H&N13 - Functional Anatomy & Disorders of the Ear Flashcards

1
Q

6 signs and symptoms of ear disease

A
  1. ) Otalgia - ear pain
  2. ) Tinnitus - ringing in ears
  3. ) Discharge - ottorhoea
  4. ) Vertigo - loss of balance
  5. ) Hearing Loss - conductive or sensorineural
  6. ) Facial Nerve Palsy
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2
Q

5 nerves carrying general sensation to the ear

A
  1. ) Trigeminal (CN V) - auriculotemporal nerve from the mandibular branch (Vc)
    - lateral surface of tympanic membrane, external acoustic meatus
  2. ) Facial (CN VII) - nervus intermedius
    - lateral surface of tympanic membrane, external acoustic meatus, concha
  3. ) Glossopharyngeal (CN IX) - tympanic nerve
    - medial surface of tympanic membrane, middle ear cavity, mastoid air cells
  4. ) Vagus Nerve (CN X)
    - lateral surface of tympanic membrane, external acoustic meatus, concha

5.) C2/C3 Spinal Nerves - lesser occipital nerve (superior pinna) and great auricular nerve (majority of pinna)

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3
Q

Function of the external ear

3 structures of the external ear

A

Function - collects, transmits and focuses sound waves onto the tympanic membrane

  1. ) Pinna - visible part of the ear (auricle)
    - made up of cartilage, skin, and fatty tissue
    - abnormalities can be congenital, inflammatory/infective (perichondritis) or traumatic (haematoma)
  2. ) External Acoustic Meatus - sigmoid shaped pathway running from the pinna to the lateral surface of the tympanic membrane
  3. ) Tympanic Membrane (eardrum) - transmits sound vibrations from outer air to the auditory ossicles
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4
Q

5 features of a pinna haematoma

Definition
Cause 
Type of Haematoma
Complication/Deformity
Treatment
A

1.) Definition - accumulation of blood/serum between cartilage and its overlying perichondrium

  1. ) Cause - secondary to blunt injury to pinna
    - common in contact sports
  2. ) Type of Haematoma - subperichondrial haematoma
    - deprives cartilage of blood supply causing avascular necrosis of tissue
  3. ) Complication - cauliflower deformity
    - untreated leads to fibrosis –> new asymmetrical cartilage development
  4. ) Treatment - drainage and re-accumulation of 2 layers
    - aspirate, compression bandage
    - review in 24hrs, I and D if it reaccumulates
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5
Q

3 features of the external acoustic meatus

Epithelium
Structure
Common Conditions

A
  1. ) Epithelium - keratinised, stratified squamous
    - epithelial migration provides self-cleaning function
    - desquamation and skin migration laterally off tympanic membrane and out of canal
  2. ) Structure - outer 1/3 is cartilaginous and contains:
    - hair, sebaceous and ceruminous glands (ear wax) to form barrier to foreign objects
    - inner 2/3 is bony and has no hair and glands
  3. ) Common Conditions
    - wax/foreign bodies
    - otitis externa (inflammation)
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6
Q

3 common abnormalities of the tympanic membrane

A

1.) Bulging - caused by bacterial acute otitis media

  1. ) Retraction and Fluid Accumulation - fluid and -ve pressure in the middle ear decreases mobility of TM and ossicles which affects hearing
    - caused by otitis media w/ effusion

3.) Cholesteatoma - abnormal skin growth behind the tympanic membrane

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7
Q

4 features of a cholesteatoma

Mechanism
Cause
Symptoms x3
Complication

A
  1. ) Mechanism - retraction of pars flaccida of the TM forms a pocket, trapping epithelium and keratin
    - proliferation of this forms a cholesteatoma
  2. ) Cause - often secondary to chronic ET dysfunction
    - TM is sucked inwards due to negative pressure caused by blockage of the eustachian tube (ET)
  3. ) Symptoms
    - painless, smelly otorrhoea +/- hearing loss
  4. ) Complications - not malignant but slowly expands
    - can destroy structures in the middle and inner ear
    - can erode into the nearby skull and into the brain
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8
Q

Function of the middle ear

2 structures in the middle ear

A

Function - amplify and relay fibrations from the TM to the oval window of the cochlea (inner ear)
- it is an air-filled cavity between the TM and inner ear

  1. ) Ossicles - 3 bones (malleus, incus, stapes) that help transmit sounds to the cochlea
    - movement is dampened by stapedius muscle and tensor tympani which contract to prevent excessive vibrations (acoustic reflex)
  2. ) Pharyngotympanic (Eustachian) Tube - tube that links the middle ear to the nasopharynx
    - its equilibrates the pressure of middle ear with atmospheric pressure
    - mucous membrane continously reabsorbs air in the middle ear causing negative pressure
    - allows ventilation and drainage of mucus
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9
Q

3 features of otosclerosis

What is it?
Aetiology
Complication

A

1.) What is It? - fusion of ossicles at articulations

  1. ) Aetiology - abnormal bone growth, particularly between the base plate of stapes and oval window
    - sound vibrations not transmitted effectively to cochlea
    - exact cause of abnormal bone growth is unknown
    - family history common
  2. ) Complication - causes conductive hearing loss
    - most common cause of hearing loss in young adults
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10
Q

