; may be accentuated by vacuum assist
80
What are the defining characteristics of a caput?
edema pits with pressure, edges are poorly defined and crosses suture lines
81
When can a caput be expected to resolve?
caput is noted immediately after birth and resolves within a few days (unlike a cephalhematoma)
82
How is a cephalhematoma formed?
the result from the collection of blood between the periosteum and the skull
83
Why might a cephalhematoma not be evident at birth?
may be obscured by a caput
84
What are the defining characteristics of a cephalhematoma?
clearly demarcated edges confined by suture lines
85
How long does it take for a cephalhematoma to resolve?
with time it may liquefy and become fluctunant on palpation; takes weeks to months to resolve completely
86
What are the most common locations for a cephalhematoma to present?
the parietal and occipital bones
87
How frequently are cephalhematomas associated with depressed skull fractures?
rarely
88
What is the most potentially serious lesion presenting from birth trauma?
a subgaleal hemorrhage
89
What actions in a delivery increase the risk of a resultant subgaleal hemorrhage?
most common with instrumental vaginal delivery, especially vacuum; sometimes occurs with CSX or any maneuver during delivery that produces a shearing forces to the scalp resulting in tearing of the large emissary veins
90
What is a subgaleal hemorrhage?
bleeding into the galen aponeurotica or subaponeurotica space, which extends from the orbital ridges to the nape of the neck and laterally to the ears
91
Why is a subgaleal hemorrhage potentially life threatening?
this potential space produces a large compartment capable of containing the total blood volume of an infant (5-22% mortality rate is blood loss is extensive and not diagnosed early)
92
How does a subgaleal hemorrhage typically present?
generalized scalp edema, usually with ecchymosis, BL or unilateral periorbital and periauricular edema
93
How does a subgaleal hemorrhage present on palpation?
the ballotable fluid mass crosses the sutures and can be manually repositioned from the eyebrows to the nape of the neck, differentiating it from a caput.
94
If permitted to progress, a subgaleal hemorrhage will result in, what?
severe anemia, hypotension and death
95
How is a subgaleal hemorrhage managed?
volume resuscitation, blood replacement and treatment of presenting clot abnormalities
96
What additional scalp traumas may occur resulting from delivery?
puncture from scalp electrodes, lacerations from fetal blood sampling or uterine incision, bruises, abrasions or subQ fat necrosis from an instrument delivery
97
How should trauma from a delivery be described?
by appearance, size, location near sutures or fontanel or underlying bones
98
When are open scalp defects (aplasia cutis congenita) observed?
may be a normal variant but are sometimes a/w trisomy 13
99
What skull malformations are the result of an incomplete neural tube closure?
encephalocele
100
Where do encephaloceles most commonly present?
central nervous tissue can protrude from a defect anywhere on the skull but most commonly midline and in the occipital area
101
A through examination of the scalp should include what elements?
assessment of the hair for quantity, texture, distribution and hair whorls
102
What attributes of the hair may be a/w congenital anomalies?
a low hair line, an increased quantity of hair and brittleness
103
What accounts for the presence of hair whorls?
the slope of each hair follicle is a/w the stretch of skin during brain growth
104
Why are hair whorls typically present in the posterior parietal region?
the most rapid brain growth occurs at 16 to 19 weeks GA and results in a hair whorl in it's corresponding region of concentrated growth
105
How many hair whorls is considered normal?
1-2
106
What scalp features may indicate mental retardation or aberrant brain growth?
an abnormally placed whorl, absence of a hair whorl or unusual hair growth
107
What feature of the scalp correlates with central nervous system development?
scalp hair in the newborn has variable coarseness and the thickness of scalp hair growth patterns correlates with CNS development
108
What is alopecia?
an abnormal deficiency of hair that may be diffuse or focal
109
What is typical of diffuse alopecia?
commonly due to a genetic anomaly in the hair follicles or is syndromic
110
What is typical of focal alopecia?
often traumatic or a/w underlying scalp lesions
111
What is hirsutism?
excessive hair growth
112
What can cause hirsutism?
genetic, syndromic, metabolic, drug-induced or an isolated finding
113
What is aplasia cutis congenita and where is it commonly found?
an uncommon scalp lesion found primarily at the vertex just in front of the lambda
114
How does aplasia cutis congenita typically present?
a hairless, circumscribed area of 1cm or more, surface is often shiny, cicatrical and flat/keloid; occasional blistering/ulceration w/serosanguinous exudate or fresh granulation
115
What is the risk of aplasia cutis congenita?
potential underlying defect and other assoc major defects; usually is an isolated finding
116
What are the long term implications for an aplasia cutis congenita?
resolves with a residual scar formation and absence of hair growth
117
How should examination of the face begin?
