General Principles Flashcards

1
Q

What does cardiac coughing occur?

A

At night - compression of the corina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

When does respiratory coughing occur?

A

During exercise / excitement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What do you want to ask about in a cardiac specific history?

A
Coughs
Dysnpnoea
Tachypnoea
Exercise tolerance 
Syncope
Mm colour
Resting respiratory rate
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is the normal respiratory rate for a dog?

A

20-30

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How should you categories dysponea?

A

Inspiratory / expiratory
Restrictive - LRT
Or obstructive - URT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What should you check on a cardiac exam?

A

CO
Pulse quality, mm colour, CRT, warmth of extremities

Assess for signs of heart failure 
Forward:
- reduced pulses 
- slow CRT and pale mm
- cold extremities

Left sided congestive:

  • pulmonary crackles
  • cough
  • restrictive inspiratory and expiratory breathing pattern

Right sided congestive:

  • positive hepatojugular reflex
  • ascites
  • subcut oedema and muscle wastage
  • hepatomegaly
Cardiac auscultation 
Respiratory auscultation 
Palpate the larynx and trachea 
Thoracic percussion 
Thoracic compressibility - more useful in cats
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are eye clinical signs of forward heart failure?

A
= signs of poor cardiac output
Lethargy and exercise intolerance 
Weak femoral pulse
Unable to detect distal pulses
Pale MM and slow CRT
Cold extremities 
Hypothermia
Cardiogenic shock
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How should you treat forward heart failure?

A

DO NOT GIVE FLUIDS

Give positive inotropes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the signs of right sided congestive heart failure?

A
Ascites 
Distended jugular veins
Positive hepatojugular reflex 
Pleural effusion 
Subcutaneous oedema 
Muscle wasting
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What are the signs of left sided congestive heart failure?

A

Tachypnoea
Inspiratory and expiratory restrictive breathing pattern
Cough due to left atrial enlargement
Soft respiratory crackles on auscultation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is the normal heart rate in dogs?

A

70-160

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is the normal heart rate in cats?

A

160-240

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How should you describe a heart murmur?

A

PMI - left or right, base or apex
Timing - systolic, diastolic, continuous
Character - plateau, descrescendo, crescendo descrescendo, brief mid systolic, blowing
Radiation
Grade - I - VI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How do you grade heart murmurs?

A

I - very quiet murmur
II - murmur quieter than the heart sounds
III - murmur as loud as the hear sounds
IV - murmur louder than the heart sounds
V - louder the heart sounds and you can palpate precordial thrill
VI - louder than the heart sounds, precordial thrill, can heart murmur with the stethoscope lifted off the chest wall slightly

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is a diastolic gallop?

A

S3 and S4 heart sounds that are not normally audible in small animals

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How can you auscultate diastolic gallops?

A

Using the bell of the stethoscope

Applying very little pressure over the left apex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What are normal lung sounds called?

A

Bronchovesicular lung sounds - harsher when there is an increased respiratory rate

18
Q

Describe some adventitious lung sounds.

A

= abnormal lung sounds

Crackles - inspiratory - smaller airways opening
Wheezes - expiratory - due to narrowed airways - pulmonary fibrosis

19
Q

What does dull thoracic percussion suggest?

A

Pleural effusion

20
Q

What does resonance on thoracic percussion suggest?

A

Pneumothorax

21
Q

What can reduced thoracic compressibility in a cat mean?

A

Cranial mediastinal mass

22
Q

What is preload?

A

Venous return to the heart

Determines the end diastolic stretch on the atrial cardiomyocytes before contraction

23
Q

What is afterload?

A

The resistance to ventricular ejection during systole

24
Q

What are the major consequences of congestive heart failure?

A
Oedema and effusions
Peripheral vasoconstriction 
Tachycardia
Arrhythmias
Remodelling
Fibrosis of the myocardium
25
Q

What are the pathophysiological events that occur in response to a reduced CO in heart failure?

A

Vasoconstriction
Increased heart rate
Myocardial remodelling and fibrosis via RAAs
Na+ and water retention via aldosterone - oedema and effusion

26
Q

What adrenoreceptor as are involved in the baro-reflex?

A

Beta 1 adrenoreceptors

27
Q

What cardiac decompensation does increasing the heart rate via the baroreflex cause?

A

Increases myocardial oxygen consumption

Reduces coronary perfusion

28
Q

What cardiac decompensation does vasconstriction via the baroreflex cause?

A

Increases blood pressure

29
Q

What does sympathetic stimulation of the beta 2 adrenoreceptors in the kidney cause?

A

Renin secretion

30
Q

What is the effect of angiotensin II on the body

A
Potent vasoconstrictor 
Myocardial hypertrophy and fibrosis 
Causes ADH release
Increases GFR 
Increases sympathetic activity
Stimulates aldosterone release 

= attempts to increase preload by increasing venous return

31
Q

What substances in the body naturally counter-act the RAAs system?

A

Atrial natriuretic peptide - released following atrial stretch
Brain natriuretic peptide - released following ventricular stretch

  • antagonise the RAAs - vasodilation, cause diuresis and sodium excretion
32
Q

What clinical pathology tests are available for heart failure?

A

Pro-BNP - longer half life than normal BNP - can be assayed

Endothelin - possibly a future assay

33
Q

What are the major pathological consequences of congestive heart failure?

A

Oedema and effusions
Peripheral vasconstriction
Tachycardia / arrhythmias
Remodelling and fibrosis of the myocardium

34
Q

What are the mediators of vasoconstriction?

A

Noradrenaline
Angiotensin II
Reduced bradykinin levels vasopressin
Endothelin

35
Q

What mediators stimulate cardiac remodelling?

A

Angiotensin II
Aldosterone
Endothelin
Catecholamines

36
Q

What kind of hypertrophy does volume overload cause?

A

Eccentric hypertrophy = chamber dilation

37
Q

What kind of hypertrophy does pressure overload cause?

A

Concentric hypertrophy - wall thickened

38
Q

What cardiac diseases cause eccentric hypertrophy?

A
Mitral valve regurgitation 
Ventricular septal defects 
Patent ductus arteriosus 
Tricuspid regurgitation 
Anaemia 
Exercise
39
Q

What cardiac disease cause concentric hypertrophy?

A
Aortic stenosis
Systemic hypertension
Pulmonic stenosis
Pulmonary hypertension 
Exercise
40
Q

Define maladaptive remodelling.

A

Changes in relative wall thickness + altered geometry of the heart chamber

41
Q

Why do tachycardia and arrhythmias occur during heart failure?

A

Elevated catecholamine levels
Increased sympathetic drive
Reduced vagal tone
Myocardial fibrosis and ischaemia due to remodelling