Gynecology Flashcards

1
Q

Day 1 of the menstrual cycle is slated by the onset of _____?

A

Menses

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2
Q

Days 1-14 of the menstrual cycle are described as which phase?

A

Follicular

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3
Q

Day 14 is typically the day of ________?

A

Ovulation

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4
Q

Days 14-30 of the menstrual cycle are described as which phase?

A

Luteal

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5
Q

GnRH from the hypothalamus stimulate which organ?

A

Anterior pituitary

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6
Q

The anterior pituitary, once stimulated by GnRH, secretes ____ and _____?

A

FSH and LH

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7
Q

LH is responsible for which stimulating production of which hormone(s)?

A

Androgens and progesterone

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8
Q

LH stimulates what process?

A

Ovulation

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9
Q

FSH is responsible for stimulating production of which hormone(s)?

A

Estrogen (E2) “Estradiol”

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10
Q

What are signs of Estrogen excess?

A
  • dysmenorrhea
  • menorrhagia
  • nausea
  • edema
  • enlarged uterus/fibroids
  • fibrocystic breast changes
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11
Q

What are signs of Estrogen deficiency?

A

Scant menses and mid-cycle spotting

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12
Q

What are signs of progesterone excess?

A
  • Edema
  • Bloating
  • Weight gain
  • Fatigue
  • HTN
  • Varicose veins
    “Things you would expect in a pregnant woman”
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13
Q

What are signs of progesterone deficiency?

A
  • Endometriosis
  • Adenomyosis
  • Endometrial hyperplasia
  • Prolonged/heavy menses
  • Severe cramps
  • Luteal spotting and BTB (breakthrough bleeding)
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14
Q

How many days are in a “normal” menstrual cycle?

A

21-35 days

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15
Q

Define amenorrhea

A

absence/abnormal cessation of menses for more than 3mo

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16
Q

Define oligomenorrhea

A

scant menses; periods occur >35 days apart

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17
Q

Define polymenorrhea

A

menses of increased frequency; periods occur <21 days apart

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18
Q

Define menorrhagia

A

AKA Hypermenorrhea; prolonged or profuse menses (>7days or 80cc)

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19
Q

Define metrorrhagia

A

any irregular uterine bleeding between cycles; spotting out of cycle

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20
Q

Define menometrorrhagia

A

both profuse bleeding during menstruation and between periods

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21
Q

Define dysmenorrhea

A

painful menses

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22
Q

Define mittelschmertz

A

pain with ovulation; “middle of month pain”

- typically unilateral, front or back

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23
Q

Define contact bleeding

A

cervix bleeding from tip of condom/penis/manual physical exam

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24
Q

What would contact bleeding indicate?

A

Cervical cancer, cervicitis (CT/Gon), period starting

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25
Q

What is dyspareunia?

A

Painful intercourse

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26
Q

Why could there be pain with entry at introitus (vaginal opening)?

A
  • vaginismus mm. contractions

- infection, sores, low Estrogen

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27
Q

Why could there be pain with sexual friction?

A
  • infection

- low estrogen

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28
Q

Why could there be pain with deep penetration?

A
  • Pelvic pathology

- Cervix/uterus/adnexal inflammation

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29
Q

Which hormone is responsible for reducing vaginal pH and increasing cervical pH?

A

Estrogen

  • low vag pH to prevent infection
  • high cx mucous pH to welcome sperm
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30
Q

Which hormone is responsible for vaginal cornification?

A

Estrogen

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31
Q

Which hormone and contraceptives is/are responsible for decreasing cervical pH?

A

Progesterone

- Progesterone OCP, IUD, Prog injection

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32
Q

T/F: Hypothyroid disease often presents with hypermenorrhea (menorrhagia)

A

True

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33
Q

What is primary amenorrhea?

A

No secondary sex characteristics by age 14; no menses by age 16

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34
Q

What are potential causes for primary amenorrhea?

A

CNS hypothalamic disorder, hymen blockage, eating disorder, hypoglycemia, thyroid condition

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35
Q

What is secondary amenorrhea?

A

no menses for 3 cycles OR 6 months (whichever sooner), in a woman with previous menses

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36
Q

What are potential causes of secondary amenorrhea?

A

PREGNANCY, weight changes, stress/depression, thyroid conditions, PCOS, increased prolactin, early menopause, medications

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37
Q

Hypoestrogenic amenorrhea puts women at a higher risk for developing which condition?