observation of the relationships b/w eyes, ears, nose & mouth
118
What is the relative size of the newborn forehead and why?
takes up the upper half of the face and reflects the large cranial volume needed for rapid brain growth
119
What may indicate face or brow presentation at delivery or nuchal cord?
facial bruising, petechiae and progressive edema
120
What might occur as the result of prolonged intrauterine compression from oligohydramnios?
unusual flattening of facial features
121
What can occur from a forceps assisted delivery?
bruises, abrasions or subQ fat necrosis
122
How are facial movements assessed for symmetry?
during crying
123
How will an infant present if there has been damage to CN7 prior to or during delivery?
paralysis of the affected side of the face; extensive damage can involve the entire side of the face and cause drooping of the muscles on the affected side of the face
124
What portion of CN7 is most commonly damaged?
the mandibular branch which controls the muscles around the lips
125
How do infants present if the mandibular branch of CN7 has been damaged?
decreased mv'mt on the affected side bc weakened facial muscles allow the mouth to be pulled to the unaffected side ("drooping mouth")
126
How can CN7 damage be differentiated from asymmetric crying facies?
loss of forehead wrinkling and nasolabial fold and partial closing of the eye on the affected side
127
if nerve damage is caused by pressure, when are facial effects expected to resolve?
hours to weeks after birth
128
When does congenital facial palsy occur?
often spontaneous but is also a/w difficult or instrumented delivery requiring forceps
129
What might a persistent palsy indicate?
an underlying, more central abnormality
130
What is asymmetric crying facies?
a congenital absence or hypopasia of the depressor anguli oris muscle (DAOM) on one side
131
What does the depressor anguli oris muscle (DAOM) control?
frowning; with resulting asymmetry of the face with crying
132
Abnormal formation or placement of the ears may be associated with what?
chromosomal anomalies and syndromes, however a wide variety of minor structural variations fall within the normal range
133
In a term infant, how should the pinna appear?
should be well formed with cartilage that recoils easily after folding
134
Why might there by temporary asymmetry of the ears?
r/t unequal intrauterine pressure on the sides of the head (common)
135
Especially hairy ears involving both the pinna and the lobes are present in what groups of infants?
may be familial, syndromic or infants of mothers with poorly controlled DM
136
Pits and tags are remnants of what embryologic structures and where are they located?
these minor malformations are located anterior to the tragus and are thought to be remnants of the first brachial cleft or arch
137
How can a preauricular sinus present?
may be blind or it may communicate with the internal ear or brain
138
In the context of chronic infx, what treatment may be necessary for a preauricular sinus?
surgical removal of the entire tract
139
What is a Darwinian tubercle and what does it indicate?
small nodule appearing on the upper helix; normal variant
140
What does a poorly formed external ear possibly indicate?
a possible chromosomal anomaly or syndrome
141
How can ear position be assessed?
by extending a line from the inner to the outer canthus of the eye toward the ear; if insertion of the ear falls below this line, it is considered low set
142
Describe the appearance of an infant's external auditory canal.
it is short and may contain vernix, interpartum blood or mec debris (making otoscopic examination difficult)
143
What should the external ear be examined for?
inspected visually to assess presence and patency of the auditory canal
144
What may increase an infant's risk for hearing loss?
a small or abnormally developed ear
145
What trauma to the eyes can occur with a NSVD?
bruises, edema of the eyelids and sclera hemorrhage
146
What causes a conjunctival or subconjunctical hemorrhage?
results from the rupture of a capillary of the mucous membrane that lines the eyelids and is reflected onto the eyeball (conjunctiva)
147
How does a conjunctival or subconjunctical hemorrhage present?
it is seen as a bright red area on the sclera near the iris
148
When does a conjunctival or subconjunctical hemorrhage resolve?
spotaneously resolves within a week to ten days without residual effect
149
What can cause inflammation of the conjunctiva?
prophylaxis silver nitrate gtt (less often with erythromycin ointment)
150
What is a coloboma?
an absence or defect of some ocular tissue, including the eyelid
151
What is ptosis?
a paralytic drooping of an eyelid when the lids are fully open
152
How does nevus simplex present on the eyelids and glabella?
as a common vascular birthmark
153
When does tear formation begin?
not usually until 2-3mo of age
154
When does the nasolacrimal duct become fully patent?
not until 5-7 mo
155
Until the nasolacrimal duct is patent, what is a common finding?
purulent of mucoid eye drainage
156
When is nasolacrimal duct drainage concerning?
