Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

GBA - Y4 > Clinical Approach for CLL, NHL and HL > Flashcards

Clinical Approach for CLL, NHL and HL Flashcards

1
Q

Main Clinical characteristics for Hodgkin Lymphoma?

“PAIRR” of owl eyes for Hodgkin Lymphoma

A 
PREDICTABLE Spread through Lymph vessels
ACCESSIBLE Lymphadenopathy
INFLAMMATORY background on Histo
RESPONDS to Treatment
REED-STERNBERG cells (Inflammatory "Owl eye" reactive cells)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Why does Nodular Lymphocyte Predominant HL

considered NON-classic HL while Lymphocyte rich classical HL does?

A

POPCORN cells (altered RS )+different CDs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q 
4 types of Classic HL with Prognosis:
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_ (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
A 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_ (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
A 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_ (Poor)
Lymphocyte rich classical HL
A 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_
A 
4 types of Classic HL with Prognosis:
Nodular Sclerosing HL (Best)
Mixed Cellularity (Intermediate)
Lymphocyte Depleted (Poor)
Lymphocyte rich classical HL
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

HL Ann Arbor Staging
1- __________________________
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

A

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- ______________________________
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

A

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- ____________________________
4- Extra lymphatic spread; Liver / Bone Marrow

A

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- _____________________________

A

HL Ann Arbor Staging
1- one LAD center on one side of Diaphragm
2- multiple LAD centers on one side of Diaphragm
3- LAD centers on two sides of Diaphragm
4- Extra lymphatic spread; Liver / Bone Marrow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Initial Lymphadenopathy in HL is usually on (Location)

A

Initial Lymphadenopathy in HL is usually on Cervical Lymph nodes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is the possible complication of thoracic/mediastinal spread of HL?

A

Superior Vena Cava Syndrome (Bulky Disease)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the name of the process Lymph nodes go through on HL?

A

Wax and Wane - Constant change in sizes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Main Clinical characteristics for Non-Hodgkin Lymphoma?

“NUN” for Non-Hodgkin Lymphoma

A

NON-RESPONSIVE
UNPREDICTABLE skipping to distant LN
NON-ACCSSESIBLE Lymphadenopathy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

3 types of Classic NHL with Aggressiveness:
_______________ (Indolent)
Diffuse Large B cell Lymphoma (Aggressive )
Burkitt’s Lymphoma (Highly Aggressive PI 100%)

A

3 types of Classic NHL with Aggressiveness:
Follicular Lymphoma (Indolent)
Diffuse Large B cell Lymphoma (Aggressive )
Burkitt’s Lymphoma (Highly Aggressive PI 100%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

3 types of Classic NHL with Aggressiveness:
Follicular Lymphoma (Indolent)
____________________ (Aggressive )
Burkitt’s Lymphoma (Highly Aggressive PI 100%)

A

3 types of Classic NHL with Aggressiveness:
Follicular Lymphoma (Indolent)
Diffuse Large B cell Lymphoma (Aggressive )
Burkitt’s Lymphoma (Highly Aggressive PI 100%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

3 types of Classic NHL with Aggressiveness:
Follicular Lymphoma (Indolent)
Diffuse Large B cell Lymphoma (Aggressive )
___________________ (Highly Aggressive PI 100%)

A

3 types of Classic NHL with Aggressiveness:
Follicular Lymphoma (Indolent)
Diffuse Large B cell Lymphoma (Aggressive )
Burkitt’s Lymphoma (Highly Aggressive PI 100%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

___________ Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

A

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Follicular Lymphoma is an indolent disease that is caused by ______. It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

A

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is __________. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

A

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to _____. It is the second most common NHL.

A

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the _______ ____ common NHL.

A

Follicular Lymphoma is an indolent disease that is caused by t(18:14). It has good prognosis yet it is untreatable. If there’s another mutation on p53 it will transform to DLBL. It is the second most common NHL.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

________________ is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

A

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in ___ of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

A

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed __. Associated with EBV and HHV8.

A

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and _____.

A

Diffuse Large B cell Lymphoma is the most common type of NHL. ~40% of patients will have B symptoms.
Although aggressive progress it is curable in 50% of cases depending on mutations. t(18:14) implies it is a transformed FL. Associated with EBV and HHV8.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(_:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

A

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

28
Q

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). _0% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

A

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

29
Q

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. _0% cure rate if treated within 48hrs.

A

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

30
Q

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 4_hrs.

A

Burkitt’s Lymphoma is that highest growth rate NHL. It is diagnosed by c-MYC mutation (t(8:14)). 80% is Endemic in African Children though it can be sporadic or Immunocompromise related. 80% cure rate if treated within 48hrs.

31
Q 
Major indicators of NHL Poor Prognosis:
Elevated \_\_\_ (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
A 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
32
Q 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
\_\_\_(above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
A 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
33
Q 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
\_\_\_\_ >2 
Ann arbor>2 
Sites of Extranidal Involvement>1
A 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
34
Q 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
\_\_\_\_\_ \_\_\_\_>2 
Sites of Extranidal Involvement>1
A 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2 (Both sides of Diaphragm)
Sites of Extranidal Involvement>1
35
Q 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of \_\_\_\_\_\_\_\_ Involvement>1
A 
Major indicators of NHL Poor Prognosis:
Elevated LDH (from TLS)
Age (above 60)
ECOG >2 (Chemotherapy Damage)
Ann arbor>2
Sites of Extranidal Involvement>1
36
Q

Common treatment in HL:
Anti-___
Anti-CD30
HSC Transplantation

A

Common treatment in HL:
Anti-PD1 = Pembrolizumab (Keytruda)
Anti-CD30
HSC Transplantation

37
Q

Common treatment in HL:
Anti-PD1
Anti-CD__

A

Common treatment in HL:
Anti-PD1
Anti-CD30 = Brentuximab vedotin

38
Q

What secondary Hematological HL treatment complication are unique to HL?

A

Secondary Myelodysplastic Syndromes or Leukemias

39
Q 
Common treatment in NHL: "R-CHOP"
(Anti-CD20?)
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
A 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
40
Q 
Common treatment in NHL: "R-CHOP"
Rituximab
(Alkylating Agent?)
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
A 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
41
Q 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
(Antibiotic type chemo?)
Oncovin (Vincristine)
Prednisone
A 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
42
Q 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
(Anti-Microtubule?)
Prednisone
A 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
43
Q 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
(Immunosuppressant?)
A 
Common treatment in NHL: "R-CHOP"
Rituximab
Cyclophosphamide
Hydroxydaunorubicin
Oncovin (Vincristine)
Prednisone
44
Q

What is a mandatory localized, accessory (to pharmacological intervention) treatment in each of the lymphomas?

A

Radiotherapy

45
Q

CLL is the most common leukemia in the ____, it occurs mostly in adult males.

A

CLL is the most common leukemia in the west, it occurs mostly in adult males.

46
Q

CLL is the most common leukemia in the west, it occurs mostly in adult ____.

A

CLL is the most common leukemia in the west, it occurs mostly in adult males.

47
Q

Diagnosis of CLL is made by two separate measurements (6 months apart) of over _ x 10^9/L CDD23/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

A

Diagnosis of CLL is made by two separate measurements (6 months apart) of over 5 x 10^9/L CDD23/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

48
Q

Diagnosis of CLL is made by two separate measurements (_ months apart) of over 5 x 10^9/L CDD23/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

A

Diagnosis of CLL is made by two separate measurements (6 months apart) of over 5 x 10^9/L CDD23/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

49
Q

Diagnosis of CLL is made by two separate measurements (6 months apart) of over 5 x 10^9/L CD__/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

A

Diagnosis of CLL is made by two separate measurements (6 months apart) of over 5 x 10^9/L CDD23/CD5+ lymphocytes that is confirmed by presence in bone marrow biopsy.

50
Q

What cells are present on peripheral blood smear of CLL ?

A

Smudge cells (aka basket cells)

51
Q

CLL that presents with the proteins CD__ or ZAP70 have poor prognosis.

A

CLL that presents with the proteins CD38 or ZAP70 have poor prognosis.

52
Q

CLL that presents with the proteins CD38 or ZAP__ have poor prognosis.

A

CLL that presents with the proteins CD38 or ZAP70 have poor prognosis.

53
Q

Poor prognosis indicators on Cytology of CLL: -1_p and -11q.
“Primal Numbers on 10-20”

A

Poor prognosis indicators on Cytology of CLL: -17p and -11q.
“Primal Numbers on 10-20”

54
Q

Poor prognosis indicators on Cytology of CLL: -17p and -1_q.
“Primal Numbers on 10-20”

A

Poor prognosis indicators on Cytology of CLL: -17p and -11q.
“Primal Numbers on 10-20”

55
Q

Good prognosis indicators on Cytology of CLL: -1_q

“Primal Numbers on 10-20”

A

Good prognosis indicators on Cytology of CLL: -13q

“Primal Numbers on 10-20”

56
Q

What are the hematological complications of CLL?

“AIR”

A

AIHA
ITP
RICHTER transformation (to DLBL)

57
Q

Rai Staging for CLL:
0: _________ Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

A

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

58
Q

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + _____________
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

A

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

59
Q

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +_________
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

A

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

60
Q

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of _____
IV: Lymphocytosis + Any case of Thrombocytopenia

A

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia

61
Q

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of ______________

A

Rai Staging for CLL:
0: Lymphocytosis Alone
I: Lymphocytosis + Lymphadenopathy
II: Lymphocytosis + Lymphadenopathy +Splenomegaly
III: Lymphocytosis + Any case of Anemia (Hb < 11)
IV: Lymphocytosis + Any case of Thrombocytopenia (Plt < 150K)

62
Q

What are the patients that should be started on treatment in CLL?

A

Rai Staging > II

Binet Staging > C

63
Q

(Purine Analogue?) and Chlorambucil/Bendamustine are common drugs that are involved in treatment of CLL.

A

Fludarabine and Chlorambucil/Bendamustine are common drugs that are involved in treatment of CLL.

64
Q

Fludarabine and (Alkylating Agent?) are common drugs that are involved in treatment of CLL.

A

Fludarabine and Chlorambucil/Bendamustine are common drugs that are involved in treatment of CLL.

65
Q

(Anti-52?) is used less than Rituximab in treatment of CLL, due to it’s more robust immunosuppression.

A

Alemtuzumab is used less than Rituximab in treatment of CLL, due to it’s more robust immunosuppression.

GBA - Y4 (12 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions