GU Flashcards
Retroperitoneum
Ant. Pararenal space - asc + desc colon, pancreas, 2nd and 3rd duod
Perirenal space- Kidneys, prox ureter, adrenals
Post. Pararenal space - only contains fat but can be involved with inflammation
Liposarcoma
Most common 1° retroperitoneal malignant tumour
Most commonly fat containing ( least aggressive)
More aggressive subtypes have minimal fat
Retroperitoneal fibrosis
Fibrosis deposition In the retroperitoneum leading to ureteric obstruction.
No displacement of aorta away from spine
Retroperiotneal haematoma
2° to ruptured AAA, trauma, renal AML or haemorrhagic cyst
Adrenal adenomas
Can cause cushings or conns syndrome.
Adrenal adenoma
Microscopic fat
Rapid wash-out characteristics on contrast enhanced CT
<10HU on non con is definite adenoma
>10 HU contrast given to assess characteristics
Adenoma has absolute washout >60% and relative washout >40%
Collsion tumour
Co-existence of two tumours within adrenal mass such as metastasis within adrenal adenoma or myelolipoma.
Absolute washout
Enhanced attenuation - delayed attenuation
/
Enhanced attenuation - unenhanced attenuation
Relative washout
Enhanced attenuation - delayed attenuation
/
Enhanced attenuation
Chemical shift imaging
Adenomas suppress on OOP images whilst metastases do not
Adrenal myelolipoma
Adrenal mass with macroscopic fat
Usually incidental and can be large (>4cm)
Pheochromocytoma
Neuroendocrine tumour of adrenals
Can be large and heterogenous due to central necrosis.
Avid enhancement on CT and ++T2 signal on MRI.
IO-123 and In-111 can be used to detect it.
Assoc. MEN 2, VHL,NF1,Carney’s triad .
If bladder is involved can cause post-micturition syncope .
Adrenal Mets
Lung and Melanoma are most common primaries.
Adrenal calcifications
Can be 2° to :
Previous haemorrhage
Granulomatosis with polyangitis
TB
Histoplasmosis
HIV nephropathy
Can cause bilateral enlarged and echogenic Kidneys
Unilateral delayed nephrogram (slow parenchymal uptake of contrast ) causes
Acute ureteral obstruction
Renal artery stenosis
Renal vein thrombosis
Acute pyelonephritis
Unilateral prolonged (hyperdense) nephrogram:
Acute ureteral obstruction
Renal artery stenosis
Renal vein thrombosis
Bilateral persistent nephrogram
Systemic hypotension
Acute tubular necrosis
Contrast or urate nephropathy
Myeloma (proteinuria)
Bilateral obstruction
Striated nephrogram causes
Acute obstruction
Pyelonephritis
Infarct
Acute tubular necrosis
Contusion
Hypotension
ARPKD
hypotension
Medullary nephrocalcinosis
Calcification of renal medullary pyramids secondary to hypercalcaemia or hypercalciuria
Preserved renal function
Causes :
Hyperparathyroidism
Sarcoid
Renal tubular acidosis
Medullary sponge kidney
Papillary necrosis
Cortical nephrocalcinosis
Dystrophic peripheral calcification of renal cortex with sparing of pyramids
Due to :
Cortical necrosis
Chronic glomerulonephritis
Transplant rejection
Alport syndrome (deaf too )
ARPKD.
Cortical necrosis
2° to hemolytic uraemic syndrome and thrombotic microangiopathy
Reduced renal cortex enhancement with preservation of medullary enhancement.
Papillary necrosis
Common causes:
NSAIDS, SCD,diabetes, renal vein thrombosis
US: focal echogenic papilla
CT: pooling of contrast in papillary regions adjacent to calyces. Can have filing defects in calyces, renal pelvis or ureter due to sloughed papilla
Ball on tee sign : contrast filling central papilla
Lobster claw: contrast filling periphery of papilla
Signet ring sign : contrast surrounding sloughed papilla
Renal artery pseudoaneurysm or AV fistula characteristics
Hyperattenuating focus with density similar to aorta
Decreases in attenuation on delayed phase
Active bleed will increase in attenuation or size with delayed phase imaging
Page kidney
Cause of 2° hypertension due to extrinsic compression of kidney by haematoma after trauma . Takes several months to develop.