Gastroenterology Flashcards

1
Q

Acute pancreatitis first step in management

A

Always IV fluid at rate of 250cc/h for the first1-2 days.

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2
Q

Acute pancreatitis Xray finding

A

Sentinel loop (proximal small bowel distention due to focal pancreatic inflammation). Usually doesn’t need NGT decompression

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3
Q

Acute diverticulitis AB of choice

A
  • Metrondiazole PLUS cipro/levoFloxacin or Bactrum
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4
Q

Duodenal Vs. Gastric ulcer

A

Gastric: require repeating Upper Endoscopy after PPI
Duedonal: no need

PPI promote ulcer healing

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5
Q

Acute mesentric ischemia test of choice

A

CT angio

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6
Q

Coffee bean sign versus Pseudotumor sign

A

Coffee bean: U shaped bowel
Pseudo-tumor: fluid filled loop resemble a mass

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7
Q

Appendicitis incidence age peak

A

10-14 years

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8
Q

Best imaging for Renal stone

A
  • CT Scan without contrast
  • Renal U/S can diagnose stones, but may miss distal pathology (distal uretur)
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9
Q

Two third of patients with GERD have endoscopic findings?

A

FALSE, 2/3 have no endoscopic findings

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10
Q

GERD treatment sequence

A

1- Lifestyle and dietary modifications
2- OTC antacids
3- H2 receptor antagonist “rantidine”
4- 8w course of 20mg PPI
5- Double the dose
6- Anti-reflux surgery

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11
Q

PPI long term risks?

A

1- C diff related colitis’
2- Drug induced lupus

2013 meta analysis showed that PPI was not associated with increased risk of hospital admission due to cap

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12
Q

Dyspepsia definition

A

Upper abdominal discomfort and fullness

Red flags:
- Age >60
- bleeding, weight loss, dysphagia, family history of GI cancer

If red flags > Upper endoscopy
If no red flags > test for H.pylori

Positive> treat
Negative> PPI

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13
Q

Non alcoholic fatty liver disease

A
  • Cirrhosis is more common with alcoholic than non alcoholic
  • Level of AST and ALT are usually high in patient with non alcoholic steatohepatitis
  • Liver biopsy is the only way to confirm the diagnosis of non alcoholic steatohepatitis ( suspected in obese, abnormal LFT, DM, Splenomegaly, PE findings )
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14
Q

Serology for Hepatitis B?

A

Negative surface Ag >

1- previous infection (2 AB positive core and surface ), or
2- previous immunity ( only surface AB positive )

Positive surface Ag >

HbcAb (igM) : acute infection
HbcAb (igG) : chronic infection

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15
Q

Core antibodies in Hepatitis B ?

A
  • HbcAb total: positive from onset of symptoms, and remain for life
  • HbcAb (igM): within the first 6 months of acute infection
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