Gastrointestinal Sytem Flashcards

1
Q

Hiatal hernia

A

enlargement of lower esophageal sphincter

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Hiatal hernia prevelance

A

common, 5/1000

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Hiatal hernia risks

A

Advanced aging
Women
Can be congenital or acquired

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Type 1 Hiatal Hernia

A

Sliding hernia (most common)
Stomach and gastroesophageal junction are displaced upward into thorax

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Type 2 Hiatal Hernia

A

Rolling “paraesophageal” hernia (higher risk)
gastroesophageal junction stays below diaphragm, but all or part of the stomach pushes through into thorax

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Hiatal Hernia Clinical manifestations

A

Many asymptomatic
Heartburn
Chest pain
Dysphagia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Sliding Hernia clinical manifestations

A

Heartburn 30-60 minutes after a meal, especially in supine
substernal pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Rolling hernia clinical manifestations

A

difficult and painful swallowing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Hiatal hernia diagnosis

A

Barium swallow
xray
upper endoscopy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Hiatal hernia treatment

A

antacid
potential surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

GERD

A

backward flow of gastric contents into esophagus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

GERD Clinical manifestations

A

heartburn
dysphagia
edema and spasm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Chronic GERD can lead to

A

Barrett’s esophagus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

GERD diagnosis

A

History
Endoscopy
Barium radiography
H. plyori
Esophageal monitoring
Medications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Gastritis

A

injury and inflammation of stomach mucosa

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

etiology of gastritis

A

aspirin/NSAIDS and chemotherapy
physiologic stress
hospitalization for severe illness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

gastritis clinical manifestations

A

epigastric pain
abdominal distention
anorexia and los of appetite
nausea
indigestion
painless GI bleeding

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

gastritis diagnosis

A

history
upper GI endoscopy to rule out ulcer our tumor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

gastritis treatment

A

avoid irritants
acid suppressors and anitbiotics

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

gastritis prognosis

A

good if predisposing factor removed
chronic gastritis increases risk of stomach cancer

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

esophageal cancer types

A

squamous cell carcinoma
adenocarcinoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

squamous cell carcinoma

A

majority of esophageal cancer
middle of esophagus
african americans
alcohol/tobacco use

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

adenocarcinoma

A

distal esophagus
middle aged white men
GERD, barrets esophagus
obesity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

esophageal cancer clinical manifestations

A

dysphagia and pain swallowing
anorexia
heartburn, chest pain (spread of tumor)
cough and pneumonia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
hoarseness of voice from esophageal cancer suggests involvement of which nerve?
recurrent laryngeal nerve
26
esophageal cancer treatment
surgical resection chemo
27
esophageal cancer prognosis
poor, bc 50 percent are detected at advances stage high morbidity and mortality metastasizes rapidly
28
Peptic Ulcer
open sores that develop on the stomach lining or small intestine that can lead to perforations and GI bleed
29
Peptic ulcer (Gastric)
affects lining of stomach
30
Peptic ulcer (Duodenal)
affects duodenum
31
Protection of Stomach Lining (mucus)
secreted by cells in mucosa and coats lining
32
Protection of Stomach Lining (bicarbonate)
secreted by epithelial cells of GI neutralizes gastric acid
33
Protection of Stomach Lining (adequate blood flow)
nourishes mucosa
34
Protection of Stomach Lining (prostaglandins)
stimulate mucus and bicarbonate secretion
35
Cause of peptic ulcer disease (H. plyori)
most common cause hides between mucus and epithelial cells
36
Cause of peptic ulcer disease (NSAIDS)
Inhibit prostoglandins
37
Cause of peptic ulcer disease (Gastric Acid)
Injure mucosa cells and activate pepsin
38
Cause of peptic ulcer disease (smoking and alcohol)
increase GA reduce bicarbonate prod. delay healing
39
Best diagnosis for PUD
endoscopy
40
PUD prognosis
good unless massive hemorrhage or perforations
41
IBS (Crohn Disease) locations and pain
anywhere in GI tract, including mouth RLQ pain
42
IBS (Crohn Disease) lesions
Skip lesions
43
IBS (Crohn Disease) symptoms
weight loss, fecal urgency, pain relieved by farting, joint arthritis, abdominal pain
44
IBS (Ulcerative Colitis) locations and pain
occur in large intestine and rectum LLQ pain
45
IBS (Ulcerative Colitis) lesions
continous
46
IBS (Ulcerative Colitis) symptoms
weight loss, fecal urgency, rectal pain, bleeding, bloody diarrhea with mucus/pus
47
IBS CD prognosis
chronic, incurable, debilitating
48
IBS UC prognosis
possible by colon resection
49
General IBS prognosis
Increased risk of GI cancer mortality risk low, but increases with durations and severity of disease
50
Implications for PTs (CD IBS)
may present as LBP may cause psoas abscesses (fever, LAP, referred pain) +ve psoas signs dehydrations psychological issues
51
Diverticulum
asymptomatic outpouching in the colonic mucosa from weakness affects sigmoid colon
52
Diverticulitis
inflammation of a diverticulum
53
Diverticulitis Risk Factors
Advanced age >85 Chronic constipation low fiber low exercise obesity smoking NSAIDs and immunosuppressants prior episodes
54
Diverticulitis Pathogenesis
due to blocked diverticulum, leading to proliferation of bacteria and inflammation
55
Diverticulum Symptoms
asymptomatic
56
Diverticulitis Symptoms
Abdominal pain in LLQ, often with back pain fever change in bowel habits nausea, vomiting, anorexia
57
Diverticulitis Treatment
prevent with high fiber smoking cessation bowel rest antibiotics surgery
58
Colorectal cancer risk factors
age male poor diet IBD family history obesity
59
Prevention of colorectal cancer
screening, colonoscopy, removal of polyp
60
Colorectal cancer clinical manifestations
blood from rectum blood loss melena weight loss change in bowel habits
61
colorectal cancer prognosis
5 year survival for all stages is combined 64%
62
Appendix
hollow muscular closed ended tube 10 cm in length unclear function
63
appendicitis risk
young men peak 15-19 year olds but can occur at any age
64
appendicitis etiology
50% has no known cause infection, distention, ischemia of obstruction that forms abscess and perforates
65
appendicitis clinical manifestations
RLQ pain peri-umbilical McBurney's point fever nausea and vomiting
66
Signs of perforation in appendicitis
WBC of 20,0000/mm3 or greater tense or rigid abdomen elevated temperature (102°F)
67
appendicitis implications for PTs
early recognition and referral is important may present as right thigh pain, groin/testicular pain, pelvic or referred hip pain
68
Peritonitis
inflammation of peritoneum
69
Peritonitis clinical manifestations
Severe AP Abdominal rigidity and distention nausea/vomiting diarrhea and fever can lead to septic shock and cardiopulmonary collapse
70
hemmorrhoids
varicose veins in peri-anal region (internal and external)
71
hemorrhoids are associated with
increased IAP
72
Hemmorrhoids can also be caused by
PH pregnancy prolonged sitting standing or heavy lifting low fiber diets with constipation obesity diarrhea
73
hemorrhoid treatment
remove factors sitz baths high fiber surgery