When in the context of conjunctival inflammation with redness or swelling
157
When infection is not suspected, how can nasolacrimal duct drainage be treated?
lacrimal massage and gentle cleansing with water and a cotton ball
158
What is the relative size of symmetric eyes?
the distance bw the outer canthi can be divided into equal thirds, with one normal palpebral fissure length fitting into the inner canthal distance
159
What is hypertelorism?
if the eyes are more widely spaced
160
What is hypotelorism?
if the eyes are closely spaced
161
What is an eye characteristic found in Asian population and 20% of non-Asian population?
the epicanthal fold is a vertical fold of skin on either side of the nose that covers the lacrimal caruncle
162
Why does the epicanthal fold that covers the lacrimal duct disappear in non Asian populations?
By 10 y; the growth of the nasal bridge catches up to that of the medial canthal skin
163
What might epicanthal folds with upslanting palpebral fissures and hypertelorism?
Down syndrome
164
How are eyebrows normally positioned?
eyebrows normally extend above the eye in a curve approximately the length of the palpebral fissure
165
What do eyebrows that meet at the glabella and abnormally long or tangled eyelashes suggest?
Cornelia de Lange syndrome
166
What facilitates the eye exam?
spontaneous opening by the infant
167
What can encourage spontaneous opening of the eye?
auditory stimulus, changing the infant's position, gentle swinging or dimming the lights
168
What happens if the infant's eyes are forcefully opened?
the eyelids will evert, making visualization impossible
169
How do you conduct an opthalmoscopic exam?
first adjust for focus and then supply a small round white beam of light; direct the light into the pupil from 6" to assess constriction, PERL
170
What is a red light reflex?
when a bright light is directed at the newborn's lens, a clear red color is reflected from the retina back to the examiner
171
What will interrupt a red light reflex?
an opacity of the lens or cornea
172
What is implied by a lack of a red light reflex?
congenital cataracts, glaucoma or a retinoblastoma
173
How might the red light reflex appear in dark-skinned infants?
pales from orange toward grey in the most pigmented infants
174
What is a keyhole shaped iris?
a coloboma of the iris
175
Describe the iris of a newborn infant.
generally dark grey, blue or brown at birth
176
When do infant's acquire final iris pigmentation?
about 6mo
177
What are brushfield spots?
white specks scattered linearly around the entire circumference of the iris
178
With what syndrome do brushfield spots typically present?
Down syndrome, although may be a normal variant
179
What might a blue sclera indicate?
OI
180
What might a yellow sclera indicate?
hyperbilirubinemia
181
In a normal gaze, what part of the sclera should never be visible?
above the iris
182
What is the sunset sign and what is it associated with?
lid retraction and a downward gaze; seen in infants with hydrocephalus
183
What is nystagmus?
rapid, searching movements of the eyeballs
184
How might limited horizontal nystagmus be elicited and when does it disappear?
with rotational eye movement; 3-4 mo of age
185
What presentation of nystagmus is always aberrant and likely a/w visual and/or neurologic abnormalities?
spontaneous horizontal, vertical or torsional nystagmus and persistent nystagmus
186
From what does strabismus arise?
from muscular incoordination and gives the appearance of crossed eyes
187
When does pseudostrabismus from a flat nasal bridge or epicanthal fold usual resolve?
by 1yr
188
How do pseudostrabismus and strabismus differ?
pseudostrabismus has the presence of a symmetrical corneal light reflex
189
Proptosis or enlargement of the eyeball is associated with what congenital condition?
hyperthyroidism
190
What damage might congenital glaucoma cause to the neonatal eye?
damage to the eye with hemorrhage into its orbit
191
What is the typical presentation of an infant's nose?
generally smaller and flatter than an adult's; nose should be symmetric and placed vertically in the midline
192
Describe the nose of an infant with Down snydrome.
a very low nasal bridge with a broad base
193
What might cause lateral deviation of the nasal septum?
position in utero or by a dislocated septum
194
How can you determine if the nasal septum is deviated?
if the septum will not easily straighten and the nares remain asymmetric when the tip of the nose is pushed to midline- will require tx
195
Elevating the tip of the nose slightly will allow the examiner to assess what features?
the nasal septum, the floor of the nose and the turbinates
196
How can nasal patency be assessed?
by watching the infant breathe in a quiet state
197
How will bilateral choanal atresia present in the neonate?
cyanosis at rest and pink when crying (because of breathing through the mouth)
198
Nasal occlusion can also be acquired from accumulation of secretions and result in what signs and symptoms?
noisy respirations, feeding difficulties, respiratory distress or apnea
199
What might excessive bulb suctioning cause?
aggravate the occlusive tendency of the nares
200
How should nasal occlusion secondary to edema be treated?
intermittent installation of NS gtt
201
What may result from the use of CPAP therapy?
erosive trauma to the nasal labia, nasi columella and nares
202
How can the trauma r/t CPAP therapy be minimized?
preventative and protective skin coverings, monitoring the area for ulceration will minimize long-term disfiguring and scarring
203
What anomalies are usually syndromic or a/w major CNS anomalies?