A

osteoporosis d/t bone mineral density loss;

  • also at risk for dyslipidemia, DM, breast cancer
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38
Q

Which initial labs are important to order for amenorrhea evaluation?

A

b-HCG, TSH, PRL;

  • also r/o estrogen deficiency
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39
Q

When does exercise induced amenorrhea occur?

A

Body fat goes below 15-19%, BMI<18;

- usually not just d/t exercise alone- nutrition is often a factor

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40
Q

What initial labs should you order if you suspect premature ovarian failure?

A

FSH, LH and E2 (rule out Estrogen deficiency)

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41
Q

What age category would you consider for premature ovarian failure?

A

<40 y.o.

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42
Q

What are the criteria for Dx of PCOS?

A

1- oligomenorrhea
2- hyperandrogenism
3- exclusion of other known disorders
**polycystic ovaries not required

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43
Q

What are S/Sx of PCOS?

A

hirsutism, anovulation, + FGT, +2hr PPGT, +insulin, increased free Testosterone,

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44
Q

What is the proper management of PCOS?

A

Meds - Progesterone, Spironolactone, Metformin
Supplements - soy/flax, saw palmetto, green tea
Other - High PRO/Low CHO diet

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45
Q

What are common causes of menorrhagia?

A

pregnancy (r/o placenta prev.), infection, IUD, fibroids/polyps, Hypothyroid, blood disorder, neoplasms

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46
Q

What does a workup of menorrhagia consist of?

A

urine pregnancy -> STI screen -> endocrine workup -> PAP -> U/S -> biopsy

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47
Q

What are the S/Sx of cervicitis?

A
  • Chronic:* thick yellow d/c w. no bacterial etiology

* Acute:* (STI- NG, CT, trick), acute trauma, polyps and cancer

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48
Q

How do you diagnose cervicitis?

A

Pap smear and/or biopsy

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49
Q

What is adenomyosis? What are some sx?

A

Endometrial glands + stroma grow into the uterine wall, creating a spongelike effect;
Sx: associated w/ heavy, painful periods & uterine enlargement.

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50
Q

What is Dysfunctional uterine bleeding (DUB)?

A

Anovulatory cycles and endometrium overgrowth

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51
Q

How do you diagnose DUB?

A

Dx of exclusion w/ irregular menstrual intervals

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52
Q

What are treatment options for DUB?

A
  • Stabilize estrogen via: diet, exercise, GnRH agonist- Lupron
  • Anti-inflammatory via NSAIDs
  • Limit endometrial overgrowth via Progesterone in any form (cream, pill, IUD)
  • Dilatation and Curretage
  • Endometrial ablation
  • Historectomy
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53
Q

What is the most common solid tumor in women?

A

Uterine fibroids AKA Leiomyomata, Leiomyoma, Fibromyoma, Myoma

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54
Q

What makes uterine fibroid removal/surgery controversial?

A

They are 1% malignant (leosarcoma) and the process may lead to a spread

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55
Q

T/F: Nulliparous women have increased risk of uterine fibroid.

A

True

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56
Q

What are Sx of uterine fibroids?

A

50-80% are asymptomatic

  • UF are the MC cause of abnormal uterine bleeding
  • Pain is NOT typical
  • Urinary abnormalities
  • “Pressure”, bloating, heaviness
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57
Q

How do you dx uterine fibroids?

A

Pelvic ultrasound, however it is not definitive

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58
Q

When would surgery be an option for uterine fibroids?

A
  • Bleeding causes severe anemia
  • Unmanageable bleeding
  • Severe dysmenorrhea
  • Pelvic pain
  • Urinary tract compression- pt tolerant level and kidney function compromised
  • Infertility
  • Rapid growth
  • Affects adnexal evaluation
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59
Q

What is endometrial hyperplasia?

A

Overgrowth of endometrial cells in the endometrium.

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60
Q

What are sx of endometrial hyperplasia?

A
  • abnormal bleeding

- postmenopausal bleeding

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61
Q

How do you dx endometrial hyperplasia?

A
  • Pelvic US Endometrial stripe
  • Endometrial biopsy
  • D&C
  • Hysterectoscopy
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62
Q

What are treatment options for endometrial hyperplasia?

A

Progesterone, dietary changes, exercise, possible D&C, ablation, hysterectomy

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63
Q

What is a dangerous warning sign for endometrial cancer?

A

Postmenopausal bleeding, postmenopausal pap with abnormal cells, premenopausal inter menstrual bleeding

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64
Q

What is the difference between primary and secondary dysmenorrhea?