4 features of otitis media

Acute Otitis Media (AOM)
Signs and Symptoms of AOM x3
Otitis Media w/ Effusion (‘glue ear’)
Common Demographic

A

1.) Acute Otitis Media - acute middle ear infection
- often viral infections but can be bacterial
(S. pneumoniae, H. influenzae)

2.) Signs and Symptoms - otalgia, systemic symptoms, red +/- bulging tympanic membrane

  1. ) Otitis Media w/ Effusion - collection of fluid within the middle ear without signs of inflammation
    - can predispose to ET dysfunction (-ve pressure) —> cholesteatoma
    - most resolve spontaneously in 2-3 months
    - management: grommets (tympanostomy tube) to maintain equilibration of pressures
  2. ) Common in Infants - ET is shorter and horizontal
    - easier passage for infection from nasopharynx
    - tube is blocked easier, compromising ventilation and drainage of mucus, increasing risk of infection and glue ear
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11
Q

4 complications of acute otitis media

A
  1. ) Tympanic Membrane Perforation - can lead to hearing loss and increase susceptibiltiy to infections
  2. ) Facial Nerve Lesions (rare) - chorda tympani and stapedius nerve runs through the middle ear cavity
  3. ) Mastoiditis - middle ear infections can spread to the mastoid bone due to communication w/ mastoid air cells
    - communication via the mastoid antrum
  4. ) Intracranial Complications - spreading of infection into the skull can lead to:
    - meningitis, sigmoid sinus thrombosis, brain abscess
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12
Q

2 features/structures in the inner ear

A

1.) Cochlea - fluid-filled tube w/ specialised hair cells (stereocilia) that converts fluid movement into sound

  1. ) Vestibular Apparatus - fluid-filled tubes w/ stereocilia converting fluid movement into position sense/balance
    - includes semicircular ducts, the saccule and utricle
    - fluid movement due to position and rotation of head moves stereocilia which generate APs via CN VIII
    - helps to perceive and maintain our sense of balance
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13
Q

5 steps/structures in allowing us to hear

External Ear
Tympanic Membrane
Ossicles
Stereocilia
Primary Auditory Cortex
A
  1. ) External Ear - auricle and external auditory meatus funnels sound waves towards the tympanic membrane
  2. ) Tympanic Membrane - vibrations of the tympanic membrane spreads to the ossicles
  3. ) Ossicles - vibrations occur in stapes at oval window
    - sets up vibrations in cochlear fluid in the cochlear duct
  4. ) Stereocilia - detect movement in cochlear fluid
    - found in a part called the spiral organ of corti
    - movement of stereocilia triggers APs in cochelar part of the vestibulocochlear nerve (CN VIII)

5.) Primary Auditory Cortex - makes sense of the input from CN VIII

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14
Q

5 conditions affecting the inner ear

Presbyacusis
Benign Paroxymal Postional Vertigo
Meniere's Disease
Acute Labrynthitis
Acute Vestibular Neuronitis
A
  1. ) Presbycusis - bilateral and gradual sensorineural hearing loss associated with old age
  2. ) Benign Paroxysmal Positional Vertigo - short episodes (seconds) of vertigo triggered by the movement of the head

3.) Meniere’s Disease - recurrent episodes of vertigo,
unilateral hearing loss, and tinnitus, +/- nausea/vomiting
- symptoms last 30 mins up to 24 hours
- hearing deteriorates over time

  1. ) Acute Labrynthitis - history of URT infection w/ vertigo, hearing loss, tinnitus and vomiting
    - involvement of all inner ear structures
  2. ) Acute Vestibular Neuronitis - history of URT infection w/ severe vertigo and sudden onset of vomiting
    - usually no hearing disturbance or tinnitus
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15
Q

2 types of hearing loss (and causes)

A
  1. ) Conductive - caused by pathology involving the external or middle ear
    - e.g. wax, otitis media (both types), otosclerosis
  2. ) Sensorineural - caused by pathology involving the inner ear or CN VIII
    - e.g. presbyacusis, Meniere’s disease, acute labyrinthitis
    - acoustic neuroma, noise-related hearing loss, ototoxic medications
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16
Q

7 non-otological causes of ear pain (otalgia)

structures sharing CN sensory innervation w/ the ear

A
  1. ) Nose and Sinuses - maxillary branch of trigeminal (Vb)
  2. ) Teeth and TMJ - mandibular branch of trigeminal (Vc)
  3. ) Parotid Gland - mandibular branch of trigeminal (Vc)
    - also the facial nerve (VII) as it runs through it
  4. ) Tongue and Oropharynx - Vc (lingual branch) and IX
  5. ) Larynx and Laryngopharynx - vagus nerve (X)
  6. ) Oesophagus - vagus nerve (X)
  7. ) Cervical Spine - C2 and C3
17
Q

Rinne’s and Weber’s Test

A

Rinne’s - tests conductive hearing (place on mastoid process)

  • positive test is normal (air > bone conduction)
  • negative test is abnormal ( bone > air conduction)

Weber’s - tests sensorineural and conductive hearing (place on middle of forehead)

  • negative test is normal (equally loud in both ears)
  • conductive: louder in impaired ear
  • sensorineural: louder in unimpaired ear