Gallbladder/Biliary Tree Flashcards
Function of GB
Concentrate & store bile
Identifying landmarks of GB
Right PV & Main lobar Fissure
Anatomy of GB
Neck, Body, Fundus
Normal size of GB wall
3 mm
GB Volume Equation
(L * W * H) .52
Cystic Duct connects
GB Neck to Common Hepatic Duct
First/last to dilate if CBD is backed up
Gallbladder is first, CBD is last
Flow of bile
Liver → R/L Hepatic Ducts → Common Hepatic Duct → Cystic Duct → Gallbladder → Common Bile Duct → Pancreatic Duct → Ampulla of Vater → Sphincter of Oddi → Duodenum
Intrahepatic Dilation
Dilation which pushes bile back into liver; occurs at CHD, LHD, RHD
Extrahepatic Dilation
Pushes bile away from liver; blockage after cystic duct
Spiral Valves of Heister
Connect gallbladder neck to cystic duct; control inward/outward flow of bile
Anatomy of Ampula of Vater (aka)
Hepatopancreatic Ampulla; Consists of duodenal papilla & Sphincter of Oddi
Sphincter of Oddi
Controls flow of bile & pancreatic juices into duodenum
Cholecystokinin (CCK)
Controls the flow of bile
Vessels from Posterior → Anterior
IVC, Portal Vein, Hepatic Artery, Bile Duct
Bile
Breaks down fat & RBC; made up of bile salts cholesterol, bilirubin
Bilirubin
Pigment formed from breakdown of RBC, associated w/ jaundice
Avg size of bile duct
< 5 mm
Avg size of bile duct after cholecystectomy
10 mm or 1 cm
Direct Bilirubin
Bilirubin prevented from going into liver by an obstruction
Indirect Bilirubin
Loss of bilirubin due to hepatic damage; problem in the process of making bilirubin
Lab Values Associated w/ GB
Bilirubin, alkaline phosphatase, lactic dehydrogenase
Phygian Cap
Fold between the body and fundus
Junctional Fold
aka Hartman Pouch; fold between body & neck of GB
GB Variants
Phygian cap, junctional fold, septation, agenesis, duplication
Gallstones aka ____, formed from
Cholelithiasis; Formed by irregular bowel movements resulting in bile stasis, abnormal bile composition, infection
4 F’s
Fat, female, forty, fertile
Symptoms of Gallstones
Asymptomatic, RUQ pain, fatty food intolerance
US Appearance of Gallstones
Highly echogenic, posterior shadowing, mobile
Mirizzi Syndrome
Stone impacted in the cystic duct or neck of GB; result in dilated intrahepatic ducts
WES Triad
Wall echo shadowing; 2 thin curved parallel lines caused by an excess of gallstones
Sludge Ultrasound Appearance
Low level echoes, non-shadowing, slow moving
Cholecystisis
Inflammation of GB
Acute: Due to blockage of cystic duct
Chromic: Due to infection, obstruction, or metabolic disorder
Symptoms of Acute Cholecystitis
RUQ pain, fever, nausea, vomiting, leukocytes
Appearance of Cholecystitis
Gallstones, wall thickening, Murphy’s sign, round GB, sludge
Complications of Acute Cholecystitis
Empyema (pus), emphysematous cholecystitis, gangrenous cholecystitis, GB perforation, abscess
Hydrops (aka)
Mucocele, thin-walled, distended gallbladder due to prolonged obstruction; filled w/ mucus, pus, or bile
GB Polyps
Benign masses attached to wall of GB; no shadowing or change in position
Porcelain GB
Calcification of GB wall; associated w/ chronic cholecystitis, cholelithiasis, carcinoma
Adenomyomatosis
Benign growth of GB wall; main characteristic: comet tail artifact due to stones trapped in RA sinuses
RA Sinues
Rokitansky-Aschoff
Cholesterolosis
Accumulation of sterols/fats in GB wall; small non-shadowing masses projecting into GB; strawberry GB
GB Carcinoma Prevalance
Rare but more common in elderly & females; high association w/ gallstones
GB Carcinoma Symptoms
Loss of appetite, nausea, vomiting, RUQ pain, jaundice
Ultrasound Appearance of GB Carcinoma
Irregular wall borders, wall thickening, mass in lumen, gallstones, enlarged lymph nodes
Most common metastases of GB Carcinoma
Melanoma
Choledocal Cyst
Congenital, mostly in infants w/ failure to thrive, intermittent jaundice, and palpable RUQ mass; dilation of CBD
Caroli’s Disease
Rare congenital condition that causes the bile ducts to be larger than usual leading to the formation of bile duct stones
Complications of Caroli’s Disease
Lithiasis in bile ducts, recurring cholangitis
Appearance of Caroli’s Disease
Multiple beads resembling “string like beads”
Biliary Atresia
Narrowing/obliteration of ducts due to obstruction
Intrinsic Biliary Atresia
Obstruction within bile ducts; choledocholithiasis, cholangiocarcinoma, inflammation due to cholangitis
Extrinsic Biliary Atresia
Obstruction due to factor occurring outside bile ducts; pancreatitis, pancreatic mass, pancreatic psudocyst, hepatic mass, periductal adenopathy
Appearance of Biliary Atresia
Tubular lucencies w/ deer antlers/stellate branches, Shotgun/Parallel Sign
Choledocholithiasis
Biliary obstruction caused by stones in the bile ducts (formed in GB)
Common location of choledocholithiasis
Distal CBD near Ampulla of Vater
Choledocholithiasis is associated with patients with
Cholelithiasis and chronic cholecystitis
Symptoms of choledocholithiasis
RUQ pain, jaundice, elevated bilirubin, biliary colic, hepatomegaly
Cholangiocarcinoma
Malignant carcinoma of the bile ducts
3 Types of Cholangiocarcinoma
Scirrhous, Polyploid, Klatskin’s Tumor
Klatskin’s Tumor
Carcinoma located at the junction of the right and left hepatic ducts, or porta hepatis
Cholangitis
Chronic inflammatory and fibrotic disorder of the biliary ducts
Ultrasound Appearance of Cholangitis
Thickened wall, sludge, dilated bile ducts
Symptoms of Cholangiocarcinoma
RUQ pain, pruritis, jaundice, weight loss/anorexia, biliary colic, palpable abdominal mass