A

Primary - no underlying pelvic pathology (dx of exclusion)

Secondary - there IS underlying pathology

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65
Q

What are sx of primary dysmenorrhea?

A

Abdominal/pelvic/Low back pain that begins with onset of menses and lasts 8-72hrs, headache, diarrhea, N/V

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66
Q

What are common/possible etiologies for secondary dysmenorrhea?

A
Ovarian disorders (Endometrioma, cysts/neoplasm)
Uterine disorders (Fibroids, adenomyosis, endometriosis, PID, IUD)
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67
Q

What are sx of secondary dysmenorrhea?

A
  • Onset after painless menstrual cycles in past
  • Pain during times other than menses
  • Infertility
  • Heavy flow
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68
Q

What are the labs to R/O secondary amenorrhea?

A
  • Cervical culture, hCG, CBC/UA/ESR
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69
Q

What is the prostaglandin theory?

A

LH & Progesterone increases the release of local Pgs, the endometrium increases prostaglandin production as a response to progesterone withdrawal

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70
Q

What is the required timeframe to dx chronic pelvic pain (CPP)?

A

6 months

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71
Q

What is endometriosis?

A

Presence of endometrial glands and stroma outside the uterus

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72
Q

What is the clinical presentation fora woman with endometriosis?

A
  • Pelvic pain @ ovulation, before/during menses
  • dyspareunia
  • infertility
  • LB/leg pain
  • severe dysmenorrhea
  • irregular or heavy menses
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73
Q

How do you dx endometriosis?

A

laparoscopy is gold standard

- appearance of blue-grey powder burned lesions

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74
Q

How do you treat endometriosis?

A
  • Analgesics
  • Endocrine therapy
  • Surgery
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75
Q

What is PMS? (premenstrual syndrome)

A

Monthly recurrence of mood, cognitive or physical symptoms during the luteal phase, remits with menses
Confirmed w/ > 2 cycles

76
Q

What is PMDD? (premenstrual dysphoric disorder)

A

Psychosocial impairment, Sx worsen in luteal phase (after ovulation) and BEGIN to remit in menses.
7 symptom free days in follicular phase

77
Q

What is PMM? (premenstrual magnification)

A

Distressing physical or affective symptoms THROUGHOUT the cycle, symptoms may NEVER remit
only one that may be an exacerbation of underlying condition

78
Q

What are the different types of functional cysts?

A
  • Follicular cyst (MC ovarian mass)
  • Corpus luteum cyst
  • Theca lutein cyst (rare)
79
Q

T/F: Endometrioma, PCOS and tubo-ovarian abscesses are all examples of ovarian masses

A

True

80
Q

When would a corpus luteum cyst develop?

A

if the sac doesn’t dissolve, but seals off after the egg is released –> Fluid builds up inside

81
Q

Are corpus luteum cysts clinically important?

A

Yes, with the possibility of bleeding/torsion; whereas the other two functional cysts are not clinically important.

82
Q

What are Sx of functional ovarian cysts?

A
  • often asymptomatic & discovered during routine pelvic exam
  • unilateral pressure, fullness, or pain in lower abd
83
Q

Which cyst contains all 3 germ layers?

A

Dermoid cyst (Teratoma)

  • could be benign or malignant
  • “adnexal calcifications”*
84
Q

What is an endometrioma?

A

Part of endometriosis picture, with blood-filled cysts forming on ovaries requiring laparoscopy

85
Q

What is the typical cause of tubo-ovarian abscess?

A

Infection at the junction; d/t NG or CT

86
Q

What are sx of tubo-ovarian abscess?

A
  • Pelvic pain
  • Fever
  • Vaginal d/c
  • Tubal/ovarian swelling
    long term: CPP, infertility
87
Q

At what stage in life are ovarian masses most likely malignant?

A

In postmenopausal women (45% malignancy risk)

88
Q

What are some risk factors of ovarian cancer?

A

Fam Hx, Prior BRCA, Nullparity, early menarche or late menopause, fertility promoting drugs, ashkenazi jews

89
Q

What are some s/Sx of ovarian cancer?

A
  • Non specific GI Sx
  • Maybe asymptomatic
  • pelvic pressure
  • No spotting or other red flags for early detection
90
Q

T/F: Hormonal contraceptives and breast feeding help prevent ovarian cancer

A

true

91
Q

Where is the MC site for fibrocystic breast changes or malignant disease?

A

Upper outer quadrant

92
Q

Why is the inframammary line important?

A

Common fibrous area due to bras

93
Q

What is mastalgia and what are the s/sx?

A

Breast pain/tenderness common in premenopausal women. Can be cyclical or non-cyclical

94
Q

What is fibrocystic breast changes?

A

Benign condition w/ bilat diffuse changes with hormone fluctuation.
- Masses that don’t reduce with menses should be evaluated further

95
Q

What is fibroadenoma?

A

Fibrous stroma in breast that responds to E/P, size fluctuates with cycle.
- rubbery, firm, smooth, round, mobile, painless

96
Q

Which age do women get fibroadenoma and simple cysts?

A

15-50 yrs, NOT common in menopause unless on HRT

97
Q

What are characteristics of simple cysts?

A
  • Fluid filled breast lesions
  • Soft, firm, mobile, well-circumscribed, uni/bilat, TENDER
  • cyclical fluctuations
98
Q

What is mastitis?

A
  • infection with lactation or skin disruption

- MC in 2-4wks post partum

99
Q

What are s/sx of mastitis?

A

fever, erythema, pain, induration, N/V, malaise, chills

100
Q

What is the etiology of mastitis?

A

S. aureus, S. epidermis, strep

101
Q

What is a galactocele and how do you treat?

A

Obstruction of breast duct, becomes tender and enlarged.

Tx- excise and drain

102
Q

What is the MC cause of nipple discharge?

A

benign breast disease

103
Q

What is the MC pathologic cause of nipple discharge?

A

intraductal papilloma (still benign but pathologic)

104
Q

What characteristics of nipple discharge seem pathologic?

A
  • unilateral
  • spontaneous
  • frank blood, serous, green/grey
  • any other color (d/t carcinoma)
105
Q

What is galactorrhea and what is commonly the cause?

A

Inappropriate lactation in non puerperal woman

  • uni OR bilateral
  • evaluate for elevated PROLACTIN levels (r/o pituitary tumor)
106
Q

What is intraductal papilloma?

A

papillary growth inside lactiferous duct, d/c may be bloody or serous
Tx- surgical excision

107
Q

What causes subareolar abscess?

A

s. aureus or anaerobic organisms
- common in women w/ inverted nipples or nipple piercings
Tx- antibiotics, drainage, excision

108
Q

What is MC cancer in women?

A

Breast cancer

109
Q

What is leading cause of cancer deaths in women?

A

Lung cancer (Breast is second, cause there are two boobs)

110
Q

When would you order MRI instead of US if you suspected breast cancer?

A
  • current or past dx of BrCa
  • dense breasts
  • high risk with fam hx
111
Q

What are s/sx of early stage BrCa?

A
  • firm/hard mass
  • irregular contour
  • immobile
  • unilateral
112
Q

What are s/sx of late stage BrCa?

A
  • skin/nipple retraction
  • tenderness
  • axillary
    lymphadenopathy
  • erythema/edema/ulcer
  • pain
  • fatigue
  • PEAU D’ORANGE
113
Q

Risk factors for BrCa?

A
  • Age (older)
  • Sex (Female)
  • Race (white)
  • Genetic mutations
  • Hormone use
  • Breast feeding (decr. risk)
  • Years of exposure to ovarian estradiol
114
Q

How do you diagnose BrCa?

A

Biopsy

Screen w/ self and clinical breast exams and mammography

115
Q

What is Paget’s disease of the breast?

A
  • Adenocarcinoma of the nipple
    “Itching or burning of the nipple”
  • May also be erythema, rash and ulcer
116
Q

How is cervical dysplasia graded?

A
  • Mild (CIN 1)
  • Mod (CIN 2)
  • Severe (CIN 3)
    CIN = cervical intraepithelial neoplasia
    SIL = (low or high) squamous intraepithelial lesion
117
Q

T/F: Family history is not a risk factor for cervical cancer.

A

True

118
Q

What are RF for cervical cancer?

A
  • Early age of first intercourse
  • Multiple sex partners
  • HPV infection
  • Smoking
  • Hormonal contraception > 5yrs
  • exposure to DES
119
Q

When is a colposcopy performed?

A

To identify abnormal areas that require biopsy after the Pap smear screening test showed abnormal cells

120
Q

What is the recommended pap smear frequency?

A

Screening starts within 3 years after having vaginal intercourse or by age 21; every 1-2 years

121
Q

What is LEEP?

A

Fine wire loop with electrical energy to remove tissue

122
Q

What is conization?

