Gastrointestinal Disorders & Duodenal Ulcers Flashcards

1
Q

Inflammation of gastric mucosa

A

Gastritis

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2
Q

Non-erosive gastritis

A

H. Pylori

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3
Q

Erosive gastritis

A

NSAID, ETOH use

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4
Q

Is stress related gastritis erosive or non-erosive?

A

Non-erosive

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5
Q

Onset of gastritis

A

Rapid (acute)

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6
Q

Manifestations of gastritis

A
  • Abdominal discomfort
  • Dyspepsia (indigestion)
  • Nausea/vomiting/anorexia
  • Blood in vomit or black tarry stool
  • Fatigue
  • Burning sensation in stomach
  • Intolerance to spicy or fatty foods
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7
Q

Erosive may cause…

A

Bleeding

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8
Q

Diagnostics for gastritis (2) (Which is gold standard?)

A

Identification of causative factors
- EGD (gold standard)
- H-pylori

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9
Q

Risk with EGD

A

Perforation

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10
Q

Treatment for gastritis

A
  • Triple therapy (H. Pylori)
  • NGT and IV fluids
  • Antacids
  • H2 blockers
  • Proton pump inhibitors
  • Vitamin B12
  • Diet/lifestyle changes (for chronic) (avoid spicy)
  • Gastric resection (extreme case)
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11
Q

What is triple therapy?

A

PPI and 2 antibiotics for 14 days

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12
Q

Nursing role/Interventions for gastritis

A
  • Pain management
  • Anxiety reduction
  • Optimize nutritional status (intake & output) (treat pain so pt can eat food)
  • Fluid balance
  • Monitor/assess for hemorrhage
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13
Q

What do we not want to give for pain with gastritis?

A

No NSAIDs

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14
Q

What are we monitoring and assessing for hemorrhage?

A
  • Quantity of loss
  • Color
  • Correction of loss
  • S/S associated with hypovolemic shock

Decrease BP, urine output
Increase HR/RR, Hgb/CBC

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15
Q

S/S associated with hypovolemic shock

A

Low BP
Tachypnea

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16
Q

1 PRBC is how many mL

A

350 mL

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17
Q

Where can a peptic ulcer form?

A
  • Mucosal wall of the stomach
  • Pylorus
  • Duodenum
  • Esophagus
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18
Q

Excavation

A

Hollowed out area

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19
Q

This looks like a little hole

A

Peptic ulcer

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20
Q

Peptic ulcers are associated with..

A
  • H. Pylori
  • NSAID and ASA use
    (synergistic affect between H. Pylori and NSAIDs)
  • Excess secretion of HCL (stress may be a factor)
  • Type O blood type
  • Chronic kidney or lung disease
  • Older adults
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21
Q

Most duodenal ulcers occur in…

A

The first portion of the duodenum

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22
Q

Deep, sharply demarcated lesions that penetrate through the mucosa and submucosa

A

Duodenal ulcers

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23
Q

Use what to treat duodenal ulcers?

A

Wait for response to PPI
Then, Scope

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24
Q

Increased stress =

A

Increased gastric acid secretion

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25
Q

Acute gastric mucosa lesions occurring after an acute medical crisis or trauma

A

Stress ulcers

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26
Q

What type of ulcer is associated with head injury, major surgery, burns, respiratory failure, shock, and sepsis?

A

Stress ulcer

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27
Q

Principal manifestation of stress ulcer

A

Bleeding caused by gastric erosion

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28
Q

What is key for stress ulcers?

A

Prevention

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29
Q

What do we use for prevention of stress ulcers?

A
  • Proton pump inhibitors (Omeprazole)
  • H2 antagonists
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30
Q

Peptic ulcer disease types

A
  • Gastric ulcers
  • Duodenal ulcers
  • Stress/drug induced ulcers
31
Q

What type of ulcer is this?
- Weight loss
- Burning left epigastric area
- Food frequently aggravates pain
- No pain during sleep

A

Gastric ulcer

32
Q

Duodenal ulcer signs

A
  • Epigastric pain at HS
  • Burning, cramping, mid epigastric pain
  • Pain 2-4 hours after meal (eating decreases pain)
  • Weight gain
  • Nausea/vomiting
33
Q

Stress/drug induced ulcer signs

A

Asymptomatic

34
Q

PUD diagnosis

A
  • EGD
  • Abdominal films (looking for “free air”)
  • H. Pylori
  • CBC
35
Q

PUD H. pylori test

A

Breath test or serum (blood) antibodies

36
Q

PUD CBC test

A
  • Assessment for bleeding
  • Stool testing for occult blood
37
Q

PUD abdominal films

A
  • Looking for “free air”
    (indicates perforation)
38
Q

PUD medical treatment goals

A
  • Pain relief
  • Decrease/remove H. Pylori
  • Heal ulcerations
  • Reduce chance of reoccurrence
39
Q

Diet wise, what do we want to avoid for PUDs? What DO we want?

A

Avoid coffee, caffeine, smoking
We want small, frequent meals

40
Q

What meds do we want for PUD?

A

H2 blockers
Proton pump inhibitors

41
Q

How to we get rid of H. pylori?

A

Antibiotics/anti-infective care are only option
10-14 days triple therapy

42
Q

PUD complications

A
  • Hemorrhage
  • Perforation
  • Penetration
  • Peritonitis
42
Q

How do we heal ulcerations and reduce chance of reoccurrence?

A

Holistic care

43
Q

Erosion eats through blood vessels

A

Hemorrhage

44
Q

Hemorrhage manifestations

A
  • Bleeding
  • Hypovolemia and shock
45
Q

Types of bleeding

A
  • Hematemesis
  • Melena
  • Coffee ground (emesis)
46
Q

Lab values for hemorrhage

A

Hct
Hgb
Type and screens - need a blood transfusion

47
Q

Hemorrhage nursing responsibilities

A
  • Insert large bore IV catheter
  • Quickly determine amount of blood lost
  • Monitor H and H, Coags, Type and Screen
  • Insert NG tube
  • Administer lavage
  • Foley
  • Monitor VS and administer oxygen
  • Position patient to prevent hypotension/prevent aspiration
48
Q

Lavage

A

Washing out of body cavity
- Can be room temp or iced

49
Q

Large bore IV catheter for PUD

A

Saline, LR, blood products

50
Q

Erosion through the ulcer directly into peritoneum

A

Perforation

51
Q

Two types of peritonitis

A

Bacterial and chemical

52
Q

Would you rather have perforation of stomach or colon?

53
Q

Erosion through the gastric serosa into adjacent structures

A

Penetration

54
Q

Where does penetration take place?

A

Pancreas, biliary tract, gastro-hepatic

55
Q

What are two abdominal catastrophes that require immediate surgery?

A

Perforation and penetration

56
Q

Manifestations of perforation and penetration

A
  • Sudden, severe upper abdominal pain (may be referred to right shoulder or back)
  • Vomiting and collapse
  • Tender, board like abdomen
  • Hypotension, tachycardia
  • Shock
57
Q

Late signs of peritonitis

A

Hypotension and tachycardia

58
Q

Peritonitis manifestations

A
  • Fever
  • Abdominal pain
  • Paralytic ileus
  • Decreased or absent bowel sounds
  • Abdominal distention
59
Q

Peritonitis treatment

A

Antibiotics

60
Q

5th most common cancer diagnosis worldwide

A

Gastric cancer

61
Q

Where is gastric cancer most common?

62
Q

How do we identify gastric cancer?

A

Screening programs

63
Q

Who is at a higher risk for gastric cancer?

64
Q

90% of gastric cancer identifies as…

A

Adenocarcinomas

65
Q

How do adenocarcinomas start?

A

Arise from the mucous producing cells in the inner most lining of the stomach

66
Q

What occurs early in gastric cancer?

A

Lymph node involvement and metastasis

67
Q

Clinical manifestations of gastric cancer

A
  • Pain relieved by antacids
  • Dyspepsia
  • Weight loss
  • Pain in abdomen above umbilicus
  • Decreased appetite
  • Bloating, nausea, vomiting
68
Q

Advanced stages of gastric cancer produce symptoms similar to…

69
Q

Gold standard for diagnostics of gastric cancer

A

EGD for biopsy and cytologic washings

70
Q

Diagnostics of gastric cancer

A
  • EGD
  • Barium X-Ray of upper GI tract
  • Endoscopic Ultrasound
  • CT scan
  • CBC
  • Tumor markers
71
Q

Management of gastric cancer

A
  • Surgery (total gastrectomy, radical partial gastrectomy)
  • Chemo & radiation
72
Q

Nursing care for gastric cancer

A
  • Promotion of nutrition
  • Pain management
  • Psychosocial & anxiety support
73
Q

Medication for pain management of gastric cancer

A

Analgesics