major nasal abnormalities (including clefts, single nares, masses and partial or complete hypoplasia)
204
How should the lips and mucous membranes of a healthy term infant present?
pink; mild circumoral cyanosis is normal during transition and with crying during the first few days
205
What is microstomia
small oral opening
206
When is microstomia typically present?
storage diseases such as the mucopolysaccharidoses
207
What "facies" are typically present with fetal ETOH syndrome?
a thin upper lip with a smooth philtrum and short palpebral fissures
208
What elements of an infant's cry should be assessed?
quality, strength, pitch and hoarseness or stridor
209
Use of a tongue depressor for oral examination may result in what?
a strong protrusion reflex, making assessment difficult
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How might an infant's mouth be opened safely?
gently pressing down on the chin
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How should a typically presenting tongue appear?
the tongue should fit well into the floor of the mouth and appear symmetric
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What is the danger with macroglossia?
impedes closure of the mouth, may obscure airway
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What conditions present with macroglossia?
Bechwith-Wiedemann, hypothyroidism and mucopolysaccharidosis
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Is there macroglossia associated with Down syndrome or Robin sequence?
No, the tongue may appear large with micrognathia
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What is the frenulum?
attaches the underside of the tongue to the floor of the mouth, usually midway between the tongue's ventral surface and tip
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When does a tongue tie require treatment?
a very thick and prominent frenulum is rare; if limiting movement or pulls the tongue to a V at the tip- it is abnormal and requires tx
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What might white patches on the tongue and mucous membranes be?
residual milk or leukoplakia (normal in darkly pigmented infants)
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How do lesions of a Candida (oral thrush) present?
if the white coating cannot be easily removed with a tongue blade or cotton swab
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What is a mucocele or ranula?
a translucent of bluish swelling under the tongue, mucous or salivary gland retention cysts that usually spontaneous resolve
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What should be palpated within the oral cavity?
continuity of the hard and soft palate, strength and coordination of suck, gag reflex, lumps or masses on the gum line
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What are Epstein pearls and where do they present?
small whitish-yellow clusters- epithelial inclusion cells; at the junction of the hard and soft palates and on the gums
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When are Epstein pearls expected to resolve?
by a few weeks of age
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In what population are natal teeth a common variant?
Native American; rarely seen in Causcasian infants
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Where are natal teeth or eruption cysts with teeth usually seen?
lower incisor region
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Why are natal teeth a potential risk?
they are usually immature caps of enamel and dentine with poor root formation and may be very mobile; caus ulceration of tongue, pain w/feeding, presumed risk for aspiration
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If natal teeth are firmly planted, what is a dentist's likely recommendation for tx?
removal may impact future dentition and the dentist may choose to leave it in place and do a f/u exam to assess for complications
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What are possible etiologies for excessive oral secretions?
esophageal atresia, poor swallow from a neurologic abnormality
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How can esophageal patency be assessed?
passage of a large bore (usually 10Fr) OGT with CXR confirmation
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What is the typical presentation of an infant's neck?
normally short, severe shortness may be syndromic
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How can the shape and symmetry of the neck best be observed?
elevate the shoulders, allowing the head to fall back slightly
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Asymmetry of the neck is most likely due to what forces?
in utero positioning
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Is it possible to palpate the thyroid gland?
very difficult to do unless it is enlarged
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What is the etiology of a congenital goiter?
intrauterine deprivation of thyroid hormone, very rare
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What is the most commonly seen neck mass in neonates?
Cystic hygroma
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What is the etiology of cystic hygromas?
development of sequestered lymph channels, which dilate into cysts
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Describe a cystic hygroma.
soft and fluctuant, transilluminate well and usually visualized laterally over the clavicle
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What are the potential risks a/w cystic hygromas?
may cause severe feeding difficulties or airway compromise
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How are cystic hygromas managed?
very small lesions may regress spontaneously, but surgical resection is usually required
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A mass appearing high in the neck may be...?
a thyroglossal duct cyst or a brachial cleft cyst
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Where can a branchial sinus be visualized?
anywhere along the sternocleidomastoid muscle
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Why is surgery often indicated for a branchial sinus?
it may communicate with deeper structures and infx may necessitate surgical removal of the entire sinus tract
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What syndromes present with neck webbing?
Turner, Noonan and Down syndrome
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What birth history increases the risk for clavicle fx?
h/o shoulder dystocia or macrosomia
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How may an infant in pain relating to a clavicular fx present?
nonrespiratory tachypnea
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What might be felt from a manual exam of the clavicles?
movement of the bone ends and crepitus may not be felt soon after birth; or a fx may not be evident for wks until a callus has formed and can be palpated as a mass