A

Removes a cone-shaped piece of the cervix, may interfere with future childbearing

123
Q

What is DES and side effects?

A

Diethylstilbestrol; synthetic estrogen

- high risk of BrCa, infertility, cx dysplasia, autoimmune disorders, reproductive anomalies

124
Q

What is endometrial adenocarcinoma?

A

Uterine cancer

125
Q

What is the typical presenting complaint with endometrial adenocarcinoma?

A

abnormal bleeding

126
Q

What are the RF for endometrial adenocarcinoma?

A

Age 50-70, Fam hx and unopposed estrogen

- nulliparity, PCOS, tamoxifen, diabetes, HTN

127
Q

How many pregnancies are unintended in USA?

A

50%, MC 20-24yrs

128
Q

By what mechanism do E/P hormonal contraceptives work?

A
  • Suppress FSH/LH surge, inhibiting follicular maturation- no ovulation, Prog thickens Cx mucous, alters endometrial lining
129
Q

T/F: Nonoral methods of contraception have lower user failure rates and thus greater reliability

A

True; less human error

130
Q

What hormone does “the patch” use?

A

Progestin, changed weekly for 3 weeks

131
Q

What hormone does NuvaRing use?

A

Progestin, take out for menses.

may cause bacterial vaginosis

132
Q

What was the major issue with injectable hormones (lunelle, depo-provera)?

A

difficulties getting pregnant quickly after stopping, osteopenia

133
Q

The Mirena IUD secretes which hormone?

A

levonorgestrel, a synthetic similar to progesterone

  • spotting is MC side effect
134
Q

What are contraindications to taking P/E HCPs?

A

Liver disease, pregnancy, HTN, neurological migraines, breast cancer, smokers

135
Q

What are drug interactions with HCPs?

A

Tylenol, Alcohol, Antibiotics, St Johns wort, antidepressants, CS, bronchodilators, rifampin

136
Q

When would you take progestin only pills?

A

women who can’t take estrogen (breast feeding and risk for blood clots)

137
Q

What is Norplant?

A

Silicone rods w/ progesterone placed subcutaneously for 5yrs

138
Q

What is the minipill?

A

Prog only OC

139
Q

What element does a non-hormonal IUD use?

A

Copper

140
Q

What is the mechanism of a copper IUD?

A

reduces sperm motility (copper is its kryptonite), prevents fertilization

141
Q

When would IUD be contraindicated?

A

abnormal uterine anatomy, nulliparous, pregnancy, DUB of unknown cause, malignancy, allergy to copper

142
Q

Which receptacles are suitable for spermicide?

A

condoms, diaphragm, cervical cap

143
Q

What are possible side effects of vasectomy?

A

pain, infection, granulomas, epididymitis, abscesses, ED,

144
Q

What is Preven?

A

emergency contraception similar to plan B

145
Q

What methods are used for natural family planning?

A

cervical mucous, BBT, calendar, symptothermal

146
Q

T/F: IUD can be used as an emergency contraceptive

A

True

147
Q

What is non surgical abortion RU486?

A

Mimics SAB via Mifepristone (Antiprogesterone)

- Must be < 49d since LMP

148
Q

What are the contraindications and side effects of RU486?

A

SE- cramps, nausea, bleeding, retained tissue, need to undergo surgical abortion
Contra- ectopic pg or adnexal mass, IUD, CS therapy, hemorrhagic disorders

149
Q

What hormonal changes occur during perimenopause?

A
  • Rising FSH = marker of perimenopause*

- Ovaries become sporadically responsive to pituitary FSH/LH, with decreased gonadal hormone output

150
Q

What signifies the onset of menopause?

A

Absent menses for 12 months and elevated FHS/LH with low Estrogen

151
Q

What is the average age of menopause?

A

50-51yo

152
Q

What are the symptoms of vulvar cancer?

A
  • itching, burning, soreness
  • lump/mole/growth
  • color change (white/erythematous)
153
Q

What are symptoms of vulvodynia/vulvar vestibulitis?

A

Chronic perineal discomfort (burning, stinging, irritation), altered cutaneous perception

154
Q

Describe what occurs with cystocele

A

Herniation of the bladder wall causing an outpouching of anterior vaginal wall
- may be asymptomatic or cause incontinence

155
Q

Describe what occurs with rectocele

A

Herniation of rectal wall causing an outpouching of posterior vaginal wall
- constipation or the need to apply digital pressure in the vagina in order to defecate

156
Q

Describe what occurs with an enterocele

A

Weakening of the rectovaginal septum allowing the small intestine to herniate down between layers of the septal wall (pouch of douglas)
- usually asymptomatic

157
Q

Describe what occurs with a urethrocele

A

Round doughnut-shaped mucosa is observed protruding from the urethral opening.
- Vaginal bleeding is MC symptom

158
Q

What is procidentia?

