Gingi & Perio in Children Flashcards
What are 3 differences in anatomy of periodontium in children compared to adults?
- More reddish colour
- Lower prevalence of periodontal disease
- Enhanced rate of progression of periodontal disease
Why are children’s periodontium more reddish than adults?
- Increased vascularity of marginal gingiva
- Thinner epithelium
Why is there lower prevalence of periodontal diseases in children compared to adults?
Junctional epithelium is thicker => less permeable to toxins => more resistant to inflammation
Why is there enhanced progression of perio disease in children compared to adults?
- PDL space wider, less fibrous and more vascular
- Alveolar bone has larger marrow spaces, greater vascularity and fewer trabeculae
What are 3 classes of dental biofilm-induced gingivitis?
- Associated with bacterial dental biofilm only
- Dental biofilm-induced gingivitis with modifying factors
- Drug-induced gingival enlargements
What are potential modifying factors of dental biofilm-induced gingivitis? (relevant to children)
Systemic factors:
- Sex steroid hormones (puberty, menstrual cycle etc)
- Hyperglycemia
Local factors
- Plaque retentive restorations
What are 4 drugs associated with drug-induced gingival enlargements?
- Phenytoin
- Cyclosporine
- Nifedipine (and other Ca2+ channel blockers)
- Valproate
What is the Mx of drug-induced gingival enlargement?
- Plaque control + improve OH
- Control inflammation – NSAIDs
- Alternate drug choices
- Surgical intervention
What is the proposed pathophysiology of drug induced-gingival enlargements?
- Increased inflammatory cytokines
- Increased fibroblasts
- Increased collagen accumulation
Drug-induced gingival enlargements are more prevalent in which age group and which location in the mouth?
- Higher prevalence in younger age groups
- More often in anterior
What are some conditions that fall under gingivitis – non dental biofilm induced?
- Mechanically-induced gingival trauma
- Primary herpetic gingivostomatitis (HSV-1)
What are the clinical characteristics of primary herpetic gingivostomatitis?
Prodrome of 2-4 days
- fever, malaise, headaches, cervical lymphadenopathy
Oral: generalised gingival inflammation, vesicles that rupture into ulcers
Resolves in 10-14 days
What is the management for primary herpetic gingivostomatitis?
- Oral acyclovir within first 72h
- Symptomatic care (anti-pyretics, hydration advice, soft diet)
- Prevention of secondary infection: CHX mouthwash
- Review: 7-10 days later (take immune status into consideration)
What are possible complications from primary herpetic gingivostomatitis?
Meningitis, encephalitis
What are the features of necrotising periodontal disease?
- Necrosis and ulceration in the interdental papillae
- Pseudomembrane formation
- Gingival bleeding
- Halitosis
- Pain