GI Flashcards

1
Q

Focus after delivery for these two structural disorders is airway and sucking.

A

Cleft lip and cleft palate

CL can be repaired by 3 mos
CP can be repaired around 6 mos

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2
Q

Protrusion of part of the gut or bowel through the inguinal ring into the scrotal or labial area

Treatment: Outpatient Surgery

A

Inguinal hernia

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3
Q

Common in infants

Umbilical rings does not close at the end of the first trimester

Treatment: None unless persists after 5yrs

A

Umbilical hernia

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4
Q

Complete closure of the anal passage
Immediate surgical intervention

A

rectal atresia

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5
Q

Narrowing/constriction of the rectal passage
Can have ribbon-like stools

A

rectal stenosis

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6
Q

No rectal opening
Child may have a fistula going to the GU system

A

imperforate anus

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7
Q

Thickened muscle (pylorus) causing a blockage causing projectile vomiting and olive shaped mass.

A

Pyloric stenosis

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8
Q

How is pyloric stenosis treated?

A

rehydration, surgery, & early feeds after surgery

D/C home is typically within 24 hrs

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9
Q

What is it?
Telescoping of a portion of the intestine, commonly at the ileocecal valve

How is it diagnosed?
Barium or Air Enema and imaging

How is it treated?
Enema or surgery if not corrected

A

Intussusception

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10
Q

Intussusception manifestations

A

jelly stools, sausage shaped mass, intermittent colicky cry, & fever

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11
Q

Abnormal rotation of the intestine around the superior mesenteric artery during fetal development

A

may lead to malrotation of the intestine. Life threatening if twisting of the intestine around itself, or volvulus, occurs.

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12
Q

S/sx of malrotation/volvulus

A

Intermittent bilious vomit/dehydration
Abdominal distention/pain
Lower GI bleeding
A palpable epigastric mass
Shock (if untreated)

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13
Q

Chronic inflammatory disease characterized by periods of exacerbations and remissions. Can affect any portion of the GI tract.

In children, acute or slow onsetabd pain, diarrhea, blood/mucous in stool, urgency/tenesmus, weight loss, RLQ cramping, fatigue

A

Crohn’s disease

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14
Q

An acute or chronic inflammation of the colon, which is characterized by recurring bloody diarrhea.

Symptoms include abd pain, bloody diarrhea, tenesmus, LLQ cramping, weight loss.

A

Ulcerative colitis

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15
Q

How is Crohn’s disease and Ulcerative colitis diagnosed? Treated?

A

By EGD and w/ surgery

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16
Q

Diet for Crohn’s

A

High fat, high carb, low residue, smaller/frequent meals, supplements

17
Q

Diet for UC

A

High in protein, high carb, normal fat, decreased roughage, supplements

18
Q

Inflammation of the appendix (end of cecum)

A

appendicitis

19
Q

A positive Rovsing’s sign is indicative of acute appendicitis, characterized by inflammation, infection, or swelling of the appendix. What is Rovsing’s sign?

A

refers to pain felt in the right lower abdomen upon palpation

20
Q

What manifestation would indicate the appendix is perforated?

A

sudden decrease in abdominal pain

21
Q

Clinical manifestations of appendicitis

A

-Umbilical pain –> RLQ
-Vomiting
-Fever
-Perforation

22
Q

Increased frequency and fluid content of the stools with or without associated symptoms.

Important to assess the presence of other signs and symptoms such as vomiting, fever, and pain.

23
Q

Explosive, watery, pale, & odor

24
Q

Green and watery stool

25
Stool +blood
Salmonella
26
Delay or difficulty passing stool for 2 or more weeks
Constipation
27
Clinical manifestations of constipation
Hardened stool Straining Rectal Pain Encopresis Withholding behaviors
28
Return of gastric contents from the stomach through the lower esophageal sphincter back up into the esophagus Functional reflux Causes infrequent, episodic nonbilious, undigested formula
GER
29
Pathological reflux (choking, apnea, frequent OM/URIs, poor weight gain) Common in premature infants and those with neurological issues
GERD
30
No intervention/tests needed for GER & GERD if weight gain is adequate True or False
True
31
Invervention for GER & GERD
May consider thickened formula or breast milk Need cross-cut nipple! May need lactose-free formula Smaller frequent feedings Feed slowly Do not vigorously play after feeding Feed in upright position 30° for after feeds
32
A syndrome in which infants or young children fail to eat enough food to be adequately nourished and achieve age-appropriate weight gain as a result of: Inadequate caloric intake, Inadequate caloric absorption, & Excessive caloric expenditures. Organic vs inorganic etiology Treatment based on cause
Failure to thrive
33
The absence of ganglion cells results in lack of motility in the affected portion of the bowel. AKA aganglionic megacolon.
Hirschprung's disease
34
Clinical manifestations of Hirschprung's disease
Failure to pass meconium Chronic constipation Abdominal obstruction Explosive BMs Vomiting Older children: foul smelling, ribbon-like stools
35
Decreased mucosal surface area. Usually from surgical resection of small bowel (due to NEC, volvulus, Crohn’s)
Short bowel syndrome
36
Treatment for short bowel syndrome
-Administration and monitoring of TPN -Enteral feeding -Emotional and developmental needs
37
Idiopathic, progressive, inflammatory process (postnatal or fetal) that causes an absence of some or all major intrahepatic and extrahepatic biliary ducts resulting in fibrosis and obstruction
Biliary atresia
38
Clinical manifestations of biliary atresia
Jaundice Urine is dark-stains the infant’s diaper Enlarged liver and spleen Itchy
39
Sx of Cannabinoid Hyperemesis Syndrome (CHS)
nausea, stomach pain, tendency to take hot baths and showers for relief, difficulty eating/keeping food down, weight loss, severe vomiting and or diarrhea sometimes lasting days/weeks