H&N17 - Anatomy of the Eye & Orbit Flashcards

1
Q

6 features of the orbital cavity

Shape
Borders (roof, floor, lateral and medial walls)
Contents x5
Blood Supply (arterial and venous drainage)
Nerve Innervation (GS, SS, motor)
Anatomical Relations x3

A

1.) Shape - pyramidal shaped with apex pointing posteriorly

  1. ) Borders
    - roof: frontal bone and sphenoid (lesser wing)
    - floor: maxilla, palatine, zygomatic bones
    - medial wall: ethmoid, maxilla, lacrimal, sphenoid (body)
    - lateral wall: zygomatic bone and sphenoid (greater)

3.) Contents - eyeball (globe), extra-ocular muscles, fat, neurovasculature, lacrimal apparatus

  1. ) Blood Supply - main arterial supply is the ophthalmic artery (ICA) and its subsequent branches
    - ophthalmic veins (superior and inferior) drain into the cavernous sinus, pterygoid plexus and facial vein
  2. ) Nerve Innervation
    - general sensation: ophthalmic nerve (Va)
    - special sensation (vision): optic nerve
    - motor innervation: CN III, IV, VI
  3. ) Anatomical Relations - paranasal sinuses (maxillary and ethmoid), nasal cavity, anterior cranial fossa
    - implications for orbital trauma and infection spread
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2
Q

6 openings into the orbital cavity and their contents

4 Main
2 Others

A
  1. ) Optic Canal - optic nerve and ophthalmic artery
  2. ) Superior Orbital Fissure - CN III, IV, Va, VI
  3. ) Inferior Orbital Fissure - maxillary nerve (Vb), inferior ophthalmic vein, sympathetic nerves
  4. ) Nasolacrimal Canal - medial wall of the orbit
    - drains tears from the eye to the nasal cavity
  5. ) Others - supraorbital foramen and infraorbital canal
    - carry small neurovascular structures
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3
Q

4 features of orbital blow out fractures

Location
Cause
Complications x2
Signs x4

A

1.) Location - floor of the orbit and medial is the weakest part of the orbital cavity so most susceptible to fractures

  1. ) Cause - sudden increase in intra-orbital pressure
    - often retropulsion of the globe by fist or a ball
  2. ) Complications - orbital contents can prolapse and bleed into the maxillary or ethmoidal sinus
    - fracture site can trap structures near orbital floor e.g. extra-ocular muscles or soft tissue
  3. ) Signs - periorbital swelling and pain
    - can’t fully elevate eyeball (inferior rectus entrapment)
    - diplopia worse on vertical gaze
    - maxillary nerve damage causes numbness over cheek, lower eyelid, and upper lip on affected side
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4
Q

5 features of the eyelids

Function
Contents x5
Meibomian Glands (and related pathology)
Sebaceous Glands (and related pathology)
Blepharitis
A

1.) Function - protects the front of the eye

  1. ) Contents - skin, SC tissue, tarsal plate, glands
    - muscles: orbicularis oculi (palpebral part) and LPS
  2. ) Meibomian Glands - secrete oily substance onto lid edges to prevent evaporation of tear film and spillage
    - sit within the tarsal plate
    - if blocked, produces a Meibomian cyst (painless)
  3. ) Sebaceous Glands - associated w/ eyelash follicle
    - if blocked during infection causes a stye
  4. ) Blepharitis - inflammation of the eyelids
    - includes skin, lashes and Meibomian glands
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5
Q

What is the orbital septum and its function

2 types of cellulitis within the eye

A

1.) What is it? - thin fibrous sheet originating from the orbital rim periosteum, blends w/ the tarsal plates

  1. ) Function - separates SC tissue and orbicularis oculi muscle from intra-orbital contents
    - prevents deeper spread of superficial infections
  2. ) Periorbital (pre-septal) Cellulitis - infection within the eyelid tissue superficial to the orbital septum
    - secondary to superfical infections e.g. bites, wounds, or bacterial sinusitis in children
    - ocular function is unaffected
  3. ) Orbital (post-septal) Cellulitis - infection within the orbit, posterior to the orbital septum
    - symptoms: exopthalmous, reduced +/- painful eye movements, reduced visual acuity (optic nerve affected)
    - complication: can spread intracranially (venous drainage) –> cavernous sinus thrombosis or meningitis
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6
Q

3 features of the lacrimal apparatus

Tear Production
Tear Drainage
Effect of Blinking

A
  1. ) Tear Production - by the lacrimal gland
    - sits lateral and above the eye
  2. ) Tear Drainage - drained into the nasal cavity
    - lacrimal ducts are canaliculi and nasolacrimal duct
    - obstruction leads to epiphora (overflow of tears over the lower eyelid)
  3. ) Blinking - orbicularis oculi (palpebral part)
    - distributes tear film across the front of the eye, rinsing and lubricating conjunctiva and cornea
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7
Q

3 features of the anatomy of the eyeball

3 Layers
Maintaining Eyeball Position x3
Conjunctiva

A
  1. ) Layers
    - outer: sclera which is continuous anteriorly as the cornea and posteriorly as dura mater covering the CN II
    - middle - iris, ciliary body, and choroid
    - inner: retina
  2. ) Eyeball Position - maintained by:
    - suspensory ligament, extra-ocular muscles, orbital fat
  3. ) Conjunctiva - transparent mucous membrane producing the mucous component of tear film
    - covers the sclera but doesn’t cover the cornea
    - subconjunctival haemorrhage is readily visible but is often painless and self limiting
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8
Q

3 features of focusing light onto the back of the eye (macula)

Transparent Structures
Refractive Structures
Eyeball Shape

A
  1. ) Transparent Structures - so light can pass through
    - lens and cornea are avascular in order to be transparent so they get their nutrients from aq. humour
  2. ) Refractive Structures - bend light to a focal point
    - cornea (main), lens, aqueous and vitreous humour
  3. ) Eyeball Shape - affects ability to see
    - short-sighted: myopia (eyeball too long)
    - long-sighted: hypermetropia (eyeball too short)
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9
Q

What is the accommodation reflex and why is it needed?

3 ways the eye ‘accommodates’

Presbyopia

A

What is it? - reflex action of the eye, in response to focusing on a near object

  • light rays from near objects are more divergent so more refraction is required
  • cornea is fixed in shape so can’t bring into focus
  1. ) Pupils Constrict - limits amount of light entering
  2. ) Eyes Converge - ensures image remains focused on same point of retina in both eyes
  3. ) Lens Becomes Fatter - becomes more biconvex
    - caused by contraction of the ciliary muscle relaxing the suspensory ligament

Presbyopia - age-related inability to focus near objects
- as you get older, the lens become stiffer

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10
Q

4 features of photoreceptors

Rods
Cones
Fovea
Optic Disc

A
  1. ) Rods - see in black and white, active in low-light
    - abundant in peripheral parts of the retina
  2. ) Cones - see in colour, active in high light levels
    - concentrated within the macula of the retina

3.) Fovea - region in retina only containing cones

  1. ) Optic Disc - where retinal ganglion cells collect to go on to form the optic nerve
    - ‘blind spot’ because it has no photoreceptors
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11
Q

blurred vision (decreased acuity) is caused by pathologies affecting ….

3 things

A
  1. ) Transparency - of structures anterior to the retina
    - e.g. cataracts causes the lens to be more opaque
  2. ) Refraction - ability of structures to refract light
    - astigmatism: irregularity of corneal surface
    - shape of eyeball or presbyopia
    - refractive error has no effect on light travelling perpendicular to the cornea (pin-hole testing)

3.) Retina/Optic Nerve - e.g. retinal detachment, age-related macular degeneration, optic neuritis

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12
Q

4 features of aqueous humour

Production
Function
Flow
Drainage

A

1.) Production - by ciliary processes in the ciliary body

  1. ) Function - nourishes the lens and cornea
    - lens and cornea are avascular to be transparent

3.) Flow - from posterior chamber (bathing the lens), through pupil into anterior chamber (cornea)

  1. ) Drainage - through iridocorneal angle
    - via trabecular meshwork into canal of Schlemm
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13
Q

4 features of glaucoma

Definition
Cause
Chronic
Acute

A
  1. ) Definition - optic nerve damage secondary to raised intraocular pressure
  2. ) Cause - blocked drainage of aqeuous humour from the anterior chamber
  3. ) Chronic - open angle glaucoma (most common)
    - trabecular meshwork deteriorates with age
    - increased IOP –> optic disc cupping –> gradual loss of peripheral vision
  4. ) Acute - closed angle glaucoma (emergency)
    - narrowing of iridocorneal angle
    - occurs in older patients (55+, 70-80s most common)
    - symptoms: painful red eye, oval shaped pupil, blurred vision, halo around eyes, nausea and vomiting
    - treament: drugs to reduce IOP then surgery
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