Harrison's 308 - Nephrotic & Nephritic Syndromes Flashcards
Criteria of Nephrotic Syndrome: \_\_\_\_ < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypo\_\_\_\_\_\_\_\_\_\_\_ Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyper\_\_\_\_\_\_ (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly \_\_\_\_\_\_) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hyper\_\_\_\_\_\_
Criteria of Nephrotic Syndrome: 3.5 g/day < Proteinuria Hypoalbuminemia Hyperlipidemia (Mostly Periorbital) Edema Hypertension
If 3.5 g/day < Proteinuria is present without all the other criteria of Nephrotic Syndrome. The most probable cause will be -
Advanced Diabetes Mellitus
In Nephrotic Syndrome, There is a inverse relationship between the severity of Proteinuria and ___.
In Nephrotic Syndrome, There is a inverse relationship between the severity of Proteinuria and GFR.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to __, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
______ _________ ______
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or _____, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see ________ of _______ and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to _______.
Minimal Change Disease
Nephrotic Syndrome causing disease that can be Primary or Secondary to HL, Allergies or NSAIDs, in IM we see Effacement of Podocytes and on Uranalysis there is selective proteinuria. In children (~90% of patients) it is very responsive to Steroids.
_____ _________ ____________
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, _____and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and ________ as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, ________ and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. __% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with _______ and ACEi.
Focal Segmental Glomerulosclerosis
Nephrotic Syndrome causing disease that can be Primary or Secondary to HIV, HBV, PVB19 and Hypertension as well as Analgesics, Bisphosphonates and Radiation. It involves mostly Corticomedullary Junction Glomeruli. 50% of patients will reach CRF in 6 years. it is treated carefully with Cyclosporine and ACEi.
___________ _____________
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in ___, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, _____, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to ______, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a ______ glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, Ig_ and __ deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic _________. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for _____________ events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and ________.
Membranous Glomerulonephritis
Nephrotic Syndrome causing disease that is more common in Men, often due to Malignancy, HBV, Malaria, SLE or RA. It Involves a thickened glomerular membrane (IF-Granular), non-selective proteinuria, IgG and C3 deposits and microscopic hematuria. High risk for thromboembolic events. Treated with immunosuppressants; Cyclophosphamide, Steroids and rituximab.
_________
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on _____ _____ stain an apple green birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an _____ ______ birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the mesangium.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the _______.
Amyloidosis
Nephrotic Syndrome causing disease that is mostly part of systemic amyloid deposition. On light microscopy we can see on Congo red stain an apple green birefringence under polarized light in the mesangium.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common ___ cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on ___. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual _______. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and ________-_____ _______. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t _______ the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ____/ ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ___ but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
_______ __________
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Diabetic Nephropathy
Nephrotic Syndrome causing disease that is the most common CHF cause in DM patients, and the cause of ~45% of all patients on RRT. Pathogenesis involves Thickening of BM, Mesangium Dilation and eventual sclerosis. There is direct correlation between retinopathy and Kimmelsteil-Wilson Nodules. It is the only GN disease that doesn’t shrink the kidney. Treated with ACEi / ARB but never both.
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
\_\_\_\_uria with casts and \_\_uria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria
Subnephrotic Range ______uria (
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) \_\_\_\_emia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hyper\_\_\_\_\_ Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyper\_\_\_\_\_\_\_\_\_
Nephritic Syndrome Criteria:
Hematuria with casts and Pyuria Subnephrotic Range Proteinuria (<3.5 mg/day ) Azotemia Hypertension Hyperparathyroidism
________________ ___________
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries ______. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type _ . presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, _______ edema, ___-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-_____. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but ________ on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and ______ on IF.
Post-streptococcal Glomerulonephritis
Nephritic Syndrome causing disease that is more common in developing countries children. Results from M protein antigen mimicry - HSN Type 3. presents with flack pain, periorbital edema, cola-urine due to glomerular inflammation, low C3 levels, IC complexes, Anti-DNase. usually subclinical with no need for biopsy but hypercellular on LM and Granular on IF.
______ ________
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE.
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE.
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in _______-_______ females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves _____________ and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-___ antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with __ deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and _______pathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of ____ in SLE
Lupus Nephritis
Nephritic Syndrome causing disease that occurs mostly in african-american females involves hypocomplementia and Anti-DNA antibody. Six Types of this disease are possible with IC deposition and Coagulopathies and have to occur with extrarenal inflammatory signs. Most common cause of death in SLE
Membranoproliferative Glomerulonephritis
3 types involving ________ GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
______________ Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “_________” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low __ with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor _ deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where __ Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ____ will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to ____ and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and ______ ________,may be needed.
Membranoproliferative Glomerulonephritis
3 types involving thickened GBM; type 1 involves a “tram-track” appearance and low C3 with IC deposits, type 2 involves Complement factor H deficiency and C3 Nephritic factor (Convertase activator) and type 3 where C3 Nephritic factor is present also. ESRD will occur in 10 years with poor prognosis’ possible progression to RPGN and plasma exchange ,may be needed.
\_\_\_ \_\_\_\_\_\_\_\_\_\_\_\_ (\_\_\_\_\_\_\_\_ \_\_\_\_\_\_) Most common nephritic syndrome cause. Occurs in children and men up to 40 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 40 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most _________ nephritic syndrome cause. Occurs in children and men up to __y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 40 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI _______. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with _______ _________ __________. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. _______ Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually __% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If _ month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases _________ or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or __________ may be needed.
IgA Nephropathy (Berger Disease)
Most common nephritic syndrome cause. Occurs in children and men up to 30 y.o. Episodic, progresses with respiratory or GI infection. Associated with Henoch Schonlein Purpura. Mesangial Proliferation on LM and IC deposits on IF. Usually 75% wont progress to CRF and will be transient. If 6 month of hematuria is present CRF will be more likely. In severe cases tonsillectomy or Plasmapheresis may be needed.
________ _______
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young _____ and elder _______ . Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type _ cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and __________ are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if __________ (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when _ month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if _____ IF pattern is present.
Goodpasture Syndrome
Classically in young males and elder females. Antibodies against Collagen type 4 cause GBM inflammation following exposure to smoking, oxidants or infections. Anemia and Hemoptysis are present. Biopsy is needed for diagnosis. Poor prognosis if Crescentic (RPGN) pattern is present. Renal transplant is the only definitive treatment possible only when 6 month no Ab are present. Plasmapheresis is the initial tx if Linear IF pattern is present.
What is a cardiac cause of Nephritic Syndrome?
Subacute Bacterial Endocarditis
_______ ___________ ____________
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
____ ______ Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with __ deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. ______________, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated __ and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type _. Treatment involves 4-5 weeks of _____________________ . Good prognosis upon Eradication.
Subacute Bacterial Endocarditis
Flee bitten Kidney on Autopsy, May progress to RPGN. On biopsy Focal proliferation around necrotic regions with IC deposits. Hypocomplementia, Elevated RF and Cryoglobulins type III. Treatment involves 4-5 weeks of antibiotics to eradicate the infection. Good prognosis upon Eradication.
What are the false positive causes of a Dipstick test?
for Albuminuria
Alkalinized Urine
Hematuria
Very Concentrated Urine
What are the false negative causes of a Dipstick test?
for Albuminuria
Very Diluted Urine
What does the test measures:
Leukocytes Nitrite Urobilinogen Albumin pH Hemoglobin Specific gravity Ketone Bilirubin Glucose
Dipstick test
What is a good solution for patients that cannot have 24hr urine collection? (Children or Special Conditions)
Urine Albumin to Creatinine Ratio
What are the normal protein urine range in adults? (maximal value)
1.5 g/day
What are the normal albumin urine range in adults? (maximal value)
0.3 g/day
usually will be 7 mg/day
What are the false positive causes of a Dipstick test?
for Microscopic Hematuria
Myoglobinuria (Rhabdomyolysis)
Sperm in urine with High pH
What are the false negative causes of a Dipstick test?
for Microscopic Hematuria
Large dose of Vitamin C
What is considered as Positive Microscopic Hematuria on dipstick test (number)?
Microscopic Hematuria > 2 RBC/HPF
If Microscopic Hematuria is found along with Dysmorphic ____ or ____ casts and Proteinuria than Glomerular diseases should be considered.
If Microscopic Hematuria is found along with Dysmorphic RBCs or RBC casts and Proteinuria than Glomerular diseases should be considered.
What Nephrotic Syndrome cause is associated with SLE or Penicillamine?
Membranous Nephropathy