A

Complete uterine prolapse (3rd degree)

159
Q

T/F: Many RTIs are asymptomatic, even serious ones requiring treatment

A

True

160
Q

What is the most common gynecologic complaint?

A

Vaginitis

161
Q

What are the common infectious agents for vaginitis?

A
  • Bacterial vaginosis
  • Trichomonas vaginitis
  • Candida vaginitis
  • Atrophic vaginitis (STI)
162
Q

Which organism is responsible for maintaining a healthy vagina?

A

Lactobacillus

163
Q

Describe bacterial vaginitis.

A

Overgrowth of normal bacteria

  • not sexually transmitted!!
  • gardnerella, haemophilus, GBS
164
Q

What are the clinical criteria for bacterial vaginitis?

A
  • amine “fishy” odor; whiff test
  • elevated pH
  • clue cells
  • discharge
165
Q

What are s/sx of trichomonas vaginalis?

A
  • Frothy yellow-green discharge*
  • Strawberry cervix
  • may be malodorous
  • erythema
166
Q

Which organs does trichomonas vag. infect?

A

Vagina, scene’s ducts, lower urinary tract

167
Q

What are s/sx of candida albicans aka yeast infection?

A

Pruritis, erythema, WHITE CURD-LIKE DISCHARGE :s

168
Q

What RTI is caused by low estrogen?

A

Atrophic vaginitis

169
Q

Which organisms infect the upper reproductive tracts?

A

Gonorrhea, Chlamydia trachomatis

170
Q

What are s/sx of CT?

A

May be asymptomatic

  • cervicitis, urethritis, PID
  • Reiter’s syndrome
171
Q

What are s/sx of NG?

A

May be asymptomatic

  • cervicitis, urethritis, PID
  • pharyngitis and arthritis
172
Q

Which conditions would cause mucopurulent cervicitis?

A

NG and CT; and PID

173
Q

What are s/sx of PID?

A
  • mucopurulent cervicitis
  • adnexal tenderness
  • disturbed menses
  • chills and fever
  • elevated ESR and WBC
174
Q

Which infection is caused by treponema pallidum?

A

syphilis by the spirochete itself!

175
Q

Describe primary syphilis

A
  • Within 3 weeks
  • Highly contagious
  • CHANCRE (Painless ulcer w/ firm borders)
  • regional lymphadenopathy
176
Q

Describe secondary syphilis AKA the great mimicker

A
  • 2-8wks after chancre, they develop a RASH on palms and soles
  • CONDYLOMA LATA
  • fever, sore throat, weakness, wt loss, hair loss (patchy)
177
Q

Describe tertiary syphilis

A
  • May begin as early as 1 year after infection
  • Gummata (sores) develop
  • Cardiovascular and neuro effects occur
178
Q

How do you dx syphilis?

A

Antibody testing (VDRL) and MHA-TP

179
Q

What STI is caused by haemophilus ducreyi?

A

Chancroid

180
Q

Describe chancroid

A

MUCHO painful lesion with ulceration of lymph node in the groin

181
Q

What should be your DDX for genital ulcers?

A

HSV, Syphilis, Chancroid, Lymphogranuloma venereum (LGV)-CT

182
Q

Number off the herpes viruses

A
HSV 1 = herpes simplex
HSV 2 
VZV (type 3)
EBV (type 4)
CMV (type 5)
HHV roseola (type 6)
HHV (type 7)
HHV Kaposi's sarcoma (type 8)
183
Q

Describe primary herpes progression

A
  • erythematous papule
  • vesicle
  • pustule
  • ulceration
  • encrustation
184
Q

What are the s/sx of primary herpes?

A

golden crusty ulceration, may be asymptomatic, Blisters/vesicles, pain/itching, local/systemic symptoms; sexual hx with partner known to have herpes; new partner

185
Q

T/F: HSV may cause unilateral keratitis (cornea), blepharitis and keratoconjunctavitis

A

True

186
Q

HPV is also known as _________________

A

Condyloma accuminata

187
Q

How do you treat HPV?

A
  • Podophyllin resin
  • Freezing via cryoprobe/nitrogen
  • CO2 laser- anesthesia