Guillain-Barre Syndrome Flashcards
guillain-barre syndrome
edema and inflammation which cause segmental loss of myelin sheath and expand nerve membranes r/t viral or bacterial infection
pathophysiology of GBS
nerve transmission is slowed or stopped, leading to flaccid paralysis with muscle denervation and atrophy; neruologic function returns in a proximal-distal pattern
main clinical manifestation of GBS
acute, ascending (feet and hands moving up), rapidly progressive, symmetric weakness of limbs
first symptoms of GBS
weakness; paresthesia (numbness, tingling); hypotonia (decreased muscle tone); reflexes in affected limbs may be absent
autonomic nervous system dysfunction from GBS
orthostatic hypotension; hypertension; abnormal vagal response (bradycardia, heart block, asystole); LOC; bowel and bladder dysfunction; facial flushing; diaphoresis; CN involvement (LOOK UP LATER)
clinical manifestations and complications of GBS
pain, paresthesia, muscular aches/cramps, hyperesthesia, worse at night, decreased appetite, sleep disruption, respiratory failure (paralysis progresses to nerves in thorax)
motor nursing management of GBS
ascending paralysis, reflexes, CN functions (LOOK UP), and LOC
nursing management for respiratory in GBS
ABGs and vital capacity
nursing management for cardiac in GBS
BP, rate, and rhythm
nursing management for fever and nutrition in GBS
antibiotics; high risk for aspiration
nursing management for acute phase of GBS
venilatory suppoirt
nursing management in the ICU for GBS
hemodynamic monitors
immunomodulating drugs for GBS
plasma exchange, high-dose IV immunoglobulin
when does recovery start for GBS
spontaneously approx. in